text: The role, if any, that the meningeal lymphatics play in diseases of the nervous system is an area of active research - particularly with regard to neurological disorders in which immunity is a fundamental player such as multiple sclerosis, Alzheimer’s Disease, amyotrophic lateral sclerosis, Hennekam syndrome, and Prader-Willi syndrome. Importantly, however, preliminary data suggest that there seem to be identifiable meningeal lymphatic vessels in post-mortem human tissue, suggesting the possibility that they contribute to human disease.

text: The role, if any, that the meningeal lymphatics play in diseases of the nervous system is an area of active research - particularly with regard to neurological disorders in which immunity is a fundamental player such as

entity_name: Multiple sclerosis

text: multiple sclerosis

entity_name: Alzheimer's disease

text: Alzheimer’s Disease

entity_name: Amyotrophic lateral sclerosis

text: amyotrophic lateral sclerosis

entity_name: Hennekam syndrome

text: Hennekam syndrome

text: , and

entity_name: Prader–Willi syndrome

text: Prader-Willi syndrome

text: . Importantly, however, preliminary data suggest that there seem to be identifiable meningeal lymphatic vessels in

entity_name: Autopsy

text: post-mortem

text: human tissue, suggesting the possibility that they contribute to human disease.

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text: Born in Paris on 17 August 1828 he became a doctor of medicine in 1857 and conducted extensive research on the anatomy, pathology and functions of the central nervous system. In 1865 he published a treatise entitled Studies on the Structure, Functions and Diseases of the Cerebro-spinal System, this book was accompanied by a hand-drawn three-dimensional atlas of the brain. It was within this book that Luys provided the first description of the structure that is today called the subthalamic nucleus. Luys termed this nucleus the bandelette accessoire des olives supérieures (accessory band of the superior olives) and concluded that it was a centre for the dispersion of cerebellar influence upon the striatum. Luys also traced the projection from the subthalamic nucleus to the globus pallidus and the projection to the subthalamic nucleus from the cerebral cortex. Today these pathways and structures are thought to be central to the pathophysiology of Parkinson's disease, the subthalamic nucleus being one of the major targets for deep brain stimulation.

text: Born in

entity_name: Paris

text: Paris

text: on 17 August 1828 he became a doctor of medicine in 1857 and conducted extensive research on the anatomy, pathology and functions of the central nervous system. In 1865 he published a treatise entitled Studies on the Structure, Functions and Diseases of the Cerebro-spinal System, this book was accompanied by a hand-drawn three-dimensional atlas of the

entity_name: Brain

text: brain

text: . It was within this book that Luys provided the first description of the structure that is today called the

entity_name: Subthalamic nucleus

text: subthalamic nucleus

text: . Luys termed this nucleus the bandelette accessoire des olives supérieures (accessory band of the superior olives) and concluded that it was a centre for the dispersion of

entity_name: Cerebellum

text: cerebellar

text: influence upon the

entity_name: Striatum

text: striatum

text: . Luys also traced the projection from the subthalamic nucleus to the

entity_name: Globus pallidus

text: globus pallidus

text: and the projection to the subthalamic nucleus from the

entity_name: Cerebral cortex

text: cerebral cortex

text: . Today these pathways and structures are thought to be central to the

entity_name: Pathophysiology

text: pathophysiology

text: of

entity_name: Parkinson's disease

text: Parkinson's disease

text: , the subthalamic nucleus being one of the major targets for

entity_name: Deep brain stimulation

text: deep brain stimulation

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answers: Parkinson's disease

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answers: NDQ_010497

answers: Parkinson's disease

answers: NDQ_010498

answers: Parkinson's disease

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: Acetylcholine has functions both in the peripheral nervous system (PNS) and in the central nervous system (CNS) as a neuromodulator. In the peripheral nervous system, acetylcholine activates muscles, and is a major neurotransmitter in the autonomic nervous system.

text: Acetylcholine has functions both in the

entity_name: Peripheral nervous system

text: peripheral nervous system

text: (PNS) and in the

entity_name: Central nervous system

text: central nervous system

text: (CNS) as a

entity_name: Neuromodulation

text: neuromodulator

text: . In the peripheral nervous system, acetylcholine activates muscles, and is a major neurotransmitter in the autonomic nervous system.

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answers: NDQ_010489

answers: (PNS)

answers: NDQ_010490

answers: central nervous system

answers: NDQ_010494

answers: central nervous system

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answers: central nervous system

answers: NDQ_010496

answers: peripheral nervous system

answers: NDQ_010497

answers: central nervous system

answers: NDQ_010498

answers: central nervous system

answer: No

answer: No

answer: No

answer: No

answer: No

answer: 1

text: The parasympathetic nervous system has been said to promote a "rest and digest" response, promotes calming of the nerves return to regular function, and enhancing digestion. Functions of nerves within the parasympathetic nervous system include:

text: The parasympathetic nervous system has been said to promote a "rest and digest" response, promotes calming of the nerves return to regular function, and enhancing digestion. Functions of nerves within the parasympathetic nervous system include:

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answers: Rest and digest

answers: NDQ_010494

answers: Alzheimer's disease

answers: NDQ_010495

answers: immune system

answers: NDQ_010496

answers: Parkinson's disease

answers: NDQ_010497

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answers: NDQ_010498

answers: epilepsy

answer: No

answer: No

answer: No

answer: No

answer: No

answer: 1

text: Cholinergic nerves play an important role in the normal function of the central nervous, endrocrine, neuromuscular, immunological, and respiratory system. As all cholinergic fibers contain high concentrations of ACh and AChE at their terminals, inhibition of AChE can impair their function. So exposure to azinphosmethyl, whereas it inhibits AChEs, may disturb a lot of important systems and may have various effects.In the autonomic nervous system, accumulation of acetylcholine leads to the overstimulation of muscarinic receptors of the parasympathetic nervous system. This can affect exocrine glands (increased salivation, perspiration, lacrimation), the respiratory system (excessive bronchial secretions, tightness of the chest, and wheezing), the gastrointestinal tract (nausea, vomiting, diarrhea), the eyes (miosis, blurred vision) and the cardiovascular system (decrease in blood pressure, and bradycardia). Overstimulation of the nicotinic receptors in the para- or sympathic nervous system may also cause adverse effects on the cardiovascular system, such as pallor, tachycardia and increased blood pressure. In the somatic nervous system, accumulation of acetylcholine may cause muscle fasciculation, paralysis, cramps, and flaccig or rigid tone. Overstimulation of the nerves in the central nervous system, specifically in the brain, may result in drowsiness, mental confusion and lethargy. More severe effects on the central nervous system include a state of coma without reflexes, cyanosis and depression of the respiratory centers. Thus the inhibition of the enzyme AChE may have a lot of different effects.

text: Cholinergic nerves play an important role in the normal function of the central nervous, endrocrine, neuromuscular, immunological, and respiratory system. As all cholinergic fibers contain high concentrations of ACh and AChE at their terminals, inhibition of AChE can impair their function. So exposure to azinphosmethyl, whereas it inhibits AChEs, may disturb a lot of important systems and may have various effects.In the autonomic nervous system, accumulation of acetylcholine leads to the overstimulation of muscarinic receptors of the parasympathetic nervous system. This can affect exocrine glands (increased

entity_name: Saliva

text: salivation

entity_name: Perspiration

text: perspiration

entity_name: Tears

text: lacrimation

text: ), the respiratory system (excessive

entity_name: Bronchus

text: bronchial

text: secretions, tightness of the chest, and wheezing), the gastrointestinal tract (nausea, vomiting, diarrhea), the eyes (

entity_name: Miosis

text: miosis

text: , blurred vision) and the cardiovascular system (decrease in blood pressure, and

entity_name: Bradycardia

text: bradycardia

text: ). Overstimulation of the nicotinic receptors in the para- or sympathic nervous system may also cause adverse effects on the cardiovascular system, such as pallor, tachycardia and increased blood pressure. In the somatic nervous system, accumulation of acetylcholine may cause muscle fasciculation, paralysis, cramps, and

text: flaccig

text: or rigid tone. Overstimulation of the nerves in the central nervous system, specifically in the brain, may result in drowsiness, mental confusion and lethargy. More severe effects on the central nervous system include a state of coma without reflexes, cyanosis and depression of the respiratory centers. Thus the inhibition of the enzyme AChE may have a lot of different effects.

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answers: 0: The answer is not relevant or complete at all.

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answers: Overstimulation

answers: NDQ_010490

answers: cyanosis

answers: NDQ_010494

answers: central nervous system

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answers: immunological

answers: NDQ_010496

answers: coma

answers: NDQ_010497

answers: central nervous system

answers: NDQ_010498

answers: central nervous system

answer: No

answer: No

answer: No

answer: No

answer: No

answer: 1

text: Multiple sclerosis is a disease that demyelinates the nerves of the central nervous system, leading to deteriorated bodily function. The underlying pathologies are mostly unknown and not understood. It is hypothesized by many that the demyelination in multiple sclerosis lesions leads to axonal transection and ultimately axonal degeneration. This axon death could potentially be the cause of the irreversible effects of multiple sclerosis. Using what is known about the axotomy response, doctors and researchers are looking to implement neuro-protective rehabilitation to patients suffering in the early stages of multiple sclerosis in order to prevent the disease from running its course and causing irreversible disability. These rehabilitations would include the utilization of trophic factors to aid in neuron survival and maintenance of synapsing function.

text: Multiple sclerosis

text: is a disease that

text: demyelinates

text: the nerves of the central nervous system, leading to deteriorated bodily function. The underlying pathologies are mostly unknown and not understood. It is hypothesized by many that the demyelination in multiple sclerosis lesions leads to axonal transection and ultimately axonal degeneration. This axon death could potentially be the cause of the irreversible effects of multiple sclerosis. Using what is known about the axotomy response, doctors and researchers are looking to implement neuro-protective rehabilitation to patients suffering in the early stages of multiple sclerosis in order to prevent the disease from running its course and causing irreversible disability. These rehabilitations would include the utilization of trophic factors to aid in neuron survival and maintenance of synapsing function.

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answers: Multiple sclerosis

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answers: Multiple sclerosis

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answers: Multiple sclerosis

answers: NDQ_010495

answers: immune-mediated

answers: NDQ_010496

answers: Multiple sclerosis

answers: NDQ_010497

answers: Multiple sclerosis

answers: NDQ_010498

answers: Multiple sclerosis

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: Neurology, psychiatry, neurosurgery, psychosurgery, anesthesiology and pain medicine, neuropathology, neuroradiology, ophthalmology, otolaryngology, clinical neurophysiology, addiction medicine, and sleep medicine are some medical specialties that specifically address the diseases of the nervous system. These terms also refer to clinical disciplines involving diagnosis and treatment of these diseases. Neurology works with diseases of the central and peripheral nervous systems, such as amyotrophic lateral sclerosis (ALS) and stroke, and their medical treatment. Psychiatry focuses on affective, behavioral, cognitive, and perceptual disorders. Anesthesiology focuses on perception of pain, and pharmacologic alteration of consciousness. Neuropathology focuses upon the classification and underlying pathogenic mechanisms of central and peripheral nervous system and muscle diseases, with an emphasis on morphologic, microscopic, and chemically observable alterations. Neurosurgery and psychosurgery work primarily with surgical treatment of diseases of the central and peripheral nervous systems. The boundaries between these specialties have been blurring recently as they are all influenced by basic research in neuroscience. Brain imaging also enables objective, biological insights into mental illness, which can lead to faster diagnosis, more accurate prognosis, and help assess patient progress over time.

text: Neurology

text: psychiatry

text: neurosurgery

text: psychosurgery

text: anesthesiology

text: and

text: pain medicine

text: neuropathology

text: neuroradiology

text: ophthalmology

text: otolaryngology

text: clinical neurophysiology

text: addiction medicine

text: , and

text: sleep medicine

text: are some medical specialties that specifically address the diseases of the nervous system. These terms also refer to clinical disciplines involving diagnosis and treatment of these diseases. Neurology works with diseases of the central and peripheral nervous systems, such as

text: amyotrophic lateral sclerosis

text: (ALS) and

text: stroke

text: , and their medical treatment. Psychiatry focuses on

text: affective

text: , behavioral,

text: cognitive

text: , and

text: perceptual

text: disorders. Anesthesiology focuses on perception of pain, and pharmacologic alteration of consciousness. Neuropathology focuses upon the classification and underlying pathogenic mechanisms of central and peripheral nervous system and muscle diseases, with an emphasis on morphologic, microscopic, and chemically observable alterations. Neurosurgery and psychosurgery work primarily with surgical treatment of diseases of the central and peripheral nervous systems. The boundaries between these specialties have been blurring recently as they are all influenced by

text: basic research

text: in neuroscience.

text: Brain imaging

text: also enables objective, biological insights into mental illness, which can lead to faster diagnosis, more accurate prognosis, and help assess patient progress over time.

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text: Insensitivity to pain may also result from abnormalities in the nervous system. This is usually the result of acquired damage to the nerves, such as spinal cord injury, diabetes mellitus (diabetic neuropathy), or leprosy in countries where that disease is prevalent. These individuals are at risk of tissue damage and infection due to undiscovered injuries. People with diabetes-related nerve damage, for instance, sustain poorly-healing foot ulcers as a result of decreased sensation.

text: Insensitivity to pain may also result from abnormalities in the

text: nervous system

text: . This is usually the result of

text: acquired

text: damage to the nerves, such as

text: spinal cord injury

text: diabetes mellitus

text: diabetic neuropathy

text: ), or

text: leprosy

text: in countries where that disease is prevalent. These individuals are at risk of tissue damage and infection due to undiscovered injuries. People with diabetes-related nerve damage, for instance, sustain poorly-healing foot ulcers as a result of decreased sensation.

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text: Children should not take medication with bismuth subsalicylate while recovering from influenza or chicken pox, as epidemiologic evidence points to an association between the use of salicylate-containing medications during certain viral infections and the onset of Reye's syndrome. For the same reason, it is typically recommended that nursing mothers not use medication containing bismuth subsalicylate because small amounts of the medication are excreted in breast milk and pose a theoretical risk of Reye's syndrome to nursing children.

text: Children should not take medication with bismuth subsalicylate while recovering from

text: influenza

text: or

text: chicken pox

text: , as epidemiologic evidence points to an association between the use of

text: salicylate

text: -containing medications during certain viral infections and the onset of

text: Reye's syndrome

text: . For the same reason, it is typically recommended that nursing mothers not use medication containing bismuth subsalicylate because small amounts of the medication are excreted in breast milk and pose a theoretical risk of Reye's syndrome to nursing children.

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text: In many English speaking countries neuropathology is considered a subfield of anatomical pathology. In contrast, there are a number of independent university chairs in neuropathology and even institutes of neuropathology in German speaking countries due to a different historical background.  A physician who specializes in neuropathology, usually by completing a fellowship after a residency in anatomical or general pathology, is called a neuropathologist. In day-to-day clinical practice, a neuropathologist is a consultant for other physicians.  If a disease of the nervous system is suspected, and the diagnosis cannot be made by less invasive methods, a biopsy of nervous tissue is taken and sent to the neuropathologist, who examines it using a microscope or certain molecular methods to make a definitive diagnosis.

text: In many English speaking countries neuropathology is considered a subfield of

text: anatomical pathology

text: . In contrast, there are a number of independent university chairs in neuropathology and even institutes of neuropathology in German speaking countries due to a different historical background.  A physician who specializes in neuropathology, usually by completing a fellowship after a residency in anatomical or general pathology, is called a neuropathologist. In day-to-day clinical practice, a neuropathologist is a consultant for other physicians.  If a disease of the nervous system is suspected, and the diagnosis cannot be made by less invasive methods, a

text: biopsy

text: of

text: nervous tissue

text: is taken and sent to the neuropathologist, who examines it using a microscope or certain molecular methods to make a definitive diagnosis.

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answers: disease of the nervous system

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answers: a biopsy of nervous tissue

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answers: neuropathology

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answers: immune system

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answers: neuropathology

answer: Yes

answer: Yes

answer: No

answer: Yes

answer: No

answer: 1

text: The initial evaluation of a patient with a newly diagnosed tumor of the nervous system is a critical step toward appropriate management and patient care. The most important portions of the initial evaluation are a detailed history and a thorough examination. This process serves to identify the extent and nature of neurological deficit, provides diagnostic clues, can help disclose a source of metastasis, or may identify a genetic process associated with a primary central nervous system tumor.

text: The initial evaluation of a patient with a newly diagnosed tumor of the nervous system is a critical step toward appropriate management and patient care. The most important portions of the initial evaluation are a detailed history and a thorough examination. This process serves to identify the extent and nature of neurological deficit, provides diagnostic clues, can help disclose a source of metastasis, or may identify a genetic process associated with a primary central nervous system tumor.

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text: Neurogenetics studies the role of genetics in the development and function of the nervous system. It considers neural characteristics as phenotypes (i.e. manifestations, measurable or not, of the genetic make-up of an individual), and is mainly based on the observation that the nervous systems of individuals, even of those belonging to the same species, may not be identical. As the name implies, it draws aspects from both the studies of neuroscience and genetics, focusing in particular how the genetic code an organism carries affects its expressed traits. Mutations in this genetic sequence can have a wide range of effects on the quality of life of the individual. Neurological diseases, behavior and personality are all aspects of man studied in the context of neurogenetics. The field of neurogenetics emerged in the mid to late 1900s with advances closely following advancements made in available technology. Currently neurogenetics is the center of much research utilizing the cutting edge of research techniques.

text: Neurogenetics studies the role of

text: genetics

text: in the development and function of the

text: nervous system

text: . It considers neural characteristics as

text: phenotype

text: s (i.e. manifestations, measurable or not, of the genetic make-up of an individual), and is mainly based on the observation that the nervous systems of individuals, even of those belonging to the same

text: species

text: , may not be identical. As the name implies, it draws aspects from both the studies of

text: neuroscience

text: and genetics, focusing in particular how the genetic code an organism carries affects its expressed

text: traits

text: Mutation

text: s in this genetic sequence can have a wide range of effects on the quality of life of the individual. Neurological diseases, behavior and personality are all aspects of man studied in the context of neurogenetics. The field of neurogenetics emerged in the mid to late 1900s with advances closely following advancements made in available technology. Currently neurogenetics is the center of much research utilizing the cutting edge of research techniques.

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text: Lymphocytic pleocytosis is an abnormal increase in the amount of lymphocytes in the cerebrospinal fluid (CSF). It is usually considered to be a sign of infection or inflammation within the nervous system, and is encountered in a number of neurological diseases, such as pseudomigraine, Susac's syndrome, and encephalitis. While lymphocytes make up roughly a quarter of all white blood cells (WBC) in the body, they are generally rare in the CSF. Under normal conditions, there are usually less than 5 white blood cells per µL of CSF.  In a pleocytic setting, the number of lymphocytes can jump to more than 1,000 cells per mm. Increases in lymphocyte count are often accompanied by an increase in cerebrospinal protein concentrations in addition to pleocytosis of other types of white blood cells.

text: Lymphocytic pleocytosis is an abnormal increase in the amount of

text: lymphocytes

text: in the

text: cerebrospinal fluid

text: (CSF). It is usually considered to be a sign of

text: infection

text: or inflammation within the

text: nervous system

text: , and is encountered in a number of

text: neurological diseases

text: , such as pseudomigraine, Susac's syndrome, and encephalitis. While lymphocytes make up roughly a quarter of all

text: white blood cells

text: (WBC) in the body, they are generally rare in the CSF. Under normal conditions, there are usually less than 5 white blood cells per µL of CSF.  In a pleocytic setting, the number of lymphocytes can jump to more than 1,000 cells per mm. Increases in lymphocyte count are often accompanied by an increase in cerebrospinal protein concentrations in addition to

text: pleocytosis

text: of other types of

text: white blood cells

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answer: Yes

answer: Yes

answer: Yes

answer: Yes

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answer: 1

text: Another commonly used neuromodulation treatment developed in the 1980s is deep brain stimulation, which may be used to help limit symptoms of movement disorder in Parkinson's disease, dystonia, or essential tremor. It was approved in 2010 in Europe for the treatment of certain types of severe epilepsy. DBS also has shown promise, although still in research, for medically intractable psychiatric syndromes of depression, obsessive compulsive disorders, intractable rage, dementia, and morbid obesity. It has also shown promise for Tourette syndrome, torticollis, and tardive dyskinesia. DBS therapy, unlike spinal cord stimulation, has a variety of central nervous system targets, depending on the target pathology. For Parkinson's disease central nervous system targets include the subthalamic nucleus, globus pallidus interna, and the ventral intermidus nucleus of the thalamus. Dystonias are often treated by implants targeting globus pallidus interna, or less often, parts of the ventral thalamic group. The anterior thalamus is the target for epilepsy.

text: Another commonly used neuromodulation treatment developed in the 1980s is

text: deep brain stimulation

text: , which may be used to help limit symptoms of movement disorder in

text: Parkinson's disease

text: dystonia

text: , or

text: essential tremor

text: . It was approved in 2010 in Europe for the treatment of certain types of severe epilepsy. DBS also has shown promise, although still in research, for medically intractable psychiatric syndromes of depression, obsessive compulsive disorders, intractable rage, dementia, and morbid obesity. It has also shown promise for Tourette syndrome, torticollis, and tardive dyskinesia. DBS therapy, unlike spinal cord stimulation, has a variety of central nervous system targets, depending on the target pathology. For Parkinson's disease central nervous system targets include the subthalamic nucleus, globus pallidus interna, and the ventral intermidus nucleus of the thalamus. Dystonias are often treated by implants targeting globus pallidus interna, or less often, parts of the ventral thalamic group. The anterior thalamus is the target for epilepsy.

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answers: DBS therapy, unlike spinal cord stimulation

answers: NDQ_010490

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answers: NDQ_010494

answers: dementia

answers: NDQ_010495

answers: Tourette syndrome

answers: NDQ_010496

answers: epilepsy

answers: NDQ_010497

answers: Parkinson's disease

answers: NDQ_010498

answers: epilepsy

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: The first report of bacterial infection underlying meningitis was by the Austrian bacteriologist Anton Weichselbaum, who in 1887 described the meningococcus. Mortality from meningitis was very high (over 90%) in early reports. In 1906, antiserum was produced in horses; this was developed further by the American scientist Simon Flexner and markedly decreased mortality from meningococcal disease. In 1944, penicillin was first reported to be effective in meningitis. The introduction in the late 20th century of Haemophilus vaccines led to a marked fall in cases of meningitis associated with this pathogen, and in 2002, evidence emerged that treatment with steroids could improve the prognosis of bacterial meningitis. World Meningitis Day is celebrated on the 24th of April each year.

text: The first report of bacterial infection underlying meningitis was by the Austrian bacteriologist

text: Anton Weichselbaum

text: , who in 1887 described the meningococcus. Mortality from meningitis was very high (over 90%) in early reports. In 1906,

text: antiserum

text: was produced in horses; this was developed further by the American scientist

text: Simon Flexner

text: and markedly decreased mortality from meningococcal disease. In 1944,

text: penicillin

text: was first reported to be effective in meningitis. The introduction in the late 20th century of

text: Haemophilus

text: vaccines led to a marked fall in cases of meningitis associated with this pathogen, and in 2002, evidence emerged that treatment with steroids could improve the prognosis of bacterial meningitis. World Meningitis Day is celebrated on the 24th of April each year.

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answers: NDQ_010489

answers: Meningitis

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answers: meningitis

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answers: meningitis

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answer: Yes

answer: Yes

answer: Yes

answer: Yes

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answer: 1

text: Like other neurons, α-MNs transmit signals as action potentials, rapid changes in electrical activity that propagate from the cell body to the end of the axon. To increase the speed at which action potentials travel, α-MN axons have large diameters and are heavily myelinated by both oligodendrocytes and Schwann cells. Oligodendrocytes myelinate the part of the α-MN axon that lies in the central nervous system (CNS), while Schwann cells myelinate the part that lies in the peripheral nervous system (PNS). The transition between the CNS and PNS occurs at the level of the pia mater, the innermost and most delicate layer of meningeal tissue surrounding components of the CNS.

text: Like other neurons, α-MNs transmit signals as

text: action potential

text: s, rapid changes in electrical activity that propagate from the

text: cell body

text: to the end of the

text: axon

text: . To increase the speed at which action potentials travel, α-MN axons have large diameters and are heavily

text: myelin

text: ated by both

text: oligodendrocyte

text: s and

text: Schwann cell

text: s. Oligodendrocytes myelinate the part of the α-MN axon that lies in the

text: central nervous system

text: (CNS), while Schwann cells myelinate the part that lies in the

text: peripheral nervous system

text: (PNS). The transition between the CNS and PNS occurs at the level of the

text: pia mater

text: , the innermost and most delicate layer of

text: meningeal tissue

text: surrounding components of the CNS.

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answers: It does not say

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answers: It does not say.

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answers: central nervous system

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answers: peripheral nervous system

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answers: central nervous system

answers: NDQ_010497

answers: central nervous system

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answers: central nervous system

answer: No

answer: No

answer: No

answer: No

answer: No

answer: 1

text: The brains of vertebrates are made of very soft tissue. Living brain tissue is pinkish on the outside and mostly white on the inside, with subtle variations in color. Vertebrate brains are surrounded by a system of connective tissue membranes called meninges that separate the skull from the brain. Blood vessels enter the central nervous system through holes in the meningeal layers. The cells in the blood vessel walls are joined tightly to one another, forming the blood–brain barrier, which blocks the passage of many toxins and pathogens (though at the same time blocking antibodies and some drugs, thereby presenting special challenges in treatment of diseases of the brain).

text: The brains of vertebrates are made of very soft tissue. Living brain tissue is pinkish on the outside and mostly white on the inside, with subtle variations in color. Vertebrate brains are surrounded by a system of

text: connective tissue

text: membrane

text: s called

text: meninges

text: that separate the skull from the brain.

text: Blood vessel

text: s enter the central nervous system through holes in the meningeal layers. The cells in the blood vessel walls are joined tightly to one another, forming the

text: blood–brain barrier

text: , which blocks the passage of many

text: toxin

text: s and

text: pathogen

text: s (though at the same time blocking

text: antibodies

text: and some drugs, thereby presenting special challenges in treatment of diseases of the brain).

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text: Dysautonomia may be due to inherited or degenerative neurologic diseases (primary dysautonomia) or it may occur due to injury of the autonomic nervous system from an acquired disorder (secondary dysautonomia). The most common causes of dysautonomia include:In the sympathetic nervous system predominant dysautonomia is common along with fibromyalgia, chronic fatigue syndrome, irritable bowel syndrome, and interstitial cystitis, raising the possibility that such dysautonomia could be their common clustering underlying pathogenesis.

text: Dysautonomia may be due to

text: inherited

text: or degenerative

text: neurologic diseases

text: (primary dysautonomia) or it may occur due to injury of the autonomic nervous system from an acquired disorder (secondary dysautonomia). The most common causes of dysautonomia include:In the

text: sympathetic nervous system

text: predominant dysautonomia is common along with

text: fibromyalgia

text: chronic fatigue syndrome

text: irritable bowel syndrome

text: , and

text: interstitial cystitis

text: , raising the possibility that such dysautonomia could be their common clustering underlying pathogenesis.

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answers: Dysautonomia

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answers: autonomic

answer: Yes

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answer: 1

text: Down syndrome (DS), caused by trisomy 21, is the most common birth defect associated with intellectual disability. Recently, the novel gene DSCAM has been identified in the DS critical region. DSCAM is predicted to be a transmembrane protein with a very high structural and sequence homology to the immunoglobulin (Ig) superfamily of cell adhesion molecules. It is expressed in the developing nervous system with the highest level of expression occurring in the fetal brain.  When this gene is over-expressed in the developing fetal central nervous system, it leads to Down syndrome. Diverse glycoproteins of cell surfaces and extracellular matrices, operationally termed as 'adhesion molecules' are important in the specification of cell interactions during development as well as maintenance and regeneration of the nervous system.

text: Down syndrome

text: (DS), caused by

text: trisomy 21

text: , is the most common birth defect associated with

text: intellectual disability

text: . Recently, the novel gene DSCAM has been identified in the DS critical region. DSCAM is predicted to be a

text: transmembrane protein

text: with a very high structural and sequence homology to the

text: immunoglobulin (Ig) superfamily

text: of

text: cell adhesion molecule

text: s. It is expressed in the developing nervous system with the highest level of expression occurring in the fetal brain.  When this gene is over-expressed in the developing fetal central nervous system, it leads to Down syndrome. Diverse

text: glycoprotein

text: s of cell surfaces and extracellular matrices, operationally termed as 'adhesion molecules' are important in the specification of cell interactions during development as well as maintenance and regeneration of the nervous system.

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text: Hoff wrote more than 500 papers on psychiatry and neurology, including ten books. In his scientific work, he focused on experimental encephalitis studies, postural reflexes, psychosomatic problems, the influence of hypnotic orders on gastric and intestinal function, the psycho-vegetative circuit, the influence of endocrine glands by psychological factors, and the function of the hypothalamus , The central nervous system of the metabolism and the endocrine glands, brainpathological phenomena, the function of the frontal, thalamic and cerebellum, time lapse phenomena, nervous vascular regulation, sleep studies, adrenal disturbances in infectious diseases, epilepsy, multiple sclerosis, psychopathy, The alcoholism.

text: Hoff wrote more than 500 papers on psychiatry and neurology, including ten books. In his scientific work, he focused on experimental encephalitis studies, postural reflexes, psychosomatic problems, the influence of hypnotic orders on gastric and intestinal function, the psycho-vegetative circuit, the influence of endocrine glands by psychological factors, and the function of the hypothalamus , The central nervous system of the metabolism and the endocrine glands, brainpathological phenomena, the function of the frontal, thalamic and cerebellum, time lapse phenomena, nervous vascular regulation, sleep studies, adrenal disturbances in infectious diseases, epilepsy, multiple sclerosis, psychopathy, The alcoholism.

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answer: 1

text: Fibrous scarring can also be a complication within the subarachnoid space.  It is notoriously difficult to detect and evaluate.  Prior to the development of magnetic resonance imaging, the only way to ascertain the presence of arachnoiditis was by opening the dura.   In the days of CT scanning and Pantopaque and later, Metrizamide myelography, the presence of arachnoiditis could be speculated based on radiographic findings.  Often, myelography prior to the introduction of Metrizamide was the cause of arachnoiditis.   It can also be caused by the long term pressure brought about with either a severe disc herniation or spinal stenosis.   The presence of both epidural scarring and arachnoiditis in the same patient are probably quite common.  Arachnoiditis is a broad term denoting inflammation of the meninges and subarachnoid space. A variety of etiologies exist, including infectious, inflammatory, and neoplastic processes.Infectious etiologies include bacterial, viral, fungal, and parasitic agents. Noninfectious inflammatory processes include surgery, intrathecal hemorrhage, and the administration of intrathecal (inside the dural canal) agents such as myelographic contrast media, anesthetics (e.g. chloroprocaine), and steroids (e.g. Depo-Medrol, Kenalog). Lately iatrogenic arachnoiditis has been attributed to misplaced Epidural Steroid Injection therapy when accidentally administered intrathecally. The preservatives and suspension agents found in all steroid injectates, which aren't indicated for epidural administration by the U.S. Food & Drug Administration due to reports of severe adverse events including arachnoiditis, paralysis and death, have now been directly linked to the onset of the disease following the initial stage of chemical meningitis.  Neoplasia includes the hematogenous spread of systemic tumors, such as breast and lung carcinoma, melanoma, and non-Hodgkin lymphoma. Neoplasia also includes direct seeding of the cerebrospinal fluid (CSF) from primary central nervous system (CNS) tumors such as glioblastoma multiforme, medulloblastoma, ependymoma, and choroid plexus carcinoma.    Strictly speaking, the most common cause of arachnoiditis in failed back syndrome is not infectious or from cancer.  It is due to non-specific scarring secondary to the surgery or the underlying pathology.

text: Fibrous scarring can also be a complication within the subarachnoid space.  It is notoriously difficult to detect and evaluate.  Prior to the development of

text: magnetic resonance imaging

text: , the only way to ascertain the presence of

text: arachnoiditis

text: was by opening the dura.   In the days of

text: CT

text: scanning and

text: Pantopaque

text: and later,

text: Metrizamide

text: myelography, the presence of arachnoiditis could be speculated based on radiographic findings.  Often, myelography prior to the introduction of Metrizamide was the cause of arachnoiditis.   It can also be caused by the long term pressure brought about with either a severe disc herniation or spinal stenosis.   The presence of both epidural scarring and arachnoiditis in the same patient are probably quite common.  Arachnoiditis is a broad term denoting inflammation of the meninges and subarachnoid space. A variety of etiologies exist, including infectious, inflammatory, and neoplastic processes.Infectious etiologies include bacterial, viral, fungal, and parasitic agents. Noninfectious inflammatory processes include surgery, intrathecal hemorrhage, and the administration of intrathecal (inside the dural canal) agents such as myelographic contrast media, anesthetics (e.g.

text: chloroprocaine

text: ), and steroids (e.g. Depo-Medrol, Kenalog). Lately iatrogenic arachnoiditis has been attributed to misplaced Epidural Steroid Injection therapy when accidentally administered intrathecally. The preservatives and suspension agents found in all steroid injectates, which aren't indicated for epidural administration by the U.S. Food & Drug Administration due to reports of severe adverse events including arachnoiditis, paralysis and death, have now been directly linked to the onset of the disease following the initial stage of chemical meningitis.  Neoplasia includes the hematogenous spread of systemic tumors, such as breast and lung carcinoma, melanoma, and non-Hodgkin lymphoma. Neoplasia also includes direct seeding of the cerebrospinal fluid (CSF) from primary central nervous system (CNS) tumors such as

text: glioblastoma multiforme

text: medulloblastoma

text: ependymoma

text: , and

text: choroid plexus carcinoma

text: .    Strictly speaking, the most common cause of arachnoiditis in failed back syndrome is not infectious or from cancer.  It is due to non-specific scarring secondary to the surgery or the underlying pathology.

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answers: Arachnoiditis

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text: Human nervous system consists of the central nervous system (CNS), which comprises the brain and spinal cord, and the peripheral nervous system (PNS) comprising the nerve fibers that branch off from the spinal cord to all parts of the body. Both parts of the nervous system are actively involved in communicating signals between various parts of the body to ensure the smooth and efficient transfer of information that controls and coordinates the movement of muscles, and regulates organ functions. Neurons, which form the elemental unit of the nervous system, receive messages from their dendrites, relay the information as an electrical signal down the axon and releases chemical messengers known as neurotransmitters, thus converting the electrical signal into a chemical signal.

text: Human nervous system consists of the

text: central nervous system

text: (CNS), which comprises the brain and spinal cord, and the

text: peripheral nervous system

text: (PNS) comprising the nerve fibers that branch off from the spinal cord to all parts of the body. Both parts of the nervous system are actively involved in communicating signals between various parts of the body to ensure the smooth and efficient transfer of information that controls and coordinates the movement of muscles, and regulates organ functions.

text: Neurons

text: , which form the elemental unit of the nervous system, receive messages from their

text: dendrite

text: s, relay the information as an electrical signal down the

text: axon

text: and releases chemical messengers known as

text: neurotransmitter

text: s, thus converting the electrical signal into a chemical signal.

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answers: peripheral nervous system

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text: Solomon’s research is on emerging brain infections, especially encephalitis (inflammation and swelling of the brain, usually caused by a virus). He is an expert on Japanese encephalitis, an emerging infectious disease that is a zoonosis   spread from animals to humans  by mosquitoes.    He showed that Japanese encephalitis virus can cause an illness with leg paralysis which could be confused for polio. He also highlighted the importance of dengue, a related mosquito-borne virus, as a cause of neurological disease. He works on the origins, evolution and spread of Japanese encephalitis. He has played a major role in the global campaign to control Japanese encephalitis through vaccination. This included developing the Liverpool Outcome Score for quantifying the disability caused by Japanese encephalitis, and helping produce the WHO Surveillance Standards for detecting the disease. He is also an expert on enterovirus 71, which causes hand foot and mouth disease and encephalitis. He works on improving the diagnosis, better understanding the disease mechanisms, and strengthening clinical management.

text: Solomon’s research is on emerging brain infections, especially

text: encephalitis

text: (inflammation and swelling of the brain, usually caused by a virus). He is an expert on

text: Japanese encephalitis

text: , an

text: emerging infectious disease

text: that is a

text: zoonosis

text: spread from animals to humans  by mosquitoes.    He showed that Japanese encephalitis virus can cause an illness with leg paralysis which could be confused for polio. He also highlighted the importance of

text: dengue

text: , a related mosquito-borne virus, as a cause of neurological disease. He works on the origins, evolution and spread of Japanese encephalitis. He has played a major role in the global campaign to control Japanese encephalitis through vaccination. This included developing the Liverpool Outcome Score for quantifying the disability caused by Japanese encephalitis, and helping produce the WHO Surveillance Standards for detecting the disease. He is also an expert on

text: enterovirus 71

text: , which causes hand foot and mouth disease and encephalitis. He works on improving the diagnosis, better understanding the disease mechanisms, and strengthening clinical management.

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answers: neurological disease

answers: NDQ_010490

answers: brain infections

answers: NDQ_010494

answers: polio

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answers: polio

answers: NDQ_010496

answers: polio

answers: NDQ_010497

answers: encephalitis

answers: NDQ_010498

answers: encephalitis

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: Muckle–Wells syndrome (MWS), also known as  urticaria-deafness-amyloidosis syndrome (UDA), is a rare autosomal dominant disease which causes sensorineural deafness, recurrent hives, and can lead to amyloidosis. Individuals with MWS often have episodic fever, chills, and joint pain. As a result, MWS is considered a type of periodic fever syndrome. MWS is caused by a defect in the CIAS1 gene which creates the protein cryopyrin. MWS is closely related to two other syndromes, familial cold urticaria and neonatal onset multisystem inflammatory disease—in fact, all three are related to mutations in the same gene and subsumed under the term cryopyrin-associated periodic syndromes (CAPS).

text: Muckle–Wells syndrome (MWS), also known as  urticaria-deafness-amyloidosis syndrome (UDA), is a

text: rare

text: autosomal dominant

text: disease which causes

text: sensorineural

text: deafness

text: , recurrent

text: hives

text: , and can lead to

text: amyloidosis

text: . Individuals with MWS often have episodic fever, chills, and

text: joint pain

text: . As a result, MWS is considered a type of

text: periodic fever syndrome

text: . MWS is caused by a defect in the

text: gene which creates the protein

text: cryopyrin

text: . MWS is closely related to two other syndromes,

text: familial cold urticaria

text: and

entity_name: Neonatal onset multisystem inflammatory disease

text: neonatal onset multisystem inflammatory disease

text: —in fact, all three are related to mutations in the same gene and subsumed under the term

text: cryopyrin-associated periodic syndrome

text: s (CAPS).

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answers: Muckle–Wells syndrome (MWS)

answers: NDQ_010490

answers: Muckle–Wells syndrome

answers: NDQ_010494

answers: Muckle–Wells syndrome

answers: NDQ_010495

answers: inflammatory disease

answers: NDQ_010496

answers: Muckle–Wells syndrome

answers: NDQ_010497

answers: sensorineural deafness

answers: NDQ_010498

answers: Muckle–Wells syndrome

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: No

answer: 1

text: Semliki Forest virus (SFV) is a virus that naturally infects cells of the central nervous system and causes encephalitis. A genetically engineered form has been pre-clinically tested as an oncolytic virus against the severe brain tumour type glioblastoma. The SFV was genetically modified with microRNA target sequences so that it only replicated in brain tumour cells and not in normal brain cells. The modified virus reduced tumour growth and prolonged survival of mice with brain tumours. The modified virus was also found to efficiently kill human glioblastoma tumour cell lines.

text: Semliki Forest virus

text: (SFV) is a virus that naturally infects cells of the central nervous system and causes

text: encephalitis

text: . A

text: genetically engineered

text: form has been

text: pre-clinically tested

text: as an oncolytic virus against the severe brain tumour type

text: glioblastoma

text: . The SFV was genetically modified with

text: microRNA

text: target sequences so that it only replicated in brain tumour cells and not in normal brain cells. The modified virus reduced tumour growth and prolonged survival of mice with brain tumours. The modified virus was also found to efficiently kill human glioblastoma tumour cell lines.

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answers: central nervous system

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answer: 1

text: After a typical human infection by bite, the virus enters the peripheral nervous system. It then travels along the nerves towards the central nervous system. During this phase, the virus cannot be easily detected within the host, and vaccination may still confer cell-mediated immunity to prevent symptomatic rabies. Once the virus reaches the brain, it rapidly causes encephalitis and symptoms appear. This is called the "prodromal" phase and at this time, treatment is usually unsuccessful. Rabies may also inflame the spinal cord producing myelitis.

text: After a typical human infection by bite, the virus enters the

text: peripheral nervous system

text: . It then travels along the

text: nerve

text: s towards the

text: central nervous system

text: . During this phase, the virus cannot be easily detected within the host, and vaccination may still confer cell-mediated immunity to prevent symptomatic rabies. Once the virus reaches the

text: brain

text: , it rapidly causes

text: encephalitis

text: and symptoms appear. This is called the "prodromal" phase and at this time, treatment is usually unsuccessful. Rabies may also inflame the

text: spinal cord

text: producing

text: myelitis

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answer: 1

text: The optic nerve is the second of twelve paired cranial nerves and is technically part of the central nervous system, rather than the peripheral nervous system because it is derived from an out-pouching of the diencephalon (optic stalks) during embryonic development. As a consequence, the fibers of the optic nerve are covered with myelin produced by oligodendrocytes, rather than Schwann cells of the peripheral nervous system, and are encased within the meninges. Peripheral neuropathies like Guillain–Barré syndrome do not affect the optic nerve. However, most typically the optic nerve is grouped with the other eleven cranial nerves and considered to be part of the peripheral nervous system.

text: The optic nerve is the second of twelve paired

text: cranial nerve

text: s and is technically part of the

text: central nervous system

text: , rather than the

text: peripheral nervous system

text: because it is derived from an out-pouching of the

text: diencephalon

text: optic stalks

text: ) during embryonic development. As a consequence, the fibers of the optic nerve are covered with myelin produced by

text: oligodendrocyte

text: s, rather than

text: Schwann cell

text: s of the

text: peripheral nervous system

text: , and are encased within the

text: meninges

text: . Peripheral neuropathies like

text: Guillain–Barré syndrome

text: do not affect the optic nerve. However, most typically the optic nerve is grouped with the other eleven cranial nerves and considered to be part of the peripheral nervous system.

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answers: Peripheral neuropathies

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answers: Peripheral neuropathies

answer: No

answer: Yes

answer: No

answer: No

answer: No

answer: 1

text: Neuroinflammation is widely regarded as chronic, as opposed to acute, inflammation of the central nervous system. Acute inflammation usually follows injury to the central nervous system immediately, and is characterized by inflammatory molecules, endothelial cell activation, platelet deposition, and tissue edema.  Chronic inflammation is the sustained activation of glial cells and recruitment of other immune cells into the brain. It is chronic inflammation that is typically associated with neurodegenerative diseases. Common causes of chronic neuroinflammation include:

text: Neuroinflammation is widely regarded as chronic, as opposed to acute, inflammation of the

text: central nervous system

text: . Acute inflammation usually follows injury to the central nervous system immediately, and is characterized by inflammatory molecules,

text: endothelial cell

text: activation,

text: platelet

text: deposition, and tissue

text: edema

text: .  Chronic inflammation is the sustained activation of glial cells and recruitment of other immune cells into the brain. It is chronic inflammation that is typically associated with

text: neurodegenerative diseases

text: . Common causes of chronic neuroinflammation include:

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answers: 1: The answer has limited relevance and completeness, with substantial shortcomings.

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answers: neurodegenerative diseases

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answers: neurodegenerative diseases

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answers: neurodegenerative diseases

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answers: neurodegenerative diseases

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: No

answer: 1

text: Sandhoff disease, also known as Sandhoff-Jatzkewitz disease, variant 0 of GM2-Gangliosidosis or Hexosaminidase A and B deficiency, is a lysosomal genetic, lipid storage disorder caused by the inherited deficiency to create functional beta-hexosaminidases A and B. These catabolic enzymes are needed to degrade the neuronal membrane components, ganglioside GM2, its derivative GA2, the glycolipid globoside in visceral tissues, and some oligosaccharides. Accumulation of these metabolites leads to a progressive destruction of the central nervous system and eventually to death. The rare autosomal recessive neurodegenerative disorder is clinically almost indistinguishable from Tay-Sachs disease, another genetic disorder that disrupts beta-hexosaminidases A and S.  There are three subsets of Sandhoff disease based on when first symptoms appear: classic infantile, juvenile and adult late onset.

text: Sandhoff disease, also known as Sandhoff-Jatzkewitz disease, variant 0 of GM2-Gangliosidosis or Hexosaminidase A and B deficiency, is a lysosomal genetic,

text: lipid storage disorder

text: caused by the inherited deficiency to create functional beta-hexosaminidases A and B. These catabolic enzymes are needed to degrade the neuronal membrane components, ganglioside GM2, its derivative GA2, the glycolipid globoside in visceral tissues, and some oligosaccharides. Accumulation of these metabolites leads to a progressive destruction of the central nervous system and eventually to death. The rare autosomal recessive neurodegenerative disorder is clinically almost indistinguishable from

text: Tay-Sachs disease

text: , another genetic disorder that disrupts beta-hexosaminidases A and S.  There are three subsets of Sandhoff disease based on when first symptoms appear: classic infantile, juvenile and adult late onset.

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answer: Yes

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text: A few years after his retirement he was diagnosed with a degenerative syndrome of the central nervous system, later revealed to be transverse myelitis, which causes damage to the spine, and forced him into a wheelchair. Later, his former Springbok teammate, Joost van der Westhuizen developed a degenerative nervous disease, amyotrophic lateral sclerosis.

text: A few years after his retirement he was diagnosed with a degenerative syndrome of the central nervous system, later revealed to be

text: transverse myelitis

text: , which causes damage to the spine, and forced him into a wheelchair. Later, his former Springbok teammate,

text: Joost van der Westhuizen

text: developed a degenerative nervous disease,

text: amyotrophic lateral sclerosis

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answers: transverse myelitis

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answers: degenerative syndrome of the central nervous system

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answers: transverse myelitis

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answers: transverse myelitis

answer: Yes

answer: Yes

answer: Yes

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answer: 1

text: Venezuelan equine encephalitis virus is a mosquito-borne viral pathogen that causes Venezuelan equine encephalitis or encephalomyelitis (VEE). VEE can affect all equine species, such as horses, donkeys, and zebras. After infection, equines may suddenly die or show progressive central nervous system disorders. Humans also can contract this disease. Healthy adults who become infected by the virus may experience flu-like symptoms, such as high fevers and headaches. People with weakened immune systems and the young and the elderly can become severely ill or die from this disease.

text: Venezuelan equine encephalitis virus is a

text: mosquito

text: -borne

text: viral

text: pathogen that causes Venezuelan equine encephalitis or encephalomyelitis (VEE). VEE can affect all equine species, such as

text: horse

text: s,

text: donkey

text: s, and

text: zebra

text: s. After infection, equines may suddenly die or show progressive

text: central nervous system

text: disorders. Humans also can contract this disease. Healthy adults who become infected by the virus may experience

text: flu

text: -like symptoms, such as high fevers and headaches. People with weakened immune systems and the young and the elderly can become severely ill or die from this disease.

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text: In adults, the disease is more severe, though the incidence is much less common. Infection in adults is associated with greater morbidity and mortality due to pneumonia (either direct viral pneumonia or secondary bacterial pneumonia), bronchitis (either viral bronchitis or secondary bacterial bronchitis), hepatitis, and encephalitis. In particular, up to 10% of pregnant women with chickenpox develop pneumonia, the severity of which increases with onset later in gestation. In England and Wales, 75% of deaths due to chickenpox are in adults. Inflammation of the brain, or encephalitis, can occur in immunocompromised individuals, although the risk is higher with herpes zoster. Necrotizing fasciitis is also a rare complication.

text: In adults, the disease is more severe, though the incidence is much less common. Infection in adults is associated with greater morbidity and mortality due to

text: pneumonia

text: (either direct

text: viral pneumonia

text: or secondary

text: bacterial pneumonia

text: bronchitis

text: (either viral bronchitis or secondary bacterial bronchitis),

text: hepatitis

text: , and

text: encephalitis

text: . In particular, up to 10% of pregnant women with chickenpox develop pneumonia, the severity of which increases with onset later in gestation. In England and Wales, 75% of deaths due to chickenpox are in adults. Inflammation of the brain, or

text: encephalitis

text: , can occur in immunocompromised individuals, although the risk is higher with

text: herpes zoster

text: Necrotizing fasciitis

text: is also a rare complication.

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text: Infants that are infected by the protozoanToxoplasma gondii in utero can be born with chorioretinitis or ocular toxoplasmosis. Globally, it is the most common cause of infections of the back of the eye. (posterior segment). The most common sign is decreased vision in one eye. Other signs and symptoms may appear after the neonatal period and include: miscarriage, stillbirth, chorioretinitis development later in life, intracranial calcification hydrocephalus or central nervous system abnormalities.

text: Infants that are infected by the protozoan

text: Toxoplasma gondii

text: in utero can be born with

text: chorioretinitis

text: or ocular toxoplasmosis. Globally, it is the most common cause of infections of the back of the eye. (posterior segment). The most common sign is decreased vision in one eye. Other signs and symptoms may appear after the neonatal period and include: miscarriage, stillbirth, chorioretinitis development later in life, intracranial calcification hydrocephalus or central nervous system abnormalities.

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answer: Yes

answer: No

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text: Although the ANS is also known as the visceral nervous system, the ANS is only connected with the motor side. Most autonomous functions are involuntary but they can often work in conjunction with the somatic nervous system which provides voluntary control.

text: Although the ANS is also known as the visceral nervous system, the ANS is only connected with the motor side. Most autonomous functions are involuntary but they can often work in conjunction with the

text: somatic nervous system

text: which provides voluntary control.

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answers: Most autonomous functions are involuntary

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answers: Visceral nervous system

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answers: visceral

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answers: autoimmune

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answers: visceral

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answers: Visceral

answer: No

answer: No

answer: No

answer: No

answer: No

answer: 1

text: Infections most commonly associated with IRIS include Mycobacterium tuberculosis and cryptococcal meningitis. Persons living with AIDS are more at risk for IRIS if they are starting  for the first time, or if they have recently been treated for an opportunistic infection (OI). It is generally advised that when patients have low initial CD4 T cell count and opportunistic infection at the time of their HIV diagnosis, they receive treatment to control the opportunistic infections before HAART is initiated approximately two weeks later. This is true for most OIs, except for OIs involving the central nervous system.

text: Infections most commonly associated with IRIS include

text: Mycobacterium tuberculosis

text: and

text: cryptococcal meningitis

text: . Persons living with AIDS are more at risk for IRIS if they are starting  for the first time, or if they have recently been treated for an

text: opportunistic infection

text: (OI). It is generally advised that when patients have low initial CD4 T cell count and opportunistic infection at the time of their HIV diagnosis, they receive treatment to control the opportunistic infections before HAART is initiated approximately two weeks later. This is true for most OIs, except for OIs involving the

text: central nervous system

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text: Meningiomas are a diverse set of tumors arising from the meninges, the membranous layers surrounding the central nervous system. Many meningiomas produce no symptoms throughout a person's life, and if discovered, require no treatment other than periodic observation. Typically, symptomatic meningiomas are treated with either radiosurgery or conventional surgery.

text: Meningiomas are a diverse set of tumors arising from the

text: meninges

text: , the membranous layers surrounding the

text: central nervous system

text: . Many meningiomas produce no symptoms throughout a person's life, and if discovered, require no treatment other than periodic observation. Typically, symptomatic meningiomas are treated with either

text: radiosurgery

text: or

text: conventional surgery

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answer: Yes

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answer: 1

text: The specific causes of neurological problems vary, but can include genetic disorders, congenital abnormalities or disorders, infections, lifestyle or environmental health problems including malnutrition, and brain injury, spinal cord injury or nerve injury. The problem may start in another body system that interacts with the nervous system. For example, cerebrovascular disorders involve brain injury due to problems with the blood vessels (cardiovascular system) supplying the brain; autoimmune disorders involve damage caused by the body's own immune system; lysosomal storage diseases such as Niemann-Pick disease can lead to neurological deterioration.

text: The specific causes of neurological problems vary, but can include

text: genetic disorder

text: s,

text: congenital abnormalities

text: or

text: disorders

text: infections

text: lifestyle

text: or

text: environmental health

text: problems including

text: malnutrition

text: , and

text: brain injury

text: spinal cord injury

text: or

text: nerve injury

text: . The problem may start in another

text: body system

text: that interacts with the nervous system. For example,

text: cerebrovascular disorders

text: involve brain injury due to problems with the

text: blood vessels

text: cardiovascular system

text: ) supplying the brain;

text: autoimmune

text: disorders involve damage caused by the body's own

text: immune system

text: lysosomal storage diseases

text: such as

text: Niemann-Pick disease

text: can lead to neurological deterioration.

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answers: neurological problems

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answers: Niemann-Pick

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answer: Yes

answer: Yes

answer: Yes

answer: Yes

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text: Neurodegenerative diseases are a heterogenous group of complex disorders linked by the degeneration of neurons in either the peripheral nervous system or the central nervous system. Their underlying causes are extremely variable and complicated by various genetic and/or environmental factors. These diseases cause progressive deterioration of the neuron resulting in decreased signal transduction and in some cases even neuronal death. Peripheral nervous system (PNS) diseases may be further categorized by the type of nerve cell (motor, sensory, or both) affected by the disorder. Effective treatment of these diseases is often prevented by lack of understanding of the underlying molecular and genetic pathology. Epigenetic therapy is being investigated as a method of correcting the expression levels of misregulated genes in neurodegenerative diseases.

text: Neurodegenerative disease

text: s are a heterogenous group of complex disorders linked by the degeneration of

text: neuron

text: s in either the

text: peripheral nervous system

text: or the

text: central nervous system

text: . Their underlying causes are extremely variable and complicated by various genetic and/or environmental factors. These diseases cause progressive deterioration of the neuron resulting in decreased

text: signal transduction

text: and in some cases even neuronal death. Peripheral nervous system (PNS) diseases may be further categorized by the type of nerve cell (

text: motor

text: sensory

text: , or both) affected by the disorder. Effective treatment of these diseases is often prevented by lack of understanding of the underlying molecular and genetic pathology.

text: Epigenetic therapy

text: is being investigated as a method of correcting the expression levels of misregulated genes in neurodegenerative diseases.

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answer: Yes

answer: Yes

answer: Yes

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answer: Yes

answer: 1

text: Meningitis can be diagnosed after death has occurred. The findings from a post mortem are usually a widespread inflammation of the pia mater and arachnoid layers of the meninges. Neutrophil granulocytes tend to have migrated to the cerebrospinal fluid and the base of the brain, along with cranial nerves and the spinal cord, may be surrounded with pus – as may the meningeal vessels.

text: Meningitis can be diagnosed after death has occurred. The findings from a

text: post mortem

text: are usually a widespread inflammation of the

text: pia mater

text: and

text: arachnoid

text: layers of the meninges.

text: Neutrophil granulocyte

text: s tend to have migrated to the cerebrospinal fluid and the base of the brain, along with

text: cranial nerve

text: s and the

text: spinal cord

text: , may be surrounded with

text: pus

text: – as may the meningeal vessels.

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answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: Glial scar formation is induced following damage to the nervous system. In the central nervous system, this glial scar formation significantly inhibits nerve regeneration, which leads to a loss of function. Several families of molecules are released that promote and drive glial scar formation. For instance, transforming growth factors B-1 and -2, interleukins, and cytokines play a role in the initiation of scar formation. The accumulation of reactive astrocytes at the site of injury and the up regulation of molecules that are inhibitory for neurite outgrowth contribute to the failure of neuroregeneration. The up-regulated molecules alter the composition of the extracellular matrix in a way that has been shown to inhibit neurite outgrowth extension. This scar formation involves several cell types and families of molecules.

text: Glial scar formation is induced following damage to the nervous system. In the central nervous system, this glial scar formation significantly inhibits nerve regeneration, which leads to a loss of function. Several families of molecules are released that promote and drive glial scar formation. For instance, transforming growth factors B-1 and -2, interleukins, and cytokines play a role in the initiation of scar formation. The accumulation of reactive astrocytes at the site of injury and the up regulation of molecules that are inhibitory for neurite outgrowth contribute to the failure of neuroregeneration. The up-regulated molecules alter the composition of the extracellular matrix in a way that has been shown to inhibit neurite outgrowth extension. This scar formation involves several cell types and families of molecules.

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answers: central nervous system

answer: Yes

answer: Yes

answer: Yes

answer: No

answer: Yes

answer: 1

text: If this wall of protection was to break down, then the infection could become more widespread. This property may be explained by looking at the location and arrangement of the SGCs, as they are centered on the neurons, allowing them to protect the neurons. It has also been proposed that SGCs may have a job in ridding the ganglia of the virus and in protecting and repairing the nervous system after the virus has left the dormant stage.

text: If this wall of protection was to break down, then the infection could become more widespread. This property may be explained by looking at the location and arrangement of the SGCs, as they are centered on the neurons, allowing them to protect the neurons. It has also been proposed that SGCs may have a job in ridding the ganglia of the virus and in protecting and repairing the nervous system after the virus has left the dormant stage.

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answers: 2: The answer has limited relevance and completeness, with significant gaps or inaccuracy.

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answers: The infection could become more widespread

answers: NDQ_010490

answers: SGCs

answers: NDQ_010494

answers: Alzheimer's

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answers: infection

answers: NDQ_010496

answers: the nervous system

answers: NDQ_010497

answers: Alzheimer's disease

answers: NDQ_010498

answers: the virus

answer: No

answer: No

answer: No

answer: No

answer: No

answer: 1

text: Mutations in human PHOX2B cause a rare disease of the visceral nervous system (dysautonomia): congenital central hypoventilation syndrome (associated with respiratory arrests during sleep and, occasionally, wakefulness), Hirschsprung's disease (partial agenesis of the enteric nervous system), ROHHAD, and tumours of the sympathetic ganglia.In most people, Exon 3 of the gene contains a sequence of 20 polyalanine repeats. An increase in the number of repeats is associated with congenital central hypoventilation syndrome. There may also be other pathogenic mutations further along the gene.

text: Mutations in human PHOX2B cause a rare disease of the visceral nervous system (dysautonomia):

text: congenital central hypoventilation syndrome

text: (associated with respiratory arrests during sleep and, occasionally, wakefulness),

text: Hirschsprung's disease

text: (partial agenesis of the

text: enteric nervous system

text: ROHHAD

text: , and tumours of the

text: sympathetic ganglia

text: .In most people, Exon 3 of the gene contains a sequence of 20 polyalanine repeats. An increase in the number of repeats is associated with congenital central hypoventilation syndrome. There may also be other pathogenic mutations further along the gene.

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answers: Hirschsprung's disease

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answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: The most susceptible individuals are elderly frail individuals, or persons who are dehydrated from hot environments or inadequate fluid intake. More serious orthostatic hypotension is often the result of certain commonly prescribed medications such as diuretics, β-adrenergic blockers, other anti-hypertensives (including vasodilators), and nitroglycerin. In a small percentage of cases, the cause of orthostatic hypotensive faints is structural damage to the autonomic nervous system due to systemic diseases (e.g., amyloidosis or diabetes) or in neurological diseases (e.g., Parkinson's disease).

text: The most susceptible individuals are elderly frail individuals, or persons who are dehydrated from hot environments or inadequate fluid intake. More serious orthostatic hypotension is often the result of certain commonly prescribed medications such as diuretics,

text: β-adrenergic blockers

text: , other anti-hypertensives (including vasodilators), and

text: nitroglycerin

text: . In a small percentage of cases, the cause of orthostatic hypotensive faints is structural damage to the

text: autonomic nervous system

text: due to systemic diseases (e.g.,

text: amyloidosis

text: or diabetes) or in neurological diseases (e.g., Parkinson's disease).

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answers: amyloidosis

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answers: Parkinson's disease

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answers: Parkinson's disease

answers: NDQ_010498

answers: neurological diseases

answer: Yes

answer: Yes

answer: No

answer: Yes

answer: No

answer: 1

text: Functional weakness is weakness of an arm or leg due to the nervous system not working properly. It is not caused by damage or disease of the nervous system. Patients with functional weakness experience symptoms of limb weakness which can be disabling and frightening such as problems walking or a ‘heaviness’ down one side, dropping things or a feeling that a limb just doesn’t feel normal or ‘part of them’.  Functional weakness may also be described as 'dissociative motor disorder' and less helpfully as "conversion disorder."

text: Functional weakness is weakness of an arm or leg due to the nervous system not working properly. It is not caused by damage or disease of the nervous system. Patients with functional weakness experience symptoms of limb weakness which can be disabling and frightening such as problems walking or a ‘heaviness’ down one side, dropping things or a feeling that a limb just doesn’t feel normal or ‘part of them’.  Functional weakness may also be described as 'dissociative motor disorder' and less helpfully as "

text: conversion disorder

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answers: Alzheimer's disease

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answers: Functional weakness

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answers: The nervous system not working properly

answer: No

answer: No

answer: No

answer: No

answer: No

answer: 1

text: The disease used to be classified as a "neurocristopathy", or disease of the neural crest because part of the autonomic nervous system (such as sympathetic ganglia) derives from the neural crest. However, this denomination is no longer favored because essential neurons of the autonomic nervous system, including those that underlie the defining symptom of the disease (respiratory arrests), are derived from the neural tube (the medulla), not from the neural crest, although such mixed embryological origins are also true for most other neurocristopathies.

text: The disease used to be classified as a "

text: neurocristopathy

text: ", or disease of the

text: neural crest

text: because part of the

text: autonomic nervous system

text: (such as sympathetic ganglia) derives from the neural crest. However, this denomination is no longer favored because essential neurons of the autonomic nervous system, including those that underlie the defining symptom of the disease (respiratory arrests), are derived from the

text: neural tube

text: (the

text: medulla

text: ), not from the neural crest, although such mixed embryological origins are also true for most other neurocristopathies.

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answers: neurocristopathy

answers: NDQ_010490

answers: neurocristopathies

answers: NDQ_010494

answers: autonomic nervous system

answers: NDQ_010495

answers: neurocristopathies

answers: NDQ_010496

answers: respiratory arrests

answers: NDQ_010497

answers: neurocristopathies

answers: NDQ_010498

answers: autonomic nervous system

answer: Yes

answer: Yes

answer: No

answer: Yes

answer: Yes

answer: 1

text: In vertebrates, the enteric nervous system  includes efferent neurons, afferent neurons, and interneurons, all of which make the enteric nervous system capable of carrying reflexes in the absence of CNS input.  The sensory neurons report on mechanical and chemical conditions.  Through intestinal muscles, the motor neurons control peristalsis and churning of intestinal contents.  Other neurons control the secretion of enzymes.  The enteric nervous system also makes use of more than 30 neurotransmitters, most of which are identical to the ones found in CNS, such as acetylcholine, dopamine, and serotonin. More than 90% of the body's serotonin lies in the gut, as well as about 50% of the body's dopamine and the dual function of these neurotransmitters is an active part of gut-brain research.

text: In vertebrates, the enteric nervous system  includes

text: efferent neurons

text: afferent neuron

text: s, and

text: interneurons

text: , all of which make the enteric nervous system capable of carrying reflexes in the absence of CNS input.  The sensory neurons report on mechanical and chemical conditions.  Through intestinal muscles, the motor neurons control

text: peristalsis

text: and churning of intestinal contents.  Other neurons control the secretion of

text: enzymes

text: .  The enteric nervous system also makes use of more than 30

text: neurotransmitters

text: , most of which are identical to the ones found in CNS, such as

text: acetylcholine

text: dopamine

text: , and

text: serotonin

text: . More than 90% of the body's serotonin lies in the gut, as well as about 50% of the body's dopamine and the dual function of these neurotransmitters is an active part of gut-brain research.

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text: Some parasites alter host behavior by infecting neurons in the host's central nervous system. The host's central nervous system responds to the parasite as it would to any other infection. The hallmarks of such response include local inflammation and the release of chemicals such as cytokine. The immune response itself is responsible for induced behavioral changes in many cases of parasitic infection. Parasites that are known to induce behavioral changes through central nervous system inflammation in their hosts include Toxoplasma gondii in rats, Trypanosoma cruzi in mice and Plasmodium mexicanum in the Mexican lizard.

text: Some parasites alter host behavior by infecting

text: neuron

text: s in the host's central nervous system. The host's central nervous system responds to the parasite

text: as it would to any other infection

text: . The hallmarks of such response include local inflammation and the release of chemicals such as

text: cytokine

text: . The immune response itself is responsible for induced behavioral changes in many cases of parasitic infection. Parasites that are known to induce behavioral changes through central nervous system inflammation in their hosts include

text: Toxoplasma gondii

text: in rats,

text: Trypanosoma cruzi

text: in mice and

text: Plasmodium mexicanum

text: in the Mexican lizard.

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answer: 1

text: In 1871 he published his best-known work, Treatise on diseases of the nervous system. In early 1872 he traveled to California to visit his ailing friend Letterman. In 1874 he founded, with Silas Weir Mitchell and many others, the American Neurological Association. In 1878 "he was restored to the army [...] with the grade of brigadier general, without pay or allowances".

text: In 1871 he published his best-known work, Treatise on diseases of the nervous system. In early 1872 he traveled to California to visit his ailing friend Letterman. In 1874 he founded, with Silas Weir Mitchell and many others, the

text: American Neurological Association

text: . In 1878 "he was restored to the army [...] with the grade of brigadier general, without pay or allowances".

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text: Dopamine plays a role in pain processing in multiple levels of the central nervous system including the spinal cord, periaqueductal gray, thalamus, basal ganglia, and cingulate cortex. Decreased levels of dopamine have been associated with painful symptoms that frequently occur in Parkinson's disease. Abnormalities in dopaminergic neurotransmission also occur in several painful clinical conditions, including burning mouth syndrome, fibromyalgia, and restless legs syndrome.

text: Dopamine plays a role in

text: pain

text: processing in multiple levels of the

text: central nervous system

text: including the

text: spinal cord

text: periaqueductal gray

text: thalamus

text: basal ganglia

text: , and

text: cingulate cortex

text: . Decreased levels of dopamine have been associated with painful symptoms that frequently occur in

text: Parkinson's disease

text: . Abnormalities in dopaminergic neurotransmission also occur in several painful clinical conditions, including

text: burning mouth syndrome

text: fibromyalgia

text: , and

text: restless legs syndrome

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answer: Yes

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answer: 1

text: Limbic encephalitis refers to inflammatory disease confined to the limbic system of the brain.  The clinical presentation often includes disorientation, disinhibition, memory loss, seizures, and behavioral anomalies.  MRI imaging reveals T2 hyperintensity in the structures of the medial temporal lobes, and in some cases, other limbic structures.  Some cases of limbic encephalitis are of autoimmune origin.

text: Limbic encephalitis refers to inflammatory disease confined to the

text: limbic system

text: of the brain.  The clinical presentation often includes disorientation, disinhibition, memory loss, seizures, and behavioral anomalies.  MRI imaging reveals T2 hyperintensity in the structures of the medial temporal lobes, and in some cases, other limbic structures.  Some cases of limbic encephalitis are of autoimmune origin.

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text: Another recent perspective has been offered that the Kallikaks almost certainly had undiagnosed fetal alcohol syndrome. In addition to poverty and malnourishment, prenatal alcohol exposure can create craniofacial and other physical anomalies that could account for their peculiar facial features. Furthermore, prenatal alcohol exposure may also damage the central nervous system, which can result in impaired cognitive and behavioral functioning similar to that described by Goddard.

text: Another recent perspective has been offered that the Kallikaks almost certainly had undiagnosed

text: fetal alcohol syndrome

text: . In addition to poverty and malnourishment, prenatal alcohol exposure can create

text: craniofacial

text: and other physical anomalies that could account for their peculiar facial features. Furthermore, prenatal alcohol exposure may also damage the

text: central nervous system

text: , which can result in impaired

text: cognitive

text: and behavioral functioning similar to that described by Goddard.

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answers: prenatal alcohol exposure may also damage the central nervous system

answers: NDQ_010490

answers: undiagnosed fetal alcohol syndrome

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answers: fetal alcohol syndrome

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answers: fetal alcohol syndrome

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: No

answer: 1

text: In June 1886 he was elected a Fellow of the Royal Society   and in 1891 delivered their Croonian Lecture, jointly with his brother-in-law Francis Gotch, on the subject of the mammalian nervous system. In 1894 he won their Royal Medal for his investigations relating to the physiology of the nervous system, and of the thyroid gland, and to their applications to the treatment of disease.

text: In June 1886 he was elected a

text: Fellow of the Royal Society

text: and in 1891 delivered their

text: Croonian Lecture

text: , jointly with his brother-in-law

text: Francis Gotch

text: , on the subject of the mammalian nervous system. In 1894 he won their

text: Royal Medal

text: for his investigations relating to the physiology of the nervous system, and of the thyroid gland, and to their applications to the treatment of disease.

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text: Chronic diseases of age have been saluted as prime candidates for parabiotic research because of the potential to conjoin an older animal with a younger animal. This process could be used to research cardiovascular disease, diabetes, osteoarthritis, and Alzheimer’s disease. As animals age, their oligodendrocytes reduce in efficiency, resulting in decreased myelination, causing negative effects on the central nervous system (CNS). Julia Ruckh and fellow researchers have used parabiosis to study remyelination from adult stem cells to see if conjoining young with older mice could reverse or delay this process. In the experiment, the two mice were conjoined and demyelination was induced via injection into the older mice. The experiment determined that factors from the younger mice reversed CNS demyelination in older mice by revitalizing the oligodendrocytes. The monocytes from the younger mice also enhanced the ability of the older mice to clear myelin debris because the young monocytes can clear lipids from myelin sheaths more effectively than older monocytes. The conjoining of the two animals reversed the effects of age on the myelination cells. The ability of the young mouse’s cells was unaffected. Enhanced immunity from the younger mouse also promoted the general health of the older mouse in each pair. The results of this experiment could lead to therapy processes for people with demyelinating diseases like multiple sclerosis.

text: Chronic diseases of age have been saluted as prime candidates for parabiotic research because of the potential to conjoin an older animal with a younger animal. This process could be used to research cardiovascular disease, diabetes, osteoarthritis, and Alzheimer’s disease. As animals age, their

text: oligodendrocyte

text: s reduce in efficiency, resulting in decreased myelination, causing negative effects on the

text: central nervous system

text: (CNS). Julia Ruckh and fellow researchers have used parabiosis to study

text: remyelination

text: from adult stem cells to see if conjoining young with older mice could reverse or delay this process. In the experiment, the two mice were conjoined and demyelination was induced via injection into the older mice. The experiment determined that factors from the younger mice reversed CNS demyelination in older mice by revitalizing the oligodendrocytes. The

text: monocyte

text: s from the younger mice also enhanced the ability of the older mice to clear myelin debris because the young monocytes can clear lipids from myelin sheaths more effectively than older monocytes. The conjoining of the two animals reversed the effects of age on the myelination cells. The ability of the young mouse’s cells was unaffected. Enhanced immunity from the younger mouse also promoted the general health of the older mouse in each pair. The results of this experiment could lead to therapy processes for people with demyelinating diseases like multiple sclerosis.

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answers: Negative effects on the central nervous system (CNS)

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answer: Yes

answer: Yes

answer: No

answer: No

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answer: 1

text: Degenerative diseases of the human central nervous system such as Parkinson's disease, Alzheimer's disease, multiple sclerosis, and Huntington's disease may also produce mood swings.

text: Degenerative diseases of the human

text: central nervous system

text: such as

text: Parkinson's disease

text: Alzheimer's disease

text: multiple sclerosis

text: , and

text: Huntington's disease

text: may also produce mood swings.

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text: Additionally, HIV-1-infected patients often suffer from myelin degeneration in the central nervous system. These patients have elevated levels of sulfatide in the cerebrospinal fluid (CSF) and anti-sulfatide antibodies in the serum. Elevated levels of anti-sulfatide antibodies can cause demyelination. This is caused by the binding of the anti-sulfatide antibodies to the surface of the myelin sheath and/or the surface of Schwann Cells, which then activates a complete cascade of demyelination. Also, advanced stage AIDS patients can develop Guillain–Barré syndrome (GBS). Guillain–Barré syndrome is classified as an acute autoimmune polyneuropathy, which specifically affects the peripheral nervous system of the infected patient. Experimentation has shown that anti-sulfatide autoimmune antibodies may contribute to the development of Guillain–Barré syndrome in AIDS patients as well as the development of peripheral nervous system injury in HIV-1 infected patients.

text: Additionally, HIV-1-infected patients often suffer from myelin degeneration in the

text: central nervous system

text: . These patients have elevated levels of sulfatide in the

text: cerebrospinal fluid

text: (CSF) and anti-sulfatide antibodies in the

text: serum

text: . Elevated levels of anti-sulfatide antibodies can cause

text: demyelination

text: . This is caused by the binding of the anti-sulfatide antibodies to the surface of the

text: myelin sheath

text: and/or the surface of

text: Schwann Cells

text: , which then activates a complete cascade of demyelination. Also, advanced stage

text: AIDS

text: patients can develop

text: Guillain–Barré syndrome

text: (GBS). Guillain–Barré syndrome is classified as an acute

text: autoimmune

text: polyneuropathy

text: , which specifically affects the

text: peripheral nervous system

text: of the infected patient. Experimentation has shown that anti-sulfatide autoimmune antibodies may contribute to the development of Guillain–Barré syndrome in

text: AIDS

text: patients as well as the development of

text: peripheral nervous system

text: injury in HIV-1 infected patients.

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text: While it was initially believed that both the brain and meninges were devoid of lymphatic vasculature, recent studies, the first by Antoine Louveau and Jonathan Kipnis at the University of Virginia, submitted in October 2014, and another confirming the discovery by Aleksanteri Aspelund and Kari Alitalo at the University of Helsinki submitted in December 2014, identified and described the basic biology of the meningeal lymphatics using a combination of histological, live-imaging, and genetic tools. In general, their work is thought to extend that of the Danish neuroscientist Maiken Nedergaard in identifying the pathway connecting the glymphatic system to the meningeal compartment.

text: While it was initially believed that both the brain and meninges were devoid of lymphatic vasculature, recent studies, the first by Antoine Louveau and

text: Jonathan Kipnis

text: at the

text: University of Virginia

text: , submitted in October 2014, and another confirming the discovery by Aleksanteri Aspelund and

text: Kari Alitalo

text: at the

text: University of Helsinki

text: submitted in December 2014, identified and described the basic biology of the meningeal lymphatics using a combination of

text: histological

text: live-imaging

text: , and

text: genetic

text: tools. In general, their work is thought to extend that of the Danish neuroscientist

text: Maiken Nedergaard

text: in identifying the pathway connecting the

text: glymphatic system

text: to the

text: meningeal compartment

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answers: NDQ_010489

answers: Meningeal lymphatics

answers: NDQ_010490

answers: Lymphatic vasculature

answers: NDQ_010494

answers: Alzheimer's disease

answers: NDQ_010495

answers: Lymphoid

answers: NDQ_010496

answers: Meningitis

answers: NDQ_010497

answers: Alzheimer's disease

answers: NDQ_010498

answers: Meningeal lymphatics

answer: No

answer: No

answer: No

answer: No

answer: No

answer: 1

text: If untreated, T. b. gambiense almost always results in death, with only a few individuals shown in a long-term 15 year follow-up to have survived after refusing treatment. T. b. rhodesiense, being a more acute and severe form of the disease, is consistently fatal if not treated.  Disease progression greatly varies depending on disease form.  For individuals which are infected by T. b. gambiense, which accounts for 98% of all of the reported cases, a person can be infected for months or even years without signs or symptoms until the advanced disease stage, where it is too late to be treated successfully. For individuals affected by T. b. rhodesiense, which accounts for 2% of all reported cases, symptoms appear within weeks or months of the infection. Disease progression is rapid and invades the central nervous system, causing death within a short amount of time.

text: If untreated, T. b. gambiense almost always results in death, with only a few individuals shown in a long-term 15 year follow-up to have survived after refusing treatment. T. b. rhodesiense, being a more acute and severe form of the disease, is consistently fatal if not treated.  Disease progression greatly varies depending on disease form.  For individuals which are infected by T. b. gambiense, which accounts for 98% of all of the reported cases, a person can be infected for months or even years without signs or symptoms until the advanced disease stage, where it is too late to be treated successfully. For individuals affected by T. b. rhodesiense, which accounts for 2% of all reported cases, symptoms appear within weeks or months of the infection. Disease progression is rapid and invades the central nervous system, causing death within a short amount of time.

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answers: The central nervous system

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answers: Invades the central nervous system

answers: NDQ_010494

answers: gambiense

answers: NDQ_010495

answers: T. b.

answers: NDQ_010496

answers: T. b. gambiense

answers: NDQ_010497

answers: central nervous system

answers: NDQ_010498

answers: central nervous system

answer: Yes

answer: Yes

answer: No

answer: No

answer: No

answer: 1

text: Tau proteins (or τ proteins, after the Greek letter with that name) are proteins that stabilize microtubules. They are abundant in neurons of the central nervous system and are less common elsewhere, but are also expressed at very low levels in CNS astrocytes and oligodendrocytes. Pathologies and dementias of the nervous system such as Alzheimer's disease and Parkinson's disease  are associated with tau proteins that have become defective and no longer stabilize microtubules properly.

text: Tau proteins (or τ proteins, after the

text: Greek letter with that name

text: ) are proteins that

text: stabilize

text: microtubules

text: . They are abundant in

text: neuron

text: s of the

text: central nervous system

text: and are less common elsewhere, but are also expressed at very low levels in CNS

text: astrocyte

text: s and

text: oligodendrocyte

text: s. Pathologies and

text: dementia

text: s of the

text: nervous system

text: such as

text: Alzheimer's disease

text: and

text: Parkinson's disease

text: are associated with tau proteins that have become defective and no longer stabilize microtubules properly.

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answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: No

answer: 1

text: CNS demyelinating autoimmune diseases are autoimmune diseases which primarily affect the central nervous system.

text: CNS demyelinating autoimmune diseases are

text: autoimmune disease

text: s which primarily affect the

text: central nervous system

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answer: Yes

answer: Yes

answer: Yes

answer: 1

text: Spontaneous generation and propagation of waves is seen elsewhere in developing circuits. Similar synchronized spontaneous activity early in development has been seen in neurons of the hippocampus, spinal cord, and auditory nuclei.Patterned activity shaping neuronal connections and control of synaptic efficiency in multiple systems including the retina are important for understanding interaction between presynaptic and postsynaptic cells that create precise connections essential to the function of the nervous system.

text: Spontaneous generation and propagation of waves is seen elsewhere in developing circuits. Similar synchronized spontaneous activity early in development has been seen in neurons of the

text: hippocampus

text: spinal cord

text: , and auditory nuclei.Patterned activity shaping neuronal connections and control of synaptic efficiency in multiple systems including the retina are important for understanding interaction between

text: presynaptic

text: and

text: postsynaptic

text: cells that create precise connections essential to the function of the nervous system.

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answers: hippocampus

answer: No

answer: No

answer: No

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text: Meningococcal meningitis is a form of bacterial meningitis. Meningitis is a disease caused by inflammation and irritation of the meninges, the membranes surrounding the brain and spinal cord. In meningococcal meningitis this is caused by bacteria invading the cerebrospinal fluid, circulating through the central nervous system. Sub-Saharan Africa, the Americas, Western Europe, the UK, and Ireland still face many challenges combating this disease, 200 years after the discovery of bacterial meningitis.

text: Meningococcal meningitis is a form of bacterial

text: meningitis

text: . Meningitis is a disease caused by inflammation and irritation of the

text: meninges

text: , the membranes surrounding the brain and spinal cord. In meningococcal meningitis this is caused by bacteria invading the

text: cerebrospinal fluid

text: , circulating through the

text: central nervous system

text: . Sub-Saharan Africa, the Americas, Western Europe, the UK, and Ireland still face many challenges combating this disease, 200 years after the discovery of bacterial meningitis.

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answers: Meningitis

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answers: Meningitis

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answers: Meningitis

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answers: Meningitis

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answers: Meningitis

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answers: Meningitis

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: HSV infection causes several distinct medical disorders.  Common infection of the skin or mucosa may affect the face and mouth (orofacial herpes), genitalia (genital herpes), or hands (herpetic whitlow).  More serious disorders occur when the virus infects and damages the eye (herpes keratitis), or invades the central nervous system, damaging the brain (herpes encephalitis).  People with immature or suppressed immune systems, such as newborns, transplant recipients, or people with AIDS,  are prone to severe complications from HSV infections. HSV infection has also been associated with cognitive deficits of bipolar disorder, and Alzheimer's disease, although this is often dependent on the genetics of the infected person.

text: HSV infection causes several distinct medical

text: disorders

text: .  Common infection of the skin or mucosa may affect the face and mouth (orofacial herpes), genitalia (genital herpes), or hands (

text: herpetic whitlow

text: ).  More serious disorders occur when the virus infects and damages the eye (

text: herpes keratitis

text: ), or invades the central nervous system, damaging the brain (herpes encephalitis).  People with immature or suppressed immune systems, such as newborns, transplant recipients, or people with AIDS,  are prone to severe complications from HSV infections. HSV infection has also been associated with cognitive deficits of

text: bipolar disorder

text: , and

text: Alzheimer's disease

text: , although this is often dependent on the

text: genetics

text: of the infected person.

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answers: the central nervous system

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answers: herpes encephalitis

answer: Yes

answer: Yes

answer: No

answer: Yes

answer: No

answer: 1

text: When looking at the genetics of deafness, there are 2 different forms, syndromic and nonsyndromic. Syndromic deafness occurs when there are other signs or medical problems aside from deafness in an individual. This accounts for around 30% of deaf individuals who are deaf from a genetic standpoint. Nonsyndromic deafness occurs when there are no other signs or medical problems associated with an individual other than deafness. From a genetic standpoint, this accounts for the other 70% of cases, and represents the majority of hereditary hearing loss. Syndromic cases occur with diseases such as Usher syndrome, Stickler syndrome, Waardenburg syndrome, Alport's syndrome, and neurofibromatosis type 2. These are diseases that have deafness as one of the symptoms or as a common feature associated with it. Many of the genetic mutations giving rise to syndromic deafness have been identified. In nonsyndromic cases, where deafness is the only finding, it is more difficult to identify the genetic mutation although some have been discovered.

text: When looking at the genetics of deafness, there are 2 different forms, syndromic and

text: nonsyndromic

text: . Syndromic deafness occurs when there are other signs or medical problems aside from deafness in an individual. This accounts for around 30% of deaf individuals who are deaf from a genetic standpoint. Nonsyndromic deafness occurs when there are no other signs or medical problems associated with an individual other than deafness. From a genetic standpoint, this accounts for the other 70% of cases, and represents the majority of hereditary hearing loss. Syndromic cases occur with diseases such as

text: Usher syndrome

text: Stickler syndrome

text: Waardenburg syndrome

text: Alport's syndrome

text: , and

text: neurofibromatosis type 2

text: . These are diseases that have deafness as one of the symptoms or as a common feature associated with it. Many of the genetic mutations giving rise to syndromic deafness have been identified. In nonsyndromic cases, where deafness is the only finding, it is more difficult to identify the genetic mutation although some have been discovered.

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answers: Alport's syndrome

answer: No

answer: No

answer: No

answer: No

answer: No

answer: 1

text: Evaluation of any suspected disease of the somatosensory system is included in a neurological examination of the peripheral nervous system

text: Evaluation of any suspected disease of the somatosensory system is included in a

text: neurological examination of the peripheral nervous system

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answers: 2: The answer has limited relevance and completeness, with significant gaps or inaccuracies

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answers: The peripheral nervous system

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answers: peripheral

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answers: peripheral

answer: Yes

answer: Yes

answer: Yes

answer: No

answer: Yes

answer: 1

text: Neurocritical care or neurointensive care is a branch of medicine that emerged in the 1980s and deals with life-threatening diseases of the nervous system, which are those that involve the brain, spinal cord and nerves.  The doctors who practice this type of medicine are called neurointensivists, and can have medical training in many fields, including neurology, anesthesiology, emergency medicine, or neurosurgery.  Common diseases treated in neurointensive care units include strokes, ruptured aneurysms, brain and spinal cord injury from trauma, seizures (especially those that last for a long period of time- status epilepticus, and/or involve trauma to the patient, i.e., due to a stroke or a fall), swelling of the brain (intracranial edema), infections of the brain (encephalitis) and the brain's or spine's meninges (meningitis), brain tumors (especially malignant cases; with neurological oncology), and weakness of the muscles required to breathe (such as the diaphragm).  Besides dealing with critical illness of the nervous system, neurointensivists also treat the medical complications that may occur in their patients, including those of the heart, lung, kidneys, or any other body system, including treatment of infections.  Most neurocritical care units are a collaborative effort between neurointensivists, neurosurgeons, neurologists, radiologists, pharmacists, physician extenders (such as nurse practitioners or physician's assistants), critical care nurses, respiratory therapists, rehabilitation therapists, and social workers who all work together in order to provide coordinated care for the critically ill neurologic patient.  The Neurocritical Care Society was founded in 2002 in San Francisco, California to promote quality patient care, professional collaboration, research, training, education and advocacy with the goal of improving outcomes for patients with life-threatening neurologic diseases.  In 2003, the Society first met officially in Phoenix, Arizona.  Thomas P. Bleck MD was the first president.

text: Neurocritical care or neurointensive care is a branch of

text: medicine

text: that emerged in the 1980s and deals with life-threatening diseases of the

text: nervous system

text: , which are those that involve the brain, spinal cord and nerves.  The doctors who practice this type of medicine are called neurointensivists, and can have medical training in many fields, including neurology, anesthesiology, emergency medicine, or neurosurgery.  Common diseases treated in neurointensive care units include

text: strokes

text: , ruptured

text: aneurysms

text: , brain and

text: spinal cord injury

text: from trauma,

text: seizure

text: s (especially those that last for a long period of time-

text: status epilepticus

text: , and/or involve trauma to the patient, i.e., due to a stroke or a fall), swelling of the brain (

text: intracranial edema

text: ), infections of the brain (

text: encephalitis

text: ) and the brain's or spine's meninges (

text: meningitis

text: brain tumor

text: s (especially malignant cases; with neurological

text: oncology

text: ), and weakness of the muscles required to breathe (such as the

text: diaphragm

text: ).  Besides dealing with critical illness of the nervous system, neurointensivists also treat the medical complications that may occur in their patients, including those of the heart, lung, kidneys, or any other body system, including treatment of infections.  Most neurocritical care units are a collaborative effort between neurointensivists,

text: neurosurgeons

text: neurologists

text: radiologists

text: pharmacists

text: , physician extenders (such as nurse practitioners or physician's assistants), critical care nurses, respiratory therapists, rehabilitation therapists, and social workers who all work together in order to provide coordinated care for the critically ill neurologic patient.  The Neurocritical Care Society was founded in 2002 in

text: San Francisco, California

text: to promote quality patient care, professional collaboration, research, training, education and advocacy with the goal of improving outcomes for patients with life-threatening neurologic diseases.  In 2003, the Society first met officially in

text: Phoenix, Arizona

text: .  Thomas P. Bleck MD was the first president.

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answers: Neurocritical care or neurointensive care

answers: NDQ_010490

answers: infections

answers: NDQ_010494

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answers: NDQ_010495

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answers: NDQ_010496

answers: status epilepticus

answers: NDQ_010497

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answers: NDQ_010498

answers: status epilepticus

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: People afflicted by this disease also experience a loss of neurons. Loss of neurons is a hallmark of neurodegenerative diseases. Due to the generally non-regenerative nature of neuronal cells in the adult central nervous system, this results in an irreversible and fatal process of neurodegeneration. There is also the presence of several movement related disorders including chorea, dystonia and bradykinesia, one of the more incapacitating ones includes Truncal spasms.

text: People afflicted by this disease also experience a loss of neurons. Loss of neurons is a hallmark of neurodegenerative diseases. Due to the generally non-regenerative nature of neuronal cells in the adult central nervous system, this results in an irreversible and fatal process of neurodegeneration. There is also the presence of several movement related disorders including chorea, dystonia and bradykinesia, one of the more incapacitating ones includes Truncal spasms.

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answers: neurodegenerative diseases

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answers: neurodegenerative diseases

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answers: neurodegenerative diseases

answers: NDQ_010496

answers: dystonia

answers: NDQ_010497

answers: neurodegenerative diseases

answers: NDQ_010498

answers: neurodegeneration

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: A dystenoid nervous system is an arrangement of ganglia in the anterior region of some molluscs in which the cerebral and pleural ganglia are situated closer to each other than they are in those molluscs with the more archaic hypoathroid nervous system but still farther apart than those animals with an evolutionarily more recent epiathroid nervous system.

text: A dystenoid nervous system is an arrangement of

text: ganglia

text: in the anterior region of some

text: mollusc

text: s in which the cerebral and pleural ganglia are situated closer to each other than they are in those molluscs with the more archaic

text: hypoathroid

text: nervous system but still farther apart than those animals with an evolutionarily more recent

text: epiathroid

text: nervous system.

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answers: NDQ_010489

answers: neurological system

answers: NDQ_010490

answers: hypoathroid nervous system

answers: NDQ_010494

answers: dystenoid

answers: NDQ_010495

answers: hypoathroid

answers: NDQ_010496

answers: dystenoid

answers: NDQ_010497

answers: hypoathroid

answers: NDQ_010498

answers: dystenoid

answer: No

answer: No

answer: No

answer: No

answer: No

answer: 1

text: Culex quinquefasciatus (earlier known as Culex fatigans), the southern house mosquito, is a medium-sized mosquito found in tropical and subtropical regions of the world. It is the vector of Wuchereria bancrofti, avian malaria, and arboviruses including St. Louis encephalitis virus, Western equine encephalitis virus, zika virus and West Nile virus. It is taxonomically regarded as a member of the Culex pipiens species complex. Its genome was sequenced in 2010, and was shown to have 18,883 protein-coding genes.

text: Culex quinquefasciatus (earlier known as Culex fatigans), the southern house mosquito, is a medium-sized mosquito found in

text: tropical

text: and

text: subtropical

text: regions of the world. It is the

text: vector

text: of

text: Wuchereria bancrofti

text: avian malaria

text: , and

text: arboviruses

text: including

text: St. Louis encephalitis virus

text: Western equine encephalitis virus

text: zika virus

text: and

text: West Nile virus

text: . It is taxonomically regarded as a member of the

text: Culex pipiens

text: species complex

text: . Its

text: genome was sequenced

text: in 2010, and was shown to have 18,883 protein-coding genes.

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answers: encephalitis

answers: NDQ_010490

answers: St. Louis encephalitis virus

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answers: St. Louis encephalitis

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answers: encephalitis

answers: NDQ_010496

answers: encephalitis

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answers: encephalitis

answers: NDQ_010498

answers: encephalitis

answer: Yes

answer: Yes

answer: No

answer: Yes

answer: No

answer: 1

text: Cryptococcal disease is a very rare disease that can affect the lungs (pneumonia) and nervous system (causing meningitis and focal brain lesions called cryptococcomas) in humans. The main complication of lung infection is respiratory failure. Central nervous system infection may lead to hydrocephalus, seizures and focal neurological deficit.

text: Cryptococcal disease is a very rare disease that can affect the lungs (

text: pneumonia

text: ) and nervous system (causing meningitis and focal brain lesions called cryptococcomas) in humans. The main complication of lung infection is respiratory failure. Central nervous system infection may lead to

text: hydrocephalus

text: , seizures and focal neurological deficit.

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answers: Cryptococcal disease

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answers: Central nervous system infection

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answers: neurological deficit

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answers: Central nervous system infection

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answer: Yes

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text: The central feature of the new realism was a rejection of the epistemological dualism of John Locke and of older forms of realism. The group maintained that, when one is conscious of, or knows, an object, it is an error to say that the object in itself and our knowledge of the object are two distinct facts. If we know a particular cow is black, is the blackness on that cow or in the observer's mind? Holt wrote; "That color out there is the thing in consciousness selected for such inclusion by the nervous system's specific response," Consciousness is not physically identical with the nervous system: it is "out there" with the cow, all throughout the field of sight (and smell, and hearing) and identical with the set of facts it knows at any moment. The nervous system is merely a system of selection.

text: The central feature of the new realism was a rejection of the

text: epistemological

text: dualism

text: of

text: John Locke

text: and of older forms of realism. The group maintained that, when one is conscious of, or knows, an object, it is an error to say that the object in itself and our knowledge of the object are two distinct facts. If we know a particular cow is black, is the blackness on that cow or in the observer's mind? Holt wrote; "That color out there is the thing in

text: consciousness

text: selected for such inclusion by the

text: nervous system

text: 's specific response," Consciousness is not physically identical with the

text: nervous system

text: : it is "out there" with the cow, all throughout the field of sight (and smell, and hearing) and identical with the set of facts it knows at any moment. The nervous system is merely a system of selection.

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answers: The nervous system is merely a system of selection

answers: NDQ_010490

answers: It is not physically identical with the nervous system

answers: NDQ_010494

answers: Alzheimer's

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answers: Lymphoma

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answers: epilepsy

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answers: epilepsy

answer: No

answer: No

answer: No

answer: No

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answer: 1

text: The chief diagnostic indications of lumbar puncture are for collection of cerebrospinal fluid (CSF). Analysis of CSF may exclude infectious, inflammatory, and neoplastic diseases affecting the central nervous system. The most common purpose is in suspected meningitis, since there is no other reliable tool with which meningitis, a life-threatening but highly treatable condition, can be excluded. A lumbar puncture can also be used to detect whether someone has 'Stage 1' or 'Stage 2' Trypanosoma brucei. Young infants commonly require lumbar puncture as a part of the routine workup for fever without a source. This is due to higher rates of meningitis than in older persons. Infants also do not reliably show classic symptoms of meningeal irritation (meningismus) like neck stiffness and headache the way adults do.  In any age group, subarachnoid hemorrhage, hydrocephalus, benign intracranial hypertension, and many other diagnoses may be supported or excluded with this test. It may also be used to detect the presence of malignant cells in the CSF, as in carcinomatous meningitis or medulloblastoma. CSF containing less than 10 red blood cells (RBCs)/mm³ constitutes a "negative" tap in the context of a workup for subarachnoid hemorrhage, for example. Taps that are "positive" have an RBC count of 100/mm³ or more.

text: The chief diagnostic indications of lumbar puncture are for collection of

text: cerebrospinal fluid

text: (CSF). Analysis of CSF may exclude infectious, inflammatory, and neoplastic diseases affecting the central nervous system. The most common purpose is in suspected

text: meningitis

text: , since there is no other reliable tool with which meningitis, a life-threatening but highly treatable condition, can be excluded. A lumbar puncture can also be used to detect whether someone has 'Stage 1' or 'Stage 2'

text: Trypanosoma brucei

text: . Young infants commonly require lumbar puncture as a part of the routine workup for fever without a source. This is due to higher rates of meningitis than in older persons. Infants also do not reliably show classic symptoms of meningeal irritation (

text: meningism

text: us) like neck stiffness and headache the way adults do.  In any age group,

text: subarachnoid hemorrhage

text: hydrocephalus

text: benign intracranial hypertension

text: , and many other diagnoses may be supported or excluded with this test. It may also be used to detect the presence of malignant cells in the CSF, as in

text: carcinomatous meningitis

text: or

text: medulloblastoma

text: . CSF containing less than 10

text: red blood cells

text: (RBCs)/mm³ constitutes a "negative" tap in the context of a workup for subarachnoid hemorrhage, for example. Taps that are "positive" have an RBC count of 100/mm³ or more.

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answers: NDQ_010489

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answers: 3

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answers: 0

answers: NDQ_010489

answers: Meningeal irritation (meningismus)

answers: NDQ_010490

answers: The most common purpose is in suspected meningitis

answers: NDQ_010494

answers: meningitis

answers: NDQ_010495

answers: inflammatory

answers: NDQ_010496

answers: meningitis

answers: NDQ_010497

answers: carcinomatous meningitis

answers: NDQ_010498

answers: meningitis

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: The most prominent member of Acanthamoebidae, Acanthamoeba, can be potentially pathogenic to humans and animals.  Typically, a person or animal with a normally functioning immune system can avoid infection; Edagawa et al. states, Acanthamoeba "are known to be the opportunistic pathogens in granulomatous amoebic encephalitis (GAE), a chronic disease of immuno-compromised hosts such as AIDS patients and transplants recipients.”  GAE is the result of microscopic cysts that form in the central nervous system. Acanthamoeba can also be the source of infections in the lungs, sinuses, skin, and eyes.

text: The most prominent member of Acanthamoebidae, Acanthamoeba, can be potentially pathogenic to humans and animals.  Typically, a person or animal with a normally functioning immune system can avoid infection; Edagawa et al. states, Acanthamoeba "are known to be the opportunistic

text: pathogens

text: in

text: granulomatous amoebic encephalitis

text: (GAE), a chronic disease of immuno-compromised hosts such as

text: AIDS

text: patients and transplants recipients.”  GAE is the result of microscopic cysts that form in the central nervous system. Acanthamoeba can also be the source of infections in the lungs, sinuses, skin, and eyes.

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answers: NDQ_010489

answers: Microscopic cysts that form in the central nervous system

answers: NDQ_010490

answers: encephalitis

answers: NDQ_010494

answers: encephalitis

answers: NDQ_010495

answers: amoebic encephalitis

answers: NDQ_010496

answers: encephalitis

answers: NDQ_010497

answers: GAE

answers: NDQ_010498

answers: encephalitis

answer: Yes

answer: Yes

answer: No

answer: Yes

answer: No

answer: 1

text: Tick-borne encephalitis (TBE) is a viral infectious disease involving the central nervous system. The disease most often manifests as meningitis, encephalitis, or meningoencephalitis. Although TBE is most commonly recognized as a neurological disorder, mild fever can also occur. Long-lasting or permanent neuropsychiatric sequelae are observed in 10 to 20% of infected patients.

text: Tick-borne encephalitis (TBE) is a

text: viral

text: infectious disease

text: involving the

text: central nervous system

text: . The disease most often manifests as

text: meningitis

text: encephalitis

text: , or

text: meningoencephalitis

text: . Although TBE is most commonly recognized as a

text: neurological disorder

text: , mild fever can also occur. Long-lasting or permanent neuropsychiatric

text: sequela

text: e are observed in 10 to 20% of infected patients.

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answer: 1

text: A hippocampus prosthesis  is a type of cognitive prosthesis (a prosthesis implanted into the nervous system in order to improve or replace the function of damaged brain tissue). Prosthetic devices replace normal function of a damaged body part; this can be simply a structural replacement (e.g. reconstructive surgery or glass eye) or a rudimentary, functional replacement (e.g. a pegleg or hook).  However, prosthetics involving the brain have some special categories and requirements.  "Input" prosthetics, such as retinal or cochlear implant, supply signals to the brain that the patient eventually learns to interpret as sight or sound.  "Output" prosthetics use brain signals to drive a bionic arm, hand or computer device, and require considerable training during which the patient learns to generate the desired action via their thoughts.  Both of these types of prosthetics rely on the plasticity of the brain to adapt to the requirement of the prosthesis, thus allowing the user to "learn" the use of his new body part.  A cognitive or "brain-to-brain" prosthesis involves neither learned input nor output signals, but the native signals used normally by the area of the brain to be replaced (or supported).  Thus, such a device must be able to fully replace the function of a small section of the nervous system - using that section′s normal mode of operation. In order to achieve this, developers require a deep understanding of the functioning of the nervous system. The scope of design must include a reliable mathematical model as well as the technology in order to properly manufacture and install a cognitive prosthesis. The primary goal of an artificial hippocampus is to provide a cure for Alzheimer's disease and other hippocampus—related problems. To do so, the prosthesis  has to be able to receive information directly from the brain, analyze the information and give an appropriate output to the cerebral cortex; in other words, it must behave just like a natural hippocampus.  At the same time, the artificial organ must be completely autonomous, since any exterior power source will greatly increase the risk of infection.

text: A hippocampus prosthesis  is a type of

text: cognitive prosthesis

text: (a prosthesis implanted into the nervous system in order to improve or replace the function of damaged brain tissue). Prosthetic devices replace normal function of a damaged body part; this can be simply a structural replacement (e.g. reconstructive surgery or glass eye) or a rudimentary, functional replacement (e.g. a pegleg or hook).  However, prosthetics involving the brain have some special categories and requirements.  "Input" prosthetics, such as retinal or

text: cochlear implant

text: , supply signals to the brain that the patient eventually learns to interpret as sight or sound.  "Output" prosthetics use brain signals to drive a

text: bionic

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answers: A hippocampus prosthesis is a type of cognitive prosthesis

answers: NDQ_010490

answers: not power the artificial hippocampus

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answers: Alzheimer's disease is characterized by memory loss

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answer: No

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answer: No

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answer: 1

text: The enteric nervous system is one of the main divisions of the nervous system and is the main focus of neurogastroenterology.  The enteric nervous system refers to the entire system of neurons that govern the gastrointestinal system.  It is capable of operating independently of the brain and spinal cord, but does rely on innervation from the autonomic nervous system via the vagus nerve and prevertebral ganglia in healthy subjects.  However, studies have shown that the system is operable with a severed vagus nerve.  The neurons of the enteric nervous system control the motor functions of the system, in addition to the secretion of gastrointestinal enzymes.  These neurons communicate through many neurotransmitters similar to the CNS, including acetylcholine, dopamine, and serotonin.  The large presence of serotonin and dopamine in the gut are key areas of research for neurogastroenterologists.

text: The enteric nervous system is one of the main divisions of the

text: nervous system

text: and is the main focus of neurogastroenterology.  The enteric nervous system refers to the entire system of neurons that govern the gastrointestinal system.  It is capable of operating independently of the brain and spinal cord, but does rely on innervation from the autonomic nervous system via the

text: vagus nerve

text: and

text: prevertebral ganglia

text: in healthy subjects.  However, studies have shown that the system is operable with a severed vagus nerve.  The neurons of the enteric nervous system control the motor functions of the system, in addition to the secretion of gastrointestinal enzymes.  These neurons communicate through many

text: neurotransmitters

text: similar to the CNS, including

text: acetylcholine

text: dopamine

text: , and

text: serotonin

text: .  The large presence of serotonin and dopamine in the

text: gut

text: are key areas of research for neurogastroenterologists.

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answers: The enteric nervous system

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answers: The enteric nervous system

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answers: enteric nervous system

answers: NDQ_010497

answers: enteric nervous system

answers: NDQ_010498

answers: enteric nervous system

answer: No

answer: No

answer: No

answer: No

answer: No

answer: 1

text: Granulomatous meningoencephalitis (GME) is an inflammatory disease of the central nervous system (CNS) of dogs and, rarely, cats. It is a form of meningoencephalitis.  GME is likely second only to encephalitis caused by canine distemper virus as the most common cause of inflammatory disease of the canine CNS.  The disease is more common in female toy dogs of young and middle age.  It has a rapid onset.  The lesions of GME exist mainly in the white matter of the cerebrum, brainstem, cerebellum, and spinal cord.  The cause is only known to be noninfectious and is considered at this time to be idiopathic.  Because lesions resemble those seen in allergic meningoencephalitis, GME is thought to have an immune-mediated cause, but it is also thought that the disease may be based on an abnormal response to an infectious agent.  One study searched for viral DNA from canine herpesvirus, canine adenovirus, and canine parvovirus in brain tissue from dogs with GME, necrotizing meningoencephalitis, and necrotizing leukoencephalitis (see below for the latter two conditions), but failed to find any.

text: Granulomatous meningoencephalitis (GME) is an inflammatory disease of the

text: central nervous system

text: (CNS) of

text: dog

text: s and, rarely,

text: cat

text: s. It is a form of

text: meningoencephalitis

text: .  GME is likely second only to

text: encephalitis

text: caused by

text: canine distemper virus

text: as the most common cause of inflammatory disease of the canine CNS.  The disease is more common in female toy dogs of young and middle age.  It has a rapid onset.  The lesions of GME exist mainly in the

text: white matter

text: of the

text: cerebrum

text: brainstem

text: cerebellum

text: , and

text: spinal cord

text: .  The cause is only known to be noninfectious and is considered at this time to be

text: idiopathic

text: .  Because lesions resemble those seen in allergic meningoencephalitis, GME is thought to have an

text: immune-mediated

text: cause, but it is also thought that the disease may be based on an abnormal response to an infectious agent.  One study searched for viral

text: DNA

text: from

text: canine herpesvirus

text: canine adenovirus

text: , and

text: canine parvovirus

text: in brain tissue from dogs with GME, necrotizing meningoencephalitis, and necrotizing leukoencephalitis (see below for the latter two conditions), but failed to find any.

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answers: GME

answers: NDQ_010490

answers: noninfectious

answers: NDQ_010494

answers: meningoencephalitis

answers: NDQ_010495

answers: allergic meningoencephalitis

answers: NDQ_010496

answers: meningoencephalitis

answers: NDQ_010497

answers: necrotizing leukoencephalitis

answers: NDQ_010498

answers: meningoencephalitis

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: Meningitis is an inflammation of the meninges (membranes) of the brain and spinal cord. It is most often caused by a bacterial or viral infection. Fever, vomiting, and a stiff neck are all symptoms of meningitis.

text: Meningitis is an inflammation of the

text: meninges

text: (membranes) of the brain and spinal cord. It is most often caused by a bacterial or viral infection. Fever, vomiting, and a stiff neck are all symptoms of meningitis.

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text: Herpes simplex encephalitis can be treated with acyclovir, but few of the other viral central nervous system infections have a specific therapy.

text: Herpes simplex encephalitis

text: can be treated with

text: acyclovir

text: , but few of the other viral central nervous system infections have a specific therapy.

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text: By using a variety of sense receptors, plants sense light, gravity, temperature, humidity, chemical substances, chemical gradients, reorientation, magnetic fields, infections, tissue damage and mechanical pressure. The absence of a nervous system notwithstanding, plants interpret and respond to these stimuli by a variety of hormonal and cell-to-cell communication pathways that result in movement, morphological changes and physiological state alterations at the organism level, that is, result in plant behavior. Such physiological and cognitive functions are generally not believed to give rise to mental phenomena or qualia, however, as these are typically considered the product of nervous system activity. The emergence of mental phenomena from the activity of systems  functionally or computationally analogous to that of nervous systems is, however, a hypothetical possibility explored by some schools of thought in the philosophy of mind field, such as functionalism and computationalism.

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text: It is important to note that, though they present with similar symptoms and often occur in tandem, meningitis and ventriculitis are two different diseases, so physicians must be able to distinguish between the two. Meningitis is the inflammation of the protective lining of the central nervous system, called meninges. Because of the similar pathologies and etiologies of the two types of inflammation, they are difficult to differentiate using chemical testing, but show very different visual effects in both the MRI and CT scans, hence their use as a validation that the patient does, in fact, have ventriculitis and not another, but deceptively similar condition such as meningitis.

text: It is important to note that, though they present with similar symptoms and often occur in tandem,

text: meningitis

text: and ventriculitis are two different diseases, so physicians must be able to distinguish between the two. Meningitis is the inflammation of the protective lining of the central nervous system, called

text: meninges

text: . Because of the similar

text: pathologies

text: and

text: etiologies

text: of the two types of inflammation, they are difficult to differentiate using chemical testing, but show very different visual effects in both the MRI and CT scans, hence their use as a validation that the patient does, in fact, have ventriculitis and not another, but deceptively similar condition such as meningitis.

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text: The enteric nervous system is one of the main divisions of the nervous system and consists of a mesh-like system of neurons that governs the function of the gastrointestinal system; it has been described as a "second brain" for several reasons. The enteric nervous system can operate autonomously. It normally communicates with the central nervous system (CNS) through the parasympathetic (e.g., via the vagus nerve) and sympathetic (e.g., via the prevertebral ganglia) nervous systems.  However, vertebrate studies show that when the vagus nerve is severed, the enteric nervous system continues to function.

text: The

text: enteric nervous system

text: is one of the main divisions of the

text: nervous system

text: and consists of a mesh-like system of

text: neuron

text: s that governs the function of the

text: gastrointestinal system

text: ; it has been described as a "second brain" for several reasons. The enteric nervous system can operate autonomously. It normally communicates with the

text: central nervous system

text: (CNS) through the

text: parasympathetic

text: (e.g., via the

text: vagus nerve

text: ) and

text: sympathetic

text: (e.g., via the

text: prevertebral ganglia

text: ) nervous systems.  However,

text: vertebrate

text: studies show that when the

text: vagus nerve

text: is severed, the enteric nervous system continues to function.

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text: Patients with severe infections may require hospitalization. They may become thrombocytopenic, hyponatremic, experience elevated liver enzymes, and other more pronounced symptoms.  It is not uncommon for severe cases to involve the respiratory system, central nervous system, gastrointestinal system or the renal system complications. This disease is worst for elderly patients, males, African-Americans, alcoholics, and patients with G6PD deficiency.

text: Patients with severe infections may require hospitalization. They may become

text: thrombocytopenic

text: hyponatremic

text: , experience elevated liver enzymes, and other more pronounced symptoms.  It is not uncommon for severe cases to involve the respiratory system, central nervous system, gastrointestinal system or the renal system complications. This disease is worst for elderly patients, males, African-Americans, alcoholics, and patients with

text: G6PD deficiency

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answer: Yes

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answer: Yes

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answer: 1

text: In scleromyxedema, symptoms can occur on larger part of the body. Erythema and scleroderma-like induration occurs on the skin. In addition, the mobility of the lips, hands, arms, and legs is reduced. Proximal myopathy, inflammatory polyarthritis, central nervous system symptoms, esophageal aperistalsis, and hoarseness are among the notable systemic symptoms. If viscera is involved, the disease will be fatal. The dermatoneuro syndrome is a rare neurological complication of the disease presenting with fever, seizures and altered mental status.

text: In scleromyxedema, symptoms can occur on larger part of the body.

text: Erythema

text: and

text: scleroderma

text: -like induration occurs on the skin. In addition, the mobility of the lips, hands, arms, and legs is reduced. Proximal

text: myopathy

text: , inflammatory

text: polyarthritis

text: central nervous system

text: symptoms,

text: esophageal

text: aperistalsis, and hoarseness are among the notable systemic symptoms. If

text: viscera

text: is involved, the disease will be fatal. The

text: dermatoneuro syndrome

text: is a rare neurological complication of the disease presenting with fever, seizures and altered mental status.

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text: The autonomic nervous system controls all automatic functions in the body and contains two subsections within it that aids in response to an acute stress reaction. These two subunits are the sympathetic nervous system and the parasympathetic nervous system. The sympathetic response is colloquially known as the "fight or flight" response, indicated by accelerated pulse and respiration rates, pupil dilation, and a general feeling of anxiety and hyper-awareness.  This is caused by the release of epinephrine and norepinephrine from the adrenal glands. the norepinephrine and epinephrine strike the beta receptors of the heart in order to feed the heart sympathetic nerve fibers to increase the strength of heart muscle contraction. as a result more blood is being circulated increasing the heart rate and respiratory rate. The sympathetic nervous system also stimulates the skeletal system and muscular system in an effort to pump more blood to those areas to handle the acute stress. simultaneously the sympathetic nervous system inhibits the digestive system and the urinary system in order to optimize blood flow to the heart, lungs, and skeletal muscles. this plays a role in the alarm reaction stage. The para-sympathetic response is colloquially known as the "rest and digest" response, indicated by reduced heart and respiration rates, and more obviously by a temporary loss of consciousness if the system is fired at a rapid rate. The parasympathetic nervous system stimulates the digestive system and urinary system in order to send more blood to those systems to increase the process of digestion. In order to do this, it must inhibit the cardiovascular system and respiratory system in an effort to optimize blood flow to the digestive tract causing low heart and respiratory rates. parasympathetic plays no role in acute stress response (VanPutte Regan Russo 2014).

text: The autonomic nervous system controls all automatic functions in the body and contains two subsections within it that aids in response to an acute stress reaction. These two subunits are the sympathetic nervous system and the parasympathetic nervous system. The sympathetic response is colloquially known as the "

text: fight or flight

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text: Meningitis is the swelling of the membranes around the spinal cord and brain most often caused by Viral and Bacterial infection which incidentally are the only two types which could be passed through contact with door handles.[1] Although relatively rare, meningitis can be life threatening with rapidly developing symptoms. Bacterial Meningitis is the deadliest form of this disease but throughout the years, having become more aware of the disease and its causes, we have been able to significantly lower the amount of deaths due to meningitis each year. As a child, you are more at risk of contracting meningitis which is why recommended vaccines have been put in place across the UK for the three main bacteria types: Neisseria meningitides, Streptococcus pneumoniae and Haemophilus influenzae type b (Hib).

text: Meningitis

text: is the swelling of the membranes around the spinal cord and brain most often caused by

text: Viral

text: and

text: Bacterial

text: infection which incidentally are the only two types which could be passed through contact with door handles.[1] Although relatively rare, meningitis can be life threatening with rapidly developing symptoms. Bacterial Meningitis is the deadliest form of this disease but throughout the years, having become more aware of the disease and its causes, we have been able to significantly lower the amount of deaths due to meningitis each year. As a child, you are more at risk of contracting meningitis which is why recommended vaccines have been put in place across the UK for the three main bacteria types: Neisseria meningitides, Streptococcus pneumoniae and Haemophilus influenzae type b (Hib).

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text: Hypersomnia can be secondary to disorders such as clinical depression, multiple sclerosis, encephalitis, epilepsy, or obesity. Hypersomnia can also be a symptom of other sleep disorders, like sleep apnea. It may occur as an adverse effect of taking certain medications, of withdrawal from some medications, or of drug or alcohol abuse. A genetic predisposition may also be a factor. In some cases it results from a physical problem, such as a tumor, head trauma, or dysfunction of the autonomic or central nervous system.

text: Hypersomnia can be secondary to disorders such as

text: clinical depression

text: multiple sclerosis

text: encephalitis

text: epilepsy

text: , or

text: obesity

text: . Hypersomnia can also be a symptom of other sleep disorders, like

text: sleep apnea

text: . It may occur as an

text: adverse effect

text: of taking certain medications, of withdrawal from some medications, or of drug or alcohol abuse. A genetic predisposition may also be a factor. In some cases it results from a physical problem, such as a tumor, head trauma, or dysfunction of the

text: autonomic

text: or

text: central nervous system

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answers: dysfunction of the autonomic or central nervous system

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answers: encephalitis

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answers: epilepsy

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answer: Yes

answer: Yes

answer: Yes

answer: 1

text: The next phase of T. regenti infection is represented by schistosomula migration in the central nervous system. This is accompanied by serious neurological malfunctions in birds that suffer from leg paralysis and balance disorders. At this stage, schistosomula feed on nervous tissue as demonstrated by detection of oligodendrocytes and neurons in the lumen of parasite’s intestine. A cysteine peptidase cathepsin B1 of T. regenti (TrCB1) localised in intestines of migrating schistosomula is capable of myelin basic protein degradation, thus probably serving for nervous tissue digestion. Nonetheless, the nervous tissue ingestion has likely only a minor pathogenic effect on the host central nervous tissue. This is underpinned by observations of leg paralysis only in immunocompromised hosts, whereas in experiments with immunocompetent mouse strains, the infected animals did not reveal any neurological disorders. The neurological symptoms originate probably in mechanical damage of the nervous tissue leading to dystrophic or even necrotic changes of neurons and axonal injury. The cause of it is large migrating schistosomula (approximately 340×80 μm) which are not destroyed by proper immune response.

text: The next phase of T. regenti infection is represented by schistosomula migration in the central nervous system. This is accompanied by serious neurological malfunctions in birds that suffer from leg paralysis and balance disorders. At this stage, schistosomula feed on nervous tissue as demonstrated by detection of oligodendrocytes and neurons in the lumen of parasite’s intestine. A cysteine peptidase

text: cathepsin

text: B1 of T. regenti (TrCB1) localised in intestines of migrating schistosomula is capable of

text: myelin basic protein

text: degradation, thus probably serving for nervous tissue digestion. Nonetheless, the nervous tissue ingestion has likely only a minor pathogenic effect on the host central nervous tissue. This is underpinned by observations of leg paralysis only in immunocompromised hosts, whereas in experiments with immunocompetent mouse strains, the infected animals did not reveal any neurological disorders. The neurological symptoms originate probably in mechanical damage of the nervous tissue leading to dystrophic or even necrotic changes of neurons and axonal injury. The cause of it is large migrating schistosomula (approximately 340×80 μm) which are not destroyed by proper immune response.

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text: Pholedrine (Paredrinol, Pulsotyl, Veritol), also known as 4-hydroxy-N-methylamphetamine (4-HMA), 4-hydroxymethamphetamine, and para-hydroxymethamphetamine, is a drug that stimulates the sympathetic nervous system. It is administered as a topical eye drop form for the purpose of dilating the pupil and can be used to diagnose Horner's syndrome.

text: Pholedrine (Paredrinol, Pulsotyl, Veritol), also known as 4-hydroxy-N-methylamphetamine (4-HMA), 4-hydroxymethamphetamine, and para-hydroxymethamphetamine, is a

text: drug

text: that

text: stimulates

text: the

text: sympathetic nervous system

text: . It is administered as a

text: topical

text: eye drop

text: form

text: for the purpose of dilating the

text: pupil

text: and can be used to diagnose

text: Horner's syndrome

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text: The Liverpool Neurological Infectious Diseases Course (or NeuroID) is an annual two-day course aimed at medical professionals and students with an interest in neurological infectious diseases. The course is organised by the Liverpool Brain Infections Group, a division of the Institute of Infection and Global Health at the University of Liverpool, in collaboration with the Walton Centre NHS Foundation Trust, Alder Hey Children’s NHS Trust, Royal Liverpool University Hospital, and Liverpool School of Tropical Medicine, and is chaired by the neurologist Tom Solomon. It takes place during May at the historic Liverpool Medical Institution, in Liverpool, UK. A variety of both national and international speakers contribute to a programme which covers clinical aspects of common central nervous system infections such as meningitis and encephalitis, as well as rarer neurological infections and talks on recent advances in related research. The course is accredited by the UK Royal College of Physicians, and attracts delegates from many countries worldwide.

text: The Liverpool Neurological Infectious Diseases Course (or NeuroID) is an annual two-day course aimed at medical professionals and students with an interest in neurological infectious diseases. The course is organised by the Liverpool Brain Infections Group, a division of the Institute of Infection and Global Health at the

text: University of Liverpool

text: , in collaboration with the

text: Walton Centre

text: NHS Foundation Trust,

text: Alder Hey

text: Children’s NHS Trust,

text: Royal Liverpool University Hospital

text: , and

text: Liverpool School of Tropical Medicine

text: , and is chaired by the neurologist

text: Tom Solomon

text: . It takes place during May at the historic

text: Liverpool Medical Institution

text: , in

text: Liverpool

text: UK

text: . A variety of both national and international speakers contribute to a programme which covers clinical aspects of common central nervous system infections such as

text: meningitis

text: and

text: encephalitis

text: , as well as rarer neurological infections and talks on recent advances in related research. The course is accredited by the UK

text: Royal College of Physicians

text: , and attracts delegates from many countries worldwide.

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text: For >15 years, Dr. Trojanowski has conducted research on AD, PD, motor neuron disease, dementia with Lewy bodies (DLB), frontotemporal lobar degeneration (FTLD) and other aging related nervous system disorders. Most of his >500 publications focus on the pathobiology of neurodegenerative disorders, especially the role of abnormal protein aggregates (misfolded proteins) in these diseases. The major goal of his research now is to translate advances into understanding mechanisms of aging related neurodegenerative diseases into meaningful interventions to treat or prevent these disorders.

text: For >15 years, Dr. Trojanowski has conducted research on AD, PD, motor neuron disease,

text: dementia

text: with Lewy bodies (DLB),

text: frontotemporal lobar degeneration

text: (FTLD) and other aging related nervous system disorders. Most of his >500 publications focus on the pathobiology of neurodegenerative disorders, especially the role of abnormal protein aggregates (misfolded proteins) in these diseases. The major goal of his research now is to translate advances into understanding mechanisms of aging related neurodegenerative diseases into meaningful interventions to treat or prevent these disorders.

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answer: Yes

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answer: Yes

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answer: 1

text: Wernicke's encephalopathy (or Wernicke's disease) refers to the presence of neurological symptoms caused by biochemical lesions of the central nervous system after exhaustion of B-vitamin reserves, in particular thiamine (vitamin B1). The condition is part of a larger group of diseases related to thiamine insufficiency, including beriberi in all its forms, and Korsakoff syndrome. When Wernicke's encephalopathy occurs simultaneously with Korsakoff syndrome it is known as Wernicke–Korsakoff syndrome.

text: Wernicke's encephalopathy (or Wernicke's disease) refers to the presence of neurological symptoms caused by biochemical lesions of the

text: central nervous system

text: after exhaustion of B-vitamin reserves, in particular

text: thiamine

text: (vitamin B1). The condition is part of a larger group of diseases related to thiamine insufficiency, including

text: beriberi

text: in all its forms, and

text: Korsakoff syndrome

text: . When Wernicke's encephalopathy occurs simultaneously with Korsakoff syndrome it is known as

text: Wernicke–Korsakoff syndrome

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answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: In the Yunani system, the root is used as a tonic for the liver and brain; effective against bronchitis, piles, lumbago, toothache, and boils of the scalp; it also is said to lessen inflammation and heal fractures. The root is said to be fragrant, bitter and useful in rheumatism and allied disorders, in which it is prescribed in a variety of forms. It is also used in the composition of several medicated oils for external application in rheumatism and diseases of the nervous system. In Chota Nagpur, the leaves are pounded into a paste and then applied to the body during fever. A compound decoction of this root is  administered in cases of Hemiplegia as some Indian physicians consider it useful in rheumatism and all nervous diseases. The leaves are pounded and the paste is applied to the body to bring down fever; the juice is dropped in the ear for the treatment of Otitis media and other inflammatory conditions. The roots are used in Dyspepsia, Bronchitis, Rheumatism, and also in fever; they are reported to possess antibacterial and anti-tubercular properties. The herb is also used for Sciatica. The leaves are used by the Santhal girls for making anklets.

text: In the

text: Yunani

text: system, the root is used as a tonic for the liver and brain; effective against bronchitis, piles, lumbago, toothache, and boils of the scalp; it also is said to lessen inflammation and heal fractures. The root is said to be fragrant, bitter and useful in rheumatism and allied disorders, in which it is prescribed in a variety of forms. It is also used in the composition of several medicated oils for external application in rheumatism and diseases of the nervous system. In Chota

text: Nagpur

text: , the leaves are pounded into a paste and then applied to the body during fever. A compound decoction of this root is  administered in cases of

text: Hemiplegia

text: as some Indian physicians consider it useful in rheumatism and all nervous diseases. The leaves are pounded and the paste is applied to the body to bring down fever; the juice is dropped in the ear for the treatment of

text: Otitis media

text: and other inflammatory conditions. The roots are used in

text: Dyspepsia

text: Bronchitis

text: Rheumatism

text: , and also in fever; they are reported to possess antibacterial and anti-tubercular properties. The herb is also used for

text: Sciatica

text: . The leaves are used by the

text: Santhal

text: girls for making anklets.

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answers: Diseases of the nervous system

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answer: Yes

answer: Yes

answer: Yes

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answer: 1

text: Central nervous system diseases, also known as central nervous system disorders, are a group of neurological disorders that affect the structure or function of either the spinal cord or brain, which collectively form the central nervous system (CNS).

text: Central nervous system diseases, also known as central nervous system disorders, are a group of

text: neurological disorder

text: s that affect the structure or function of either the

text: spinal cord

text: or

text: brain

text: , which collectively form the

text: central nervous system

text: (CNS).

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answers: Central nervous system diseases

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answers: Central nervous system

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answers: neurological disorders

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 2

text: Granulomatous amoebic encephalitis is caused by amoeba infection of the central nervous system. It is characterized by neurological symptoms including headache, seizures, and mental status abnormalities. These worsen progressively over weeks to months, leading to death in most patients. Infection is generally associated with underlying conditions such as immunodeficiency, diabetes, malignancies, malnutrition, systemic lupus erythematosus, and alcoholism. The parasite enters the body through cuts in the skin or by being inhaled into the upper respiratory tract. The parasite then spreads through the blood into the central nervous system. Acanthamoeba crosses the blood–brain barrier by means that are not yet understood. Subsequent invasion of the connective tissue and induction of pro-inflammatory responses leads to neuronal damage that can be fatal within days. Pure granulomatous lesions are rare in patients with AIDS and other related immunodeficiency states as the patients do not have adequate numbers of CD+ve T-cells to mount a granulomatous response to Acanthamoeba infection in CNS and other organ and tissues. A perivascular cuffing with amoebae in necrotic tissue is usual finding in the AIDS and related T-cell immunodeficiency conditions.  A post mortem biopsy reveals severe oedema and hemorrhagic necrosis. A patient who has contracted this illness usually displays subacute symptoms, including altered mental status, headaches, fever, neck stiffness, seizures, focal neurological signs (such as cranial nerve palsies and coma), all leading to death within one week to several months. Due to the rarity of this parasite and a lack of knowledge, there are currently no good diagnoses or treatments for Acanthamoeba infection. Acanthamoeba keratitis cases in the past, when were managed by atropine given as an adjuvant therapy without anti-parasitic drugs added to the regime, had been reported to halt the vision loss. Recent publications debate these successes related to an action of Atropine on muscarinic receptors on Acanthamoeba spp.

text: Granulomatous amoebic encephalitis is caused by amoeba infection of the central nervous system. It is characterized by

text: neurological symptoms

text: including headache, seizures, and mental status abnormalities. These worsen progressively over weeks to months, leading to death in most patients. Infection is generally associated with underlying conditions such as

text: immunodeficiency

text: , diabetes, malignancies, malnutrition,

text: systemic lupus erythematosus

text: , and alcoholism. The parasite enters the body through cuts in the skin or by being inhaled into the upper respiratory tract. The parasite then spreads through the blood into the central nervous system. Acanthamoeba crosses the

text: blood–brain barrier

text: by means that are not yet understood. Subsequent invasion of the connective tissue and induction of pro-inflammatory responses leads to neuronal damage that can be fatal within days. Pure granulomatous lesions are rare in patients with AIDS and other related immunodeficiency states as the patients do not have adequate numbers of CD+ve T-cells to mount a granulomatous response to Acanthamoeba infection in CNS and other organ and tissues. A perivascular cuffing with amoebae in necrotic tissue is usual finding in the AIDS and related T-cell immunodeficiency conditions.  A post mortem

text: biopsy

text: reveals severe

text: oedema

text: and

text: hemorrhagic

text: necrosis

text: . A patient who has contracted this illness usually displays subacute symptoms, including altered mental status, headaches, fever, neck stiffness, seizures, focal neurological signs (such as cranial nerve palsies and coma), all leading to death within one week to several months. Due to the rarity of this parasite and a lack of knowledge, there are currently no good diagnoses or treatments for Acanthamoeba infection. Acanthamoeba keratitis cases in the past, when were managed by atropine given as an adjuvant therapy without anti-parasitic drugs added to the regime, had been reported to halt the vision loss. Recent publications debate these successes related to an action of Atropine on muscarinic receptors on Acanthamoeba spp.

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answers: neurological symptoms including headache

answers: NDQ_010490

answers: cns diseases can be caused by bacteria and viruses getting into the brain or spinal cord

answers: NDQ_010494

answers: encephalitis

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answers: lupus erythematosus

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answers: cranial nerve palsies and coma

answers: NDQ_010497

answers: encephalitis

answers: NDQ_010498

answers: Granulomatous amoebic encephalitis

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: Ochroconis gallopava was thought to cause epidemic fatal encephalitis only in fowls, turkeys, and poults at first, however recently it has been increasingly recognized as a pathogen for human following solid organ transplantation, such as kidney, liver, heart, and lung. The first O. gallopava infection in human was reported in 1986. Prior to 1986, O. gallopava was known as an agent of phaeohyphomycosis and fatal encephalitis mainly in poultry. Even though reports of human infection by this species have increased, O. gallopava remains an extremely uncommon agent of human disease. When it does occur in humans, a wide range of sites may become involved, including the lung, heart, brain, the superficial cutaneous or subcutaneous areas, and other parts of the body. Infection accompanies brain involvement, respiratory tract involvement, pulmonary infections, and skin infections and many others. O. gallopava infection can be divided into fatal disease and manageable disease. Once the fungus penetrates into the central nervous system and involves the brain, the probability of cure by antifungal therapy falls exceedingly. When the fungal infection only concerns with systemic involvement except the brain, the probability of cure is higher. In serious infections, the typical entry point is thought to be the   respiratory tract. Like other melanized fungi, the clinical presentation of O. gallopava is phaeohyphomycosis characterized by darkly colored lesions in affected tissues, acute or chronic inflammation, microabscesses, fibrosis, granulomas, and necrosis. The onset of symptoms in immunosuppressed individuals after transplantation is very slow, almost several months to years after organ transplantation surgery, but the infection can severely damage the host once it appears. When implicated, the median onset of the infection in organ transplanted patients is 22 months after surgery. Very rarely, O. gallopava infection has been observed in immunologically normal people. Unlike infections in immunocompromised individuals, these cases tend to have a high recovery rate.

text: Ochroconis gallopava was thought to cause epidemic fatal

text: encephalitis

text: only in fowls, turkeys, and poults at first, however recently it has been increasingly recognized as a pathogen for human following solid organ transplantation, such as kidney, liver, heart, and lung. The first O. gallopava infection in human was reported in 1986. Prior to 1986, O. gallopava was known as an agent of

text: phaeohyphomycosis

text: and fatal

text: encephalitis

text: mainly in poultry. Even though reports of human infection by this species have increased, O. gallopava remains an extremely uncommon agent of human disease. When it does occur in humans, a wide range of sites may become involved, including the lung, heart, brain, the superficial

text: cutaneous

text: or

text: subcutaneous

text: areas, and other parts of the body. Infection accompanies brain involvement, respiratory tract involvement, pulmonary infections, and skin infections and many others. O. gallopava infection can be divided into fatal disease and manageable disease. Once the fungus penetrates into the central nervous system and involves the brain, the probability of cure by antifungal therapy falls exceedingly. When the fungal infection only concerns with systemic involvement except the brain, the probability of cure is higher. In serious infections, the typical entry point is thought to be the   respiratory tract. Like other melanized fungi, the clinical presentation of O. gallopava is phaeohyphomycosis characterized by darkly colored lesions in affected tissues, acute or chronic inflammation, microabscesses, fibrosis,

text: granuloma

text: s, and necrosis. The onset of symptoms in immunosuppressed individuals after transplantation is very slow, almost several months to years after organ transplantation surgery, but the infection can severely damage the host once it appears. When implicated, the median onset of the infection in organ transplanted patients is 22 months after surgery. Very rarely, O. gallopava infection has been observed in

text: immunologically normal

text: people. Unlike infections in immunocompromised individuals, these cases tend to have a high recovery rate.

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answers: Once the fungus penetrates into the central nervous system and involves the brain

answers: NDQ_010490

answers: cns diseases can be caused by bacteria and viruses getting into the brain or spinal cord.

answers: NDQ_010494

answers: encephalitis

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answers: encephalitis

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answers: encephalitis

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answers: encephalitis

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answers: encephalitis

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: The function of nervous tissue is to form the communication network of the nervous system by conducting electric signals across tissue. In the CNS, grey matter, which contains the synapses, is important for information processing. White matter, containing myelinated axons, connects and facilitates nerve impulse between grey matter areas in the CNS.In the PNS, the ganglion tissue, containing the cell bodies and dendrites, contain relay points for nerve tissue impulses. The nerve tissue, containing myelinated axons bundles, carry action potential/nerve impulses.

text: The function of nervous tissue is to form the communication network of the

text: nervous system

text: by conducting electric signals across tissue. In the CNS, grey matter, which contains the

text: synapses

text: , is important for information processing. White matter, containing myelinated axons, connects and facilitates nerve impulse between grey matter areas in the CNS.In the PNS, the ganglion tissue, containing the cell bodies and dendrites, contain relay points for

text: nerve tissue

text: impulses. The nerve tissue, containing myelinated axons bundles, carry action potential/nerve impulses.

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answers: It does not say

answers: NDQ_010490

answers: In the CNS

answers: NDQ_010494

answers: CNS

answers: NDQ_010495

answers: CNS

answers: NDQ_010496

answers: CNS

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answers: CNS

answers: NDQ_010498

answers: CNS

answer: No

answer: No

answer: No

answer: No

answer: No

answer: 1

text: Central nervous system involvement, as described above, most often takes the form of meningeal leukemia, or invasion of the subarachnoid space by leukemic cells. This condition is usually considered separately from chloroma, as it requires different treatment modalities. True chloromas (i.e. solid leukemic tumors) of the central nervous system are exceedingly rare, but has been described.

text: Central nervous system

text: involvement, as described above, most often takes the form of meningeal leukemia, or invasion of the

text: subarachnoid space

text: by leukemic cells. This condition is usually considered separately from chloroma, as it requires different treatment modalities. True chloromas (i.e. solid leukemic tumors) of the central nervous system are exceedingly rare, but has been described.

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text: The investigations completed at the Eberhard Karls University of Tuebingen in 2003 by Alfred Czarnetzki, Carsten M. Pusch and Erwin Schwaderer, showed that the owner of the skull suffered from a meningioma, which is an arachnoid tumor. Meningiomas are a diverse set of tumors that arise from the meninges, which is the membranous layers surrounding the central nervous system.

text: The investigations completed at the Eberhard Karls University of Tuebingen in 2003 by Alfred Czarnetzki, Carsten M. Pusch and Erwin Schwaderer, showed that the owner of the skull suffered from a meningioma, which is an arachnoid tumor. Meningiomas are a diverse set of tumors that arise from the meninges, which is the membranous layers surrounding the central nervous system.

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answers: Meningitis

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answers: meningitis

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answer: 1

text: Anaplastic astrocytoma, Astrocytoma, Central neurocytoma, Choroid plexus carcinoma, Choroid plexus papilloma, Choroid plexus tumor, Dysembryoplastic neuroepithelial tumour, Ependymal tumor, Fibrillary astrocytoma, Giant-cell glioblastoma, Glioblastoma multiforme, Gliomatosis cerebri, Gliosarcoma, Hemangiopericytoma, Medulloblastoma, Medulloepithelioma, Meningeal carcinomatosis, Neuroblastoma, Neurocytoma, Oligoastrocytoma, Oligodendroglioma, Optic nerve sheath meningioma, Pediatric ependymoma, Pilocytic astrocytoma, Pinealoblastoma, Pineocytoma, Pleomorphic anaplastic neuroblastoma, Pleomorphic xanthoastrocytoma, Primary central nervous system lymphoma, Sphenoid wing meningioma, Subependymal giant cell astrocytoma, Subependymoma, Trilateral retinoblastoma.

text: Anaplastic astrocytoma

text: Astrocytoma

text: Central neurocytoma

text: Choroid plexus carcinoma

text: Choroid plexus papilloma

text: Choroid plexus tumor

text: Dysembryoplastic neuroepithelial tumour

text: Ependymal tumor

text: Fibrillary astrocytoma

text: Giant-cell glioblastoma

text: Glioblastoma multiforme

text: Gliomatosis cerebri

text: Gliosarcoma

text: Hemangiopericytoma

text: Medulloblastoma

text: Medulloepithelioma

text: Meningeal carcinomatosis

text: Neuroblastoma

text: Neurocytoma

text: Oligoastrocytoma

text: Oligodendroglioma

text: Optic nerve sheath meningioma

text: Pediatric ependymoma

text: Pilocytic astrocytoma

text: Pinealoblastoma

text: Pineocytoma

text: Pleomorphic anaplastic neuroblastoma

text: Pleomorphic xanthoastrocytoma

text: Primary central nervous system lymphoma

text: Sphenoid wing meningioma

text: Subependymal giant cell astrocytoma

text: Subependymoma

text: Trilateral retinoblastoma

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answer: Yes

answer: Yes

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text: Initial infection by the virus and primary spread of the virus causes the onset of non-specific symptoms such as headache and fever. Secondary spread and the multiplication of the virus in the CNS (central nervous system) causes symptoms such as stiff neck, lethargy and seizures.  It then can result in encephalitis, when inflammation of the brain, produced by infection by the virus, damages nerve cells, which affects signaling of the brain to the body.

text: Initial infection by the virus and primary spread of the virus causes the onset of

text: non-specific symptom

text: s such as headache and fever. Secondary spread and the multiplication of the virus in the CNS (central nervous system) causes symptoms such as stiff neck,

text: lethargy

text: and seizures.  It then can result in encephalitis, when inflammation of the brain, produced by infection by the virus, damages nerve cells, which affects signaling of the brain to the body.

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answers: encephalitis

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answers: encephalitis

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answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: No

answer: 1

text: Putnam was a founder member of the American Neurological Association in December 1874, and was its president in 1888, and also a founding member of the American Psychoanalytical Association in 1911, being its first president and continuing to hold the post the following year. He was appointed Professor of Diseases of the Nervous System at Harvard in 1893 and continued to his retirement in 1912.

text: Putnam was a founder member of the

text: American Neurological Association

text: in December 1874, and was its president in 1888, and also a founding member of the American Psychoanalytical Association in 1911, being its first president and continuing to hold the post the following year. He was appointed Professor of Diseases of the Nervous System at Harvard in 1893 and continued to his retirement in 1912.

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answer: Yes

answer: Yes

answer: Yes

answer: No

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text: The most important of Krka’s therapeutic groups include treatments for: cardiovascular diseases, alimentary tract diseases and metabolic disorders, and central nervous system diseases. They are followed by the medicines for the systemic treatment of infections, the medicines for the treatment of diseases of the blood and blood-forming organs, and the medicines for urinary tract diseases as well as the medicines for respiratory system diseases.

text: The most important of Krka’s therapeutic groups include treatments for:

text: cardiovascular disease

text: s,

entity_name: ICD-10 Chapter XI: Diseases of the digestive system

text: alimentary tract diseases

text: and metabolic disorders, and

text: central nervous system diseases

text: . They are followed by the medicines for the systemic treatment of infections, the medicines for the treatment of diseases of the blood and blood-forming organs, and the medicines for

entity_name: ICD-10 Chapter XIV: Diseases of the genitourinary system

text: urinary tract diseases

text: as well as the medicines for

text: respiratory system

text: diseases.

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answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: Mortality rates are less than 1% and most patients with encephalitis clinical symptoms recover completely; however, 20% of patients develop behavioral problems or recurrent seizures.Encephalitis disease in general occurs almost exclusively in children from 6 months to 16 years old.

text: Mortality rates are less than 1% and most patients with encephalitis clinical symptoms recover completely; however, 20% of patients develop behavioral problems or recurrent seizures.Encephalitis disease in general occurs almost exclusively in children from 6 months to 16 years old.

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answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: A peripheral nerve interface is the bridge between the peripheral nervous system and a computer interface which serves as a bi‐directional information transducer recording and sending signals between the human body and a machine processor. Interfaces to the nervous system usually take the form of electrodes for stimulation and recording, though chemical stimulation and sensing are possible. Research in this area is focused on developing peripheral nerve interfaces for the restoration of function following disease or injury to minimize associated losses. Peripheral nerve interfaces also enable electrical stimulation and recording of the peripheral nervous system to study the form and function of the peripheral nervous system. Many researchers also focus in the area of neuroprosthesis, linking the human nervous system to bionics in order to mimic natural sensorimotor control and function. Successful implantation of peripheral nerve interfaces depend on a number of factors which include appropriate indication,  perioperative testing, differentiated planning, and functional training. Typically microelectrode devices are implanted adjacent to, around or within the nerve trunk to establish contact with the peripheral nervous system. Different approaches may be used depending on the type of signal desired and attainable.

text: A peripheral nerve interface is the bridge between the

text: peripheral nervous system

text: and a

text: computer interface

text: which serves as a bi‐directional information

text: transducer

text: recording and sending signals between the human body and a

text: machine processor

text: . Interfaces to the nervous system usually take the form of electrodes for stimulation and recording, though chemical stimulation and sensing are possible. Research in this area is focused on developing peripheral nerve interfaces for the restoration of function following disease or injury to minimize associated losses. Peripheral nerve interfaces also enable electrical stimulation and recording of the peripheral nervous system to study the form and function of the peripheral nervous system. Many researchers also focus in the area of

text: neuroprosthesis

text: , linking the human nervous system to

text: bionics

text: in order to mimic natural

text: sensorimotor control

text: and function. Successful implantation of peripheral nerve interfaces depend on a number of factors which include appropriate

text: indication

text: perioperative testing

text: differentiated planning

text: , and functional training. Typically

text: microelectrode

text: devices are implanted adjacent to, around or within the nerve trunk to establish contact with the peripheral nervous system. Different approaches may be used depending on the type of signal desired and attainable.

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answer: No

answer: No

answer: No

answer: No

answer: No

answer: 1

text: He co-edited the standard work, Manual of Diseases of the Nervous System, with Sir William Gowers, and also published Paralysis and other Nervous Diseases of Childhood and Early Life in 1905.

text: He co-edited the standard work, Manual of Diseases of the Nervous System, with

text: Sir William Gowers

text: , and also published Paralysis and other Nervous Diseases of Childhood and Early Life in 1905.

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answers: Paralysis

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answers: Paralysis

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answers: Paralysis

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: The fundamental hypothesis of psychoneuroimmunology is concisely that the way people think and how they feel directly influences the electrochemistry of the brain and central nervous system, which in turn has a significant influence on the immune system and its capacity to defend the body against disease infection, and ill health. Meanwhile, the immune system affects brain chemistry and its electrical activity, which in turn has a considerable effect on the way we think and feel.

text: The fundamental hypothesis of psychoneuroimmunology is concisely that the way people think and how they feel directly influences the

text: electrochemistry

text: of the brain and

text: central nervous system

text: , which in turn has a significant influence on the

text: immune system

text: and its capacity to defend the body against

text: disease

text: infection

text: , and ill health. Meanwhile, the immune system affects

text: brain

text: chemistry

text: and its electrical activity, which in turn has a considerable effect on the way we think and feel.

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text: Similar diseases that are spread by mosquitoes include: Western and Eastern equine encephalitis, Japanese encephalitis, Saint Louis encephalitis and West Nile virus.

text: Similar diseases that are spread by mosquitoes include:

text: Western

text: and

text: Eastern

text: equine encephalitis,

text: Japanese encephalitis

text: Saint Louis encephalitis

text: and

text: West Nile virus

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text: There are many central nervous system disorders that are associated with olfactory dysfunction.  Most of these dysfunctions classify as degenerative neuropsychiatric disorders.  Some of these diseases are: Alzheimer's Disease, Parkinson's Disease, Huntington's Disease, Korsakoff's Psychosis, Schizophrenia, Congenital Anosmia, Head Trauma, Brain Tumors, Acquired Immunodeficiency Syndrome(AIDS), and Multiple Sclerosis.

text: There are many

text: central nervous system disorders

text: that are associated with

text: olfactory dysfunction

text: .  Most of these dysfunctions classify as degenerative

text: neuropsychiatric disorders

text: .  Some of these diseases are:

text: Alzheimer's Disease

text: Parkinson's Disease

text: Huntington's Disease

text: Korsakoff's Psychosis

text: Schizophrenia

text: Congenital Anosmia

text: Head Trauma

text: Brain Tumors

text: Acquired Immunodeficiency Syndrome

text: (AIDS), and

text: Multiple Sclerosis

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answer: Yes

answer: Yes

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answer: 1

text: Humans are incidental hosts of this roundworm, and may become infected through ingestion of larvae in raw or undercooked snails or other vectors, or from contaminated water and vegetables. The larvae are then transported via the blood to the central nervous system (CNS), where they are the most common cause of eosinophilic meningitis, a serious condition that can lead to death or permanent brain and nerve damage. Angiostrongyliasis is an infection of increasing public health importance as globalization contributes to the geographic spread of the disease.

text: Humans are incidental hosts of this roundworm, and may become infected through ingestion of larvae in raw or undercooked snails or other vectors, or from contaminated water and vegetables. The larvae are then transported via the blood to the

text: central nervous system

text: (CNS), where they are the most common cause of

text: eosinophilic meningitis

text: , a serious condition that can lead to death or permanent brain and nerve damage. Angiostrongyliasis is an infection of increasing public health importance as

text: globalization

text: contributes to the geographic spread of the disease.

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answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: No

answer: 1

text: If the cancer has central nervous system involvement, or with meningeal disease, intrathecal chemotherapy may be administered.

text: If the cancer has

text: central nervous system

text: involvement, or with meningeal disease,

text: intrathecal

text: chemotherapy may be administered.

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text: Between 2011 and 2013, three German breeders of variegated squirrels (Sciurus variegatoides) had encephalitis with similar clinical signs and died 2 to 4 months after onset of the clinical symptoms. Genomic analysis found a previously unknown bornavirus in a contact squirrel and in brain tissue from the three men, the researchers reported, and it is the "likely causative agent" in their deaths. Prior to this, Bornavirus species were not thought to be responsible for human diseases.

text: Between 2011 and 2013, three German breeders of

text: variegated squirrel

text: s (Sciurus variegatoides) had encephalitis with similar clinical signs and died 2 to 4 months after onset of the clinical symptoms. Genomic analysis found a previously unknown bornavirus in a contact squirrel and in brain tissue from the three men, the researchers reported, and it is the "likely causative agent" in their deaths. Prior to this, Bornavirus species were not thought to be responsible for human diseases.

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answer: 1

text: Central nervous system toxicity was first described by Paul Bert in 1878. He showed that oxygen was toxic to insects, arachnids, myriapods, molluscs, earthworms, fungi, germinating seeds, birds, and other animals. Central nervous system toxicity may be referred to as the "Paul Bert effect".

text: Central nervous system toxicity was first described by

text: Paul Bert

text: in 1878. He showed that oxygen was toxic to insects,

text: arachnids

text: myriapods

text: , molluscs, earthworms, fungi, germinating seeds, birds, and other animals. Central nervous system toxicity may be referred to as the "Paul Bert effect".

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answers: Central nervous system

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answers: Central nervous system toxicity

answer: No

answer: No

answer: No

answer: No

answer: No

answer: 1

text: Japanese encephalitis (JE), formerly known as Japanese B encephalitis to distinguish it from Economo's A encephalitis—is a disease caused by the mosquito-borne Japanese encephalitis virus (JEV).

text: Japanese encephalitis (JE), formerly known as Japanese B encephalitis to distinguish it from

text: Economo

text: 's A encephalitis—is a disease caused by the

text: mosquito

text: -borne

text: Japanese encephalitis virus

text: (JEV).

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answers: 2: The answer has limited relevance and completeness, with significant gaps or inaccuracy.

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answers: Japanese encephalitis

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answers: encephalitis

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answers: Japanese encephalitis

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: Neuropathy, ataxia, and retinitis pigmentosa, also known as NARP syndrome, is a rare disease with mitochondrial inheritance that causes a variety of signs and symptoms chiefly affecting the nervous system Beginning in childhood or early adulthood, most people with NARP experience numbness, tingling, or pain in the arms and legs (sensory neuropathy); muscle weakness; and problems with balance and coordination (ataxia). Many affected individuals also have vision loss caused by changes in the light-sensitive tissue that lines the back of the eye (the retina). In some cases, the vision loss results from a condition called retinitis pigmentosa. This eye disease causes the light-sensing cells of the retina gradually to deteriorate.

text: Neuropathy, ataxia, and retinitis pigmentosa, also known as NARP syndrome, is a rare disease with mitochondrial inheritance that causes a variety of signs and symptoms chiefly affecting the

text: nervous system

text: Beginning in childhood or early adulthood, most people with NARP experience numbness, tingling, or pain in the arms and legs (

text: sensory neuropathy

text: ); muscle weakness; and problems with balance and coordination (

text: ataxia

text: ). Many affected individuals also have vision loss caused by changes in the light-sensitive tissue that lines the back of the eye (the

text: retina

text: ). In some cases, the vision loss results from a condition called

text: retinitis pigmentosa

text: . This eye disease causes the light-sensing cells of the retina gradually to deteriorate.

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text: The enteric nervous system (ENS) or intrinsic nervous system is one of the main divisions of the nervous system and consists of a mesh-like system of neurons that governs the function of the gastrointestinal system. It is now usually referred to as separate from the autonomic nervous system since it has its own independent reflex activity.

text: The enteric nervous system (ENS) or intrinsic nervous system is one of the main divisions of the

text: nervous system

text: and consists of a mesh-like system of

text: neuron

text: s that governs the function of the

text: gastrointestinal system

text: . It is now usually referred to as separate from the

text: autonomic nervous system

text: since it has its own independent reflex activity.

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text: A vaccine for horses (ATCvet code: ) based on killed viruses exists; some zoos have given this vaccine to their birds, although its effectiveness is unknown. Dogs and cats show few if any signs of infection. There have been no known cases of direct canine-human or feline-human transmission; although these pets can become infected, it is unlikely they are, in turn, capable of infecting native mosquitoes and thus continuing the disease cycle.AMD3100, which had been proposed as an antiretroviral drug for HIV, has shown promise against West Nile encephalitis. Morpholino antisense oligos conjugated to cell penetrating peptides have been shown to partially protect mice from WNV disease. There have also been attempts to treat infections using ribavirin, intravenous immunoglobulin, or alpha interferon. GenoMed, a U.S. biotech company, has found that blocking angiotensin II can treat the "cytokine storm" of West Nile virus encephalitis as well as other viruses.

text: A vaccine for horses (

entity_name: Anatomical Therapeutic Chemical Classification System

text: ATCvet

text: code: ) based on killed viruses exists; some

text: zoo

text: s have given this vaccine to their birds, although its effectiveness is unknown. Dogs and cats show few if any signs of infection. There have been no known cases of direct canine-human or feline-human transmission; although these pets can become infected, it is unlikely they are, in turn, capable of infecting native mosquitoes and thus continuing the disease cycle.

text: , which had been proposed as an antiretroviral drug for HIV, has shown promise against West Nile encephalitis.

text: Morpholino

text: antisense oligos conjugated to

text: cell penetrating peptide

text: s have been shown to partially protect mice from WNV disease. There have also been attempts to treat infections using

text: ribavirin

text: , intravenous

text: immunoglobulin

text: , or

text: alpha interferon

text: . GenoMed, a U.S. biotech company, has found that blocking angiotensin II can treat the "

text: cytokine storm

text: " of West Nile virus encephalitis as well as other viruses.

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answer: Yes

answer: Yes

answer: No

answer: Yes

answer: No

answer: 1

text: Currently, the role that the meningeal lymphatics play in neurological disease is yet to be explored. However, there is some speculation that they may contribute to autoimmune and inflammatory diseases of the CNS due to their role in connecting the immune and nervous systems.

text: Currently, the role that the meningeal lymphatics play in neurological disease is yet to be explored. However, there is some speculation that they may contribute to autoimmune and inflammatory diseases of the CNS due to their role in connecting the

text: immune

text: and

text: nervous

text: systems.

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answers: autoimmune

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answers: neurological

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: Regeneration of the central nervous system is much poorer than the peripheral nervous system and does not show the same capacity to regenerate itself. This is primarily due to the multifaceted nature of spinal cord injury. The multifaceted nature of spinal cord injuries presents a major challenge to therapeutic development however, as primary mechanical trauma to the cord induces secondary injury consisting of a complex cascade of molecular events that lead to the loss of the conductive myelin sheath (myelin degeneration) and the formation of an inhibitory glial scar.

text: Regeneration of the central nervous system is much poorer than the peripheral nervous system and does not show the same capacity to regenerate itself. This is primarily due to the multifaceted nature of spinal cord injury. The multifaceted nature of spinal cord injuries presents a major challenge to therapeutic development however, as primary mechanical trauma to the cord induces secondary injury consisting of a complex cascade of molecular events that lead to the loss of the conductive myelin sheath (myelin degeneration) and the formation of an inhibitory glial scar.

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text: The encephalitis is characterized by fever, drowsiness, and lack of mental alertness and orientation. Seizures occur in 50% of children.  Focal neurologic signs such as irregular and abnormal reflexes develop in 20% of children.  10% of patients develop coma.  The total duration of illness rarely exceeds 10–14 days.  Recurrent unprovoked seizures occur even after the illness has passed develops in 20% of patients, especially those who had seizures during the acute illness.  In adults, infection is asymptomatic, which means that the patient is a carrier of the infection, but experiences no symptoms or only mild feverish illness.

text: The encephalitis is characterized by fever, drowsiness, and lack of mental alertness and orientation. Seizures occur in 50% of children.  Focal neurologic signs such as irregular and abnormal reflexes develop in 20% of children.  10% of patients develop coma.  The total duration of illness rarely exceeds 10–14 days.  Recurrent unprovoked seizures occur even after the illness has passed develops in 20% of patients, especially those who had seizures during the acute illness.  In adults, infection is asymptomatic, which means that the patient is a carrier of the infection, but experiences no symptoms or only mild feverish illness.

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text: The theoretical basis of Burckhardt's action rested on three propositions. The first was that mental illness had a physical basis and that disordered minds were merely a reflection of disordered brains. Next, the associationist viewpoint of nerve functioning which conceived the nervous system as operating according to the following threefold division of labor: an input (or sensory or afferent) system, a connecting system which processed information and an output (or efferent or motor) system. The final assumption of Buckhardt's was that the brain was modular which meant that each mental module or mental faculty could be linked to a specific location in the brain. In accordance with such a viewpoint, Buckhardt postulated that lesions in specific areas of the brain might impact behavior in a specific manner. In other words, he thought that by cutting the connecting system, or second association state of brain's system of communication troubling symptoms might be alleviated without compromising either the nervous system's input or output systems. The procedure was aimed at relieving symptoms, not at curing a given mental disease. Thus, he wrote in 1891:

text: The theoretical basis of Burckhardt's action rested on three propositions. The first was that mental illness had a physical basis and that disordered minds were merely a reflection of disordered brains. Next, the

text: associationist

text: viewpoint of nerve functioning which conceived the nervous system as operating according to the following threefold division of labor: an input (or sensory or

text: afferent

text: ) system, a connecting system which processed information and an output (or

text: efferent

text: or motor) system. The final assumption of Buckhardt's was that the brain was modular which meant that each mental module or mental faculty could be linked to a specific location in the brain. In accordance with such a viewpoint, Buckhardt postulated that

text: lesions

text: in specific areas of the brain might impact behavior in a specific manner. In other words, he thought that by cutting the connecting system, or second association state of brain's system of communication troubling symptoms might be alleviated without compromising either the nervous system's input or output systems. The procedure was aimed at relieving symptoms, not at curing a given mental disease. Thus, he wrote in 1891:

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text: Reyes, along with the other "Filipino invasion" players revolutionized the way pool is played by their introduction to the sport of pinpoint precision kicking (going into a rail with the cue ball and then hitting an object ball). Reyes' ability to "kick safe" and to kick balls into intended pockets is legendary. This ability, coupled with his superb skill at other aspects of the game, led U.S. professionals to give him the appellation "The Magician." Before Reyes and his compatriots came over to the U.S., no one there had seen anything like their kicking skill set.

text: Reyes, along with the other "Filipino invasion" players revolutionized the way pool is played by their introduction to the sport of pinpoint precision kicking (going into a rail with the cue ball and then hitting an object ball). Reyes' ability to "kick safe" and to kick balls into intended pockets is legendary. This ability, coupled with his superb skill at other aspects of the game, led U.S. professionals to give him the appellation "The Magician." Before Reyes and his compatriots came over to the U.S., no one there had seen anything like their kicking skill set.

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text: Oocysts passed in the feces of the definitive host, such as canines or canids, are ingested by an intermediate host, such as cattle. After ingestion of an oocyst, motile and rapidly dividing tachyzoites are released. These tachyzoites disseminate throughout the host, and in response to the host immune response, differentiate into bradyzoites, which form cysts in muscle and tissue. Formation of these cysts results in chronic infection of the intermediate host.  Ingestion of infected intermediate host tissue by the definitive host completes the life cycle. A second route of transmission is the congenital transmission from mother to offspring.   Transplacental transmission (passage from mother to offspring during pregnancy) has also been shown to occur in dogs, cats, sheep and cattle. If the intermediate host acquires the disease during pregnancy, it activates these cysts, and active infection often causes spontaneous abortion.  In addition, if the aborted fetus and membranes are then eaten by the definitive host, they cause further infection and the cycle is complete.    Other carnivores, for example the red fox (Vulpes vulpes), may also be intermediate hosts, but they are not known to be definitive hosts.  Neospora caninum does not appear to be infectious to humans.  In dogs, Neospora caninum can cause neurological signs, especially in congenitally infected puppies, where it can form cysts in the central nervous system.

text: Oocyst

text: s passed in the feces of the

text: definitive host

text: , such as canines or canids, are ingested by an

text: intermediate host

text: , such as cattle. After ingestion of an oocyst, motile and rapidly dividing tachyzoites are released. These tachyzoites disseminate throughout the host, and in response to the host immune response, differentiate into bradyzoites, which form cysts in muscle and tissue. Formation of these cysts results in chronic infection of the intermediate host.  Ingestion of infected intermediate host tissue by the definitive host completes the life cycle. A second route of transmission is the congenital transmission from mother to offspring.

text: Transplacental

text: transmission (passage from mother to offspring during pregnancy) has also been shown to occur in dogs, cats, sheep and cattle. If the intermediate host acquires the disease during pregnancy, it activates these cysts, and active infection often causes spontaneous abortion.  In addition, if the aborted fetus and membranes are then eaten by the definitive host, they cause further infection and the cycle is complete.    Other

text: carnivore

text: s, for example the

text: red fox

text: (Vulpes vulpes), may also be intermediate hosts, but they are not known to be definitive hosts.  Neospora caninum does not appear to be infectious to humans.  In dogs, Neospora caninum can cause neurological signs, especially in

text: congenital

text: ly infected puppies, where it can form cysts in the

text: central nervous system

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answer: No

answer: Yes

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text: M. morganii  can produce the enzyme catalase, so is able to convert hydrogen peroxide to water and oxygen. This is a common enzyme found in most living organisms. In addition, it is indole test-positive representing this organism can split tryptophan to indole, pyruvate, and ammonia. Methyl red tests positive in M.morganii, an indicator dye that turns red in acidic solutions. Although a rare human pathogen, M. morganii has been reported as a cause of urinary tract infections, nosocomial surgical wound infections, peritonitis, central nervous system infection, endophthalmitis, pneumonia, chorioamnionitis, neonatal sepsis, pyomyositis, necrotizing fasciitis, and arthritis. Numerous cases of nosocomial infection have been described, usually as postsurgical wound infections or urinary tract infections. Patients in whom bacteremia develops are typically immunocompromised, diabetic, or elderly, or have at least one serious underlying disease.

text: M. morganii  can produce the enzyme

text: catalase

text: , so is able to convert

text: hydrogen peroxide

text: to water and oxygen. This is a common enzyme found in most living organisms. In addition, it is

text: indole test

text: -positive representing this organism can split tryptophan to indole, pyruvate, and ammonia.

text: Methyl red

text: tests positive in M.morganii, an indicator dye that turns red in acidic solutions. Although a rare human pathogen, M. morganii has been reported as a cause of urinary tract infections, nosocomial surgical wound infections, peritonitis, central nervous system infection, endophthalmitis, pneumonia, chorioamnionitis, neonatal sepsis, pyomyositis, necrotizing fasciitis, and arthritis. Numerous cases of nosocomial infection have been described, usually as postsurgical wound infections or urinary tract infections. Patients in whom bacteremia develops are typically immunocompromised, diabetic, or elderly, or have at least one serious underlying disease.

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text: Progressive multifocal leukoencephalopathy (PML) is a demyelinating disease of the central nervous system that is caused by reactivation of a latent papovavirus (the JC polyomavirus) infection, that can cross the BBB. It affects immune-compromised patients and it is usually seen with patients suffering from AIDS.

text: Progressive multifocal leukoencephalopathy

text: (PML) is a demyelinating disease of the central nervous system that is caused by reactivation of a latent

text: papovavirus

text: (the

text: JC polyomavirus

text: ) infection, that can cross the BBB. It affects immune-compromised patients and it is usually seen with patients suffering from

text: AIDS

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answers: BBB

answers: NDQ_010494

answers: demyelinating disease of the central nervous system

answers: NDQ_010495

answers: AIDS

answers: NDQ_010496

answers: AIDS

answers: NDQ_010497

answers: AIDS

answers: NDQ_010498

answers: AIDS

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 2

text: Inflammation in the body may lead to effects on the brain via the blood-brain barrier (BBB). During systemic inflammation, whether in the form of infection or sterile inflammation, the BBB may undergo changes which may be disruptive or non-disruptive. Such changes may be part of a productive host response to systemic inflammation, or may lead to deleterious consequences within the central nervous system. These BBB changes likely play a role in the generation of sickness behaviour during systemic infection (why we feel 'unwell' when we have an infection). These changes may also induce or accelerate disease within the brain, for example in multiple sclerosis and Alzheimer's disease, and may be part of the reason why patients with these conditions can deteriorate during an infection. Also, in patients with neurological disease the BBB may be abnormally sensitive to the effects of systemic inflammation.

text: Inflammation in the body may lead to effects on the brain via the blood-brain barrier (BBB). During systemic inflammation, whether in the form of infection or sterile inflammation, the BBB may undergo changes which may be disruptive or non-disruptive. Such changes may be part of a productive host response to systemic inflammation, or may lead to deleterious consequences within the central nervous system. These BBB changes likely play a role in the generation of

text: sickness behaviour

text: during systemic infection (why we feel 'unwell' when we have an infection). These changes may also induce or accelerate disease within the brain, for example in

text: multiple sclerosis

text: and

text: Alzheimer's disease

text: , and may be part of the reason why patients with these conditions can deteriorate during an infection. Also, in patients with neurological disease the BBB may be abnormally sensitive to the effects of systemic inflammation.

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answers: NDQ_010489

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answers: NDQ_010490

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answers: NDQ_010498

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answers: NDQ_010489

answers: Multiple sclerosis and Alzheimer's disease

answers: NDQ_010490

answers: Infection

answers: NDQ_010494

answers: Alzheimer's disease

answers: NDQ_010495

answers: multiple sclerosis

answers: NDQ_010496

answers: multiple sclerosis

answers: NDQ_010497

answers: multiple sclerosis

answers: NDQ_010498

answers: multiple sclerosis

answer: Yes

answer: Yes

answer: No

answer: Yes

answer: No

answer: 1

text: The Drakh also grow parasitic extensions of their being, known as Keepers, on their bodies. The Keepers are partially sentient creatures resembling a large, flesh-covered nervous system with protruding legs that also function as probosci to insert and innervate the nervous systems of other beings. The Keepers function as remote control systems for the Drakh's conscripted servants; they attach a Keeper to a host organism as a means of controlling and monitoring the person through telepathic relay. This control, however, can be neutralized through the consumption of large quantities of certain chemicals, such as alcohol. A Keeper is primarily a passive agent that merely monitors and relays information, but is also capable of direct control and neurological interference such as pain induction, paralysis, or physical violence. In their conscious state, a Keeper is invisible. A Keeper can never be fully removed from the host; the internal segments will break away from the outer shell and regenerate the full body from inside. A Keeper can only be destroyed by killing the Drakh from which it was spawned.

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answers: NDQ_010489

answers: neurological interference

answers: NDQ_010490

answers: neurological interference such as pain induction, paralysis, or physical violence

answers: NDQ_010494

answers: neurological interference

answers: NDQ_010495

answers: paralysis

answers: NDQ_010496

answers: paralysis

answers: NDQ_010497

answers: neurological interference

answers: NDQ_010498

answers: neurological interference

answer: No

answer: No

answer: No

answer: No

answer: No

answer: 1

text: There are documented benefits of mind-body interventions derived from scientific research firstly into their use in contributing to the treatment a range of conditions including headaches, coronary artery disease and chronic pain; secondly in ameliorating disease and the symptoms of  chemotherapy-induced nausea, vomiting, and localized physical pain in patients with cancer; thirdly in increasing the perceived capacity to cope with significant problems and challenges; and fourthly in improving the reported overall quality of life. In addition, there is evidence supporting the brain and central nervous system's influence on the immune system and the capacity for mind-body interventions to enhance immune function outcomes, including defense against and recovery from infection and disease.

text: There are documented benefits of mind-body interventions derived from

text: scientific research

text: firstly into their use in contributing to the treatment a range of conditions including

text: headache

text: s,

text: coronary artery disease

text: and

text: chronic pain

text: ; secondly in ameliorating

text: disease

text: and the

text: symptom

text: s of

text: chemotherapy-induced

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text: C.H. Boehringer Sohn AG & Ko. KG is the parent company of Boehringer Ingelheim GmbH, which was founded in 1885 by Albert Boehringer in Ingelheim am Rhein, Germany. The Boehringer Ingelheim group is one of the world's 20 leading pharmaceutical companies. Still headquartered in Ingelheim, it operates globally with 146 affiliates and more than 47,700 employees. The company's key assets of interest are: respiratory diseases, metabolism, immunology, oncology and diseases of the central nervous system. Boehringer Ingelheim is a full member of the European Federation of Pharmaceutical Industries and Associations EFPIA. The corporate logo of Boehringer Ingelheim depicts a stylized rendition of the central section of the imperial palace of Charlemagne.

text: C.H. Boehringer Sohn AG & Ko. KG is the parent company of Boehringer Ingelheim GmbH, which was founded in 1885 by Albert Boehringer in

text: Ingelheim am Rhein

text: , Germany. The Boehringer Ingelheim group is one of the world's 20 leading pharmaceutical companies. Still headquartered in Ingelheim, it operates globally with 146 affiliates and more than 47,700 employees. The company's key assets of interest are:

text: respiratory diseases

text: metabolism

text: immunology

text: oncology

text: and diseases of the

text: central nervous system

text: . Boehringer Ingelheim is a full member of the European Federation of Pharmaceutical Industries and Associations

entity_name: European Federation of Pharmaceutical Industries and Associations

text: EFPIA

text: . The corporate logo of Boehringer Ingelheim depicts a stylized rendition of the central section of the imperial palace of

text: Charlemagne

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text: GM1 Storage Disease, one of a family of conditions called GM1 gangliosidoses, is a recessive, genetic disorder that is inevitably fatal. It is caused by a deficiency of beta-galactosidase, with resulting abnormal storage of acidic lipid materials in cells of the central and peripheral nervous systems, but particularly in the nerve cells. Because PWDs are all rather closely related to one another and share a limited gene pool, PWDs who were GM1 Storage Disease carriers were able to be genetically identified, and the condition has now been almost entirely eliminated from the breed. All breeding stock should be tested for GM-1 storage disease or GM1 gangliosidoses, which is a fatal nerve disease that typically appears when a puppy is approximately six months of age. The affected puppy will show clinical signs of cerebellar dysfunction including ataxia, tremors, paresis, and seizures. The pet may also exhibit a change in temperament. Lesions of the retina and clouding of the cornea may occur. GM-1 storage disease is a recessive deficiency of betagalactosidase. The condition has been genetically identified and is no longer common.

text: GM1 Storage Disease

text: , one of a family of conditions called

text: GM1 gangliosidoses

text: , is a

text: recessive

text: , genetic disorder that is inevitably fatal. It is caused by a

text: deficiency

text: of

text: beta-galactosidase

text: , with resulting abnormal storage of acidic

text: lipid

text: materials in cells of the

text: central

text: and

text: peripheral nervous system

text: s, but particularly in the

text: nerve cell

text: s. Because PWDs are all rather closely related to one another and share a limited gene pool, PWDs who were GM1 Storage Disease carriers were able to be genetically identified, and the condition has now been almost entirely eliminated from the breed. All breeding stock should be tested for GM-1 storage disease or GM1 gangliosidoses, which is a fatal nerve disease that typically appears when a puppy is approximately six months of age. The affected puppy will show clinical signs of cerebellar dysfunction including ataxia, tremors, paresis, and seizures. The pet may also exhibit a change in temperament. Lesions of the retina and clouding of the cornea may occur. GM-1 storage disease is a recessive deficiency of betagalactosidase. The condition has been genetically identified and is no longer common.

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answers: GM1 Storage Disease

answers: NDQ_010490

answers: no longer common

answers: NDQ_010494

answers: cerebellar dysfunction

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answers: GM1 Storage Disease

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answers: cerebellar dysfunction

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answer: Yes

answer: Yes

answer: No

answer: Yes

answer: Yes

answer: 1

text: Examples of cases where stretch marks are common, also given by the Mayo Clinic, include weight gain (in the form of fat and/or muscle), pregnancy, and adolescent growth spurts, though it is also noted that some medications, as well as other medical conditions and diseases, may increase the likelihood of stretch marks appearing. In the case of medication, the Clinic points to "Corticosteroid creams, lotions and pills and chronic use of oral or systemic steroids" as a common contributing factor; in the case of medical conditions that can contribute to stretch marks, examples given include Ehlers-Danlos syndrome, Cushing's syndrome, Marfan syndrome, and "adrenal gland diseases".

text: Examples of cases where stretch marks are common, also given by the Mayo Clinic, include weight gain (in the form of

text: fat

text: and/or

text: muscle

text: pregnancy

text: , and

text: adolescent

text: growth spurt

text: s, though it is also noted that some medications, as well as other medical conditions and diseases, may increase the likelihood of stretch marks appearing. In the case of medication, the Clinic points to "

text: Corticosteroid

text: creams, lotions and pills and chronic use of oral or systemic

text: steroid

text: s" as a common contributing factor; in the case of medical conditions that can contribute to stretch marks, examples given include

text: Ehlers-Danlos syndrome

text: Cushing's syndrome

text: Marfan syndrome

text: , and "adrenal gland diseases".

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answers: "adrenal gland diseases"

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answers: Cushing's syndrome

answer: No

answer: Yes

answer: No

answer: No

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answer: 1

text: Encephalitis lethargica or von Economo disease is an atypical form of encephalitis. Also known as "sleepy sickness" (distinct from tsetse fly-transmitted sleeping sickness), it was first described in 1917 by the neurologist Constantin von Economoand the pathologist Jean-René Cruchet.The disease attacks the brain, leaving some victims in a statue-like condition, speechless and motionless. Between 1915 and 1926, an epidemic of encephalitis lethargica spread around the world.  Nearly five million people were affected, a third of whom died in the acute stages. Many of those who survived never returned to their pre-existing "aliveness".  "They would be conscious and aware - yet not fully awake; they would sit motionless and speechless all day in their chairs, totally lacking energy, impetus, initiative, motive, appetite, affect or desire; they registered what went on about them without active attention, and with profound indifference.  They neither conveyed nor felt the feeling of life;  they were as insubstantial as ghosts, and as passive as zombies."  (Awakenings, Oliver Sacks, p 14)  No recurrence of the epidemic has since been reported, though isolated cases continue to occur.

text: Encephalitis lethargica or von Economo disease is an atypical form of

text: encephalitis

text: . Also known as "sleepy sickness" (distinct from

text: tsetse fly

text: -transmitted

text: sleeping sickness

text: ), it was first described in 1917 by the neurologist

text: Constantin von Economo

text: and the pathologist

text: Jean-René Cruchet

text: .The disease attacks the brain, leaving some victims in a statue-like condition, speechless and motionless. Between 1915 and 1926, an

text: epidemic

text: of encephalitis lethargica spread around the world.  Nearly five million people were affected, a third of whom died in the acute stages. Many of those who survived never returned to their pre-existing "aliveness".  "They would be conscious and aware - yet not fully awake; they would sit motionless and speechless all day in their chairs, totally lacking energy, impetus, initiative, motive, appetite, affect or desire; they registered what went on about them without active attention, and with profound indifference.  They neither conveyed nor felt the feeling of life;  they were as insubstantial as ghosts, and as passive as zombies."  (Awakenings, Oliver Sacks, p 14)  No recurrence of the epidemic has since been reported, though isolated cases continue to occur.

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answers: The disease attacks the brain

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answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: Insults to neurons in the central nervous system caused by infection, trauma, ischemia, stroke, autoimmune responses, or other neurodegenerative diseases may cause reactive astrocytes.

text: Insults to neurons in the

text: central nervous system

text: caused by infection, trauma, ischemia, stroke, autoimmune responses, or other neurodegenerative diseases may cause reactive astrocytes.

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answers: Neurodegenerative diseases

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answers: neurodegenerative

answers: NDQ_010495

answers: autoimmune responses

answers: NDQ_010496

answers: stroke

answers: NDQ_010497

answers: autoimmune responses

answers: NDQ_010498

answers: neurodegenerative diseases

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: In vertebrates, the nervous system can be split into two parts, the central nervous system (brain and spinal cord), and the peripheral nervous system. In many species — including all vertebrates — the nervous system is the most complex organ system in the body, with most of the complexity residing in the brain. The human brain alone contains around one hundred billion neurons and one hundred trillion synapses; it consists of thousands of distinguishable substructures, connected to each other in synaptic networks whose intricacies have only begun to be unraveled. The majority of the approximately 20–25,000 genes belonging to the human genome are expressed specifically in the brain. Due to the plasticity of the human brain, the structure of its synapses and their resulting functions change throughout life. Thus the challenge of making sense of all this complexity is formidable.

text: In vertebrates, the nervous system can be split into two parts, the

text: central nervous system

text: brain

text: and

text: spinal cord

text: ), and the

text: peripheral nervous system

text: . In many species — including all vertebrates — the nervous system is the most complex organ system in the body, with most of the complexity residing in the brain. The

text: human brain

text: alone contains around one hundred billion neurons and one hundred trillion synapses; it consists of thousands of distinguishable substructures, connected to each other in synaptic networks whose intricacies have only begun to be unraveled. The majority of the approximately 20–25,000 genes belonging to the human genome are expressed specifically in the brain. Due to the

text: plasticity

text: of the human brain, the structure of its synapses and their resulting functions change throughout life. Thus the challenge of making sense of all this complexity is formidable.

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answers: The nervous system is the most complex organ system in the body, with most of the complexity residing in the brain

answers: NDQ_010490

answers: Peripheral nervous system

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answers: Alzheimer's

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answers: central nervous system

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answer: No

answer: No

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text: The parasympathetic nervous system (usually abbreviated PSNS, not PNS, to avoid confusion with the peripheral nervous system [PNS]) is one of the two divisions of the autonomic nervous system, the other being the sympathetic nervous system. The autonomic nervous system is responsible for regulating the body's unconscious actions. The parasympathetic system is responsible for stimulation of "rest-and-digest" or "feed and breed" activities that occur when the body is at rest, especially after eating, including sexual arousal, salivation, lacrimation (tears), urination, digestion and defecation. Its action is described as being complementary to that of the sympathetic nervous system, which is responsible for stimulating activities associated with the fight-or-flight response.

text: The parasympathetic nervous system (usually abbreviated PSNS, not PNS, to avoid confusion with the

text: peripheral nervous system

text: [PNS]) is one of the two divisions of the

text: autonomic nervous system

text: , the other being the

text: sympathetic nervous system

text: . The autonomic nervous system is responsible for regulating the body's unconscious actions. The parasympathetic system is responsible for stimulation of "rest-and-digest" or "feed and breed" activities that occur when the body is at rest, especially after eating, including

text: sexual arousal

text: salivation

text: lacrimation

text: (tears),

text: urination

text: digestion

text: and

text: defecation

text: . Its action is described as being complementary to that of the

text: sympathetic nervous system

text: , which is responsible for stimulating activities associated with the

text: fight-or-flight response

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answers: The parasympathetic nervous system

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answers: Peripheral nervous system

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answers: autonomic nervous system

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answers: peripheral nervous system

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answers: autonomic nervous system

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answers: autonomic

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answers: autonomic

answer: No

answer: No

answer: No

answer: No

answer: No

answer: 0

text: Incontinentia pigmenti (IP, also known as "Bloch–Siemens syndrome," "Bloch–Sulzberger disease," "Bloch–Sulzberger syndrome" "melanoblastosis cutis," and "nevus pigmentosus systematicus") is a genetic disorder that affects the skin, hair, teeth, nails, and central nervous system. It is named due to its microscopic appearance.

text: Incontinentia pigmenti (IP, also known as "Bloch–Siemens syndrome," "Bloch–Sulzberger disease," "Bloch–Sulzberger syndrome" "melanoblastosis cutis," and "nevus pigmentosus systematicus") is a

text: genetic disorder

text: that affects the skin, hair, teeth, nails, and central nervous system. It is named due to its microscopic appearance.

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answers: Incontinentia pigmenti

answers: NDQ_010490

answers: Incontinentia pigmenti

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answers: Incontinentia pigmenti

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answers: Incontinentia pigmenti

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answers: Incontinentia pigmenti

answers: NDQ_010497

answers: nevus pigmentosus systematicus

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answers: Incontinentia pigmenti

answer: Yes

answer: Yes

answer: No

answer: Yes

answer: No

answer: 1

text: Symptoms include severe seborrheic dermatitis of the scalp, severe diarrhea, recurrent local and systemic infection, central nervous system problems, and failure to thrive. Other symptoms also include scaling on the trunk and limbs, red patches of skin on parts of the body that bend, fevers, reduced blood protein levels, thick red skin patches, peeling of the skin, itching, corneal ulcers. wasting of the lymph nodes,  underdeveloped lymphatics, anemia, wasting, and nervous system deficiency. The disease may then spread to the rest of the epidermis with the appearance of crusty, dry, moist or greasy scaling on the scalp. Scaling  could also appear behind the ears, nose or eyebrows, or around the mouth; peeling of the skin may also happen in these areas. If left untreated, the skin infections will cause loss of protein or electrolytes. Leiner’s Disease may also be accompanied by a systemic reaction that is most evident in its gastrointestinal manifestation.

text: Symptoms include severe

text: seborrheic dermatitis

text: of the scalp, severe diarrhea, recurrent local and systemic infection, central nervous system problems, and

text: failure to thrive

text: . Other symptoms also include scaling on the trunk and limbs, red patches of skin on parts of the body that bend, fevers, reduced blood protein levels, thick red skin patches, peeling of the skin, itching, corneal ulcers. wasting of the lymph nodes,  underdeveloped lymphatics, anemia, wasting, and nervous system deficiency. The disease may then spread to the rest of the epidermis with the appearance of crusty, dry, moist or greasy scaling on the scalp. Scaling  could also appear behind the ears, nose or eyebrows, or around the mouth; peeling of the skin may also happen in these areas. If left untreated, the skin infections will cause loss of protein or electrolytes. Leiner’s Disease may also be accompanied by a systemic reaction that is most evident in its gastrointestinal manifestation.

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answers: central nervous system problems

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answers: central nervous system

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answers: central nervous system problems

answers: NDQ_010495

answers: Leiner’s Disease

answers: NDQ_010496

answers: Leiner’s Disease

answers: NDQ_010497

answers: Leiner’s Disease

answers: NDQ_010498

answers: central nervous system

answer: Yes

answer: Yes

answer: Yes

answer: No

answer: Yes

answer: 1

text: Neurodevelopment in the adult nervous system includes mechanisms such as remyelination, generation of new neurons, glia, axons, myelin or synapses. Neuroregeneration differs between the peripheral nervous system (PNS) and the central nervous system (CNS) by the functional mechanisms and especially, the extent and speed.

text: Neurodevelopment in the adult nervous system includes mechanisms such as

text: remyelination

text: , generation of new

text: neuron

text: s,

text: glia

text: axon

text: s,

text: myelin

text: or

text: synapse

text: s. Neuroregeneration differs between the

text: peripheral nervous system

text: (PNS) and the

text: central nervous system

text: (CNS) by the functional mechanisms and especially, the extent and speed.

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text: In spring training and the early part of the 2008 season, Reyes vocalized a plan to focus a little more on baseball, and a little less on the theatrics — such as his dugout dances after home runs — that drew criticism during the late part of 2007 when the Mets were struggling. One of the casualties of this change of focus was the Professor Reyes segment played between innings at Shea where he taught the fans Spanish words and phrases. This was replaced by 'Maine Street USA' with John Maine, and 'Do The Wright Thing' with David Wright, which failed to match the popularity of Reyes' spots. However, with some prodding by his teammates, Reyes was encouraged to continue playing with the same energy as he had previously.

text: In spring training and the early part of the 2008 season, Reyes vocalized a plan to focus a little more on baseball, and a little less on the theatrics — such as his dugout dances after home runs — that drew criticism during the late part of 2007 when the Mets were struggling. One of the casualties of this change of focus was the Professor Reyes segment played between innings at

text: Shea

text: where he taught the fans Spanish words and phrases. This was replaced by 'Maine Street USA' with

text: John Maine

text: , and 'Do The Wright Thing' with

text: David Wright

text: , which failed to match the popularity of Reyes' spots. However, with some prodding by his teammates, Reyes was encouraged to continue playing with the same energy as he had previously.

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text: Both the central nervous system (brain and spinal cord) and the peripheral nervous system may be involved. Emboli to the brain may cause stroke-like episodes, headache and episodes of loss of vision in one eye (known as amaurosis fugax). Emboli to the eye can be seen by ophthalmoscopy and are known as plaques of Hollenhorst. Emboli to the spinal cord may cause paraparesis (decreased power in the legs) or cauda equina syndrome, a group of symptoms due to loss of function of the distal part of the spinal cord - loss of control over the bladder, rectum and skin sensation around the anus. If the blood supply to a single nerve is interrupted by an embolus, the result is loss of function in the muscles supplied by that nerve; this phenomenon is called a mononeuropathy.

text: Both the

text: central nervous system

text: (brain and

text: spinal cord

text: ) and the

text: peripheral nervous system

text: may be involved. Emboli to the brain may cause

text: stroke

text: -like episodes,

text: headache

text: and episodes of loss of vision in one eye (known as

text: amaurosis fugax

text: ). Emboli to the eye can be seen by

text: ophthalmoscopy

text: and are known as

text: plaques of Hollenhorst

text: . Emboli to the spinal cord may cause

text: paraparesis

text: (decreased power in the legs) or

text: cauda equina syndrome

text: , a group of symptoms due to loss of function of the distal part of the spinal cord - loss of control over the

text: bladder

text: rectum

text: and skin sensation around the anus. If the blood supply to a single nerve is interrupted by an embolus, the result is loss of function in the muscles supplied by that nerve; this phenomenon is called a

text: mononeuropathy

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text: There are numerous diseases affecting the central nervous system which can cause cognitive impairment. Many of these are associated with aging. Some common examples include Alzheimer's disease and Multi-infarct dementia. Many diseases may be neurological or psychiatric and may primarily affect the brain. Others may affect many other organs, like HIV, Hashimoto's thyroiditis causing hypothyroidism, or cancer. According to a 2015 report in The American Scholar, an assortment of neglected tropical diseases as well as some recently identified pathogens such as Pseudo-nitzschia have also been found to erode human intelligence.

text: There are numerous diseases affecting the

text: central nervous system

text: which can cause cognitive impairment. Many of these are associated with aging. Some common examples include

text: Alzheimer's disease

text: and

text: Multi-infarct dementia

text: . Many diseases may be

text: neurological

text: or

text: psychiatric

text: and may primarily affect the brain. Others may affect many other organs, like

text: HIV

text: Hashimoto's thyroiditis

text: causing

text: hypothyroidism

text: , or

text: cancer

text: . According to a 2015 report in The American Scholar, an assortment of neglected tropical diseases as well as some recently identified pathogens such as Pseudo-nitzschia have also been found to erode human intelligence.

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answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: Occasionally, a patient improves for a few days, then relapses with aseptic meningitis, or very rarely, meningoencephalitis.Patients with meningitis may have a stiff neck, fever, headache, myalgia, nausea and malaise. In some occasions, meningitis occurs without a prodromal syndrome. Meningoencephalitis is characterized by more profound neurological signs such as confusion, drowsiness, sensory abnormalities and motor signs. Under reported complications include myelitis, Guillain–Barré-type syndrome, cranial nerve palsies, transient or permanent hydrocephalus, sensorineural hearing loss, orchitis, arthritis and parotitis. LCMV infections have also been associated with pancreatitis, pneumonitis, myocarditis and pericarditis. The entire illness usually lasts 1 to 3 weeks, nonetheless,  temporary or permanent neurological damage is possible in all central nervous system infections, especially in cases of meningoencephalitis. Chronic infections have not been reported in humans and deaths rarely occur.

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answers: Meningitis

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answers: central nervous system infections

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answers: Meningoencephalitis

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answers: central nervous system infections

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answers: Meningoencephalitis

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answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: In 1980, after the CDC began cautioning physicians and parents about the association between Reye syndrome and the use of salicylates in children with chickenpox or virus-like illnesses, the incidence of Reye syndrome in the United States began to decline. However, the decline began prior to the FDA's issue of warning labels on aspirin in 1986. In the United States between 1980 and 1997, the number of reported cases of Reye syndrome decreased from 555 cases in 1980 to about 2 cases per year since 1994. During this time period 93% of reported cases for which racial data were available occurred in whites and the median age was six years. In 93% of cases a viral illness had occurred  in the preceding three-week period. For the period 1991-1994, the annual rate of hospitalizations due to Reye syndrome in the US was estimated to be between 0.2 and 1.1 per million population less than 18 years of age.

text: In 1980, after the CDC began cautioning physicians and parents about the association between Reye syndrome and the use of salicylates in children with chickenpox or virus-like illnesses, the incidence of Reye syndrome in the United States began to decline. However, the decline began prior to the

text: FDA

text: 's issue of warning labels on aspirin in 1986. In the United States between 1980 and 1997, the number of reported cases of Reye syndrome decreased from 555 cases in 1980 to about 2 cases per year since 1994. During this time period 93% of reported cases for which racial data were available occurred in whites and the median age was six years. In 93% of cases a viral illness had occurred  in the preceding three-week period. For the period 1991-1994, the annual rate of hospitalizations due to Reye syndrome in the US was estimated to be between 0.2 and 1.1 per million population less than 18 years of age.

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answers: Reye syndrome

answers: NDQ_010497

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answers: NDQ_010498

answers: Reye syndrome

answer: No

answer: No

answer: No

answer: No

answer: No

answer: 1

text: An insect’s sensory, motor and physiological processes are controlled by the central nervous system along with the endocrine system. Being the principal division of the nervous system, it consists of a brain, a ventral nerve cord and a subesophageal ganglion. This is connected to the brain by two nerves, extending around each side of the oesophagus.

text: An insect’s sensory,

text: motor

text: and physiological processes are controlled by the

text: central nervous system

text: along with the

text: endocrine system

text: . Being the principal division of the nervous system, it consists of a

text: brain

text: , a

text: ventral nerve cord

text: and a

text: subesophageal ganglion

text: . This is connected to the brain by two nerves, extending around each side of the

text: oesophagus

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answers: The nervous system

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text: Neuroborreliosis is a disorder of the central nervous system caused by infection with a spirochete of the genus Borrelia. The microbiological progression of the disease is similar to that of neurosyphilis, another spirochetal infection.

text: Neuroborreliosis is a disorder of the

text: central nervous system

text: caused by infection with a

text: spirochete

text: of the genus

text: Borrelia

text: . The microbiological progression of the disease is similar to that of

text: neurosyphilis

text: , another spirochetal infection.

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answers: Neuroborreliosis

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answers: spirochete

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answers: Neuroborreliosis

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answers: Neuroborreliosis

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answers: Neuroborreliosis

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 2

text: The afferent leg of the peripheral nervous system is responsible for conveying sensory information to the brain, primarily from the sense organs like the skin. In the muscles, the muscle spindles convey information about the degree of muscle length and stretch to the central nervous system to assist in maintaining posture and joint position. The sense of where our bodies are in space is called proprioception, the perception of body awareness. More easily demonstrated than explained, proprioception is the "unconscious" awareness of where the various regions of the body are located at any one time. This can be demonstrated by anyone closing their eyes and waving their hand around. Assuming proper proprioceptive function, at no time will the person lose awareness of where the hand actually is, even though it is not being detected by any of the other senses.

text: The

text: afferent

text: leg of the peripheral nervous system is responsible for conveying sensory information to the brain, primarily from the sense organs like the skin. In the muscles, the

text: muscle spindle

text: s convey information about the degree of muscle length and stretch to the central nervous system to assist in maintaining posture and joint position. The

text: sense

text: of where our bodies are in space is called

text: proprioception

text: , the perception of body awareness. More easily demonstrated than explained, proprioception is the "unconscious" awareness of where the various regions of the body are located at any one time. This can be demonstrated by anyone closing their eyes and waving their hand around. Assuming proper proprioceptive function, at no time will the person lose awareness of where the hand actually is, even though it is not being detected by any of the other senses.

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answers: The afferent leg of the peripheral nervous system

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answers: Alzheimer's

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answers: peripheral nervous system

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answer: No

answer: No

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text: Known as viremia, the presence of a virus in the bloodstream enables it to be widely distributed throughout the body. Poliovirus can survive and multiply within the blood and lymphatics for long periods of time, sometimes as long as 17 weeks. In a small percentage of cases, it can spread and replicate in other sites, such as brown fat, the reticuloendothelial tissues, and muscle. This sustained replication causes a major viremia, and leads to the development of minor influenza-like symptoms. Rarely, this may progress and the virus may invade the central nervous system, provoking a local inflammatory response. In most cases, this causes a self-limiting inflammation of the meninges, the layers of tissue surrounding the brain, which is known as nonparalytic aseptic meningitis. Penetration of the CNS provides no known benefit to the virus, and is quite possibly an incidental deviation of a normal gastrointestinal infection. The mechanisms by which poliovirus spreads to the CNS are poorly understood, but it appears to be primarily a chance event—largely independent of the age, gender, or socioeconomic position of the individual.

text: Known as

text: viremia

text: , the presence of a virus in the bloodstream enables it to be widely distributed throughout the body. Poliovirus can survive and multiply within the blood and lymphatics for long periods of time, sometimes as long as 17 weeks. In a small percentage of cases, it can spread and replicate in other sites, such as

text: brown fat

text: , the

text: reticuloendothelial

text: tissues, and muscle. This sustained replication causes a major viremia, and leads to the development of minor influenza-like symptoms. Rarely, this may progress and the virus may invade the central nervous system, provoking a local

text: inflammatory response

text: . In most cases, this causes a self-limiting inflammation of the

text: meninges

text: , the layers of tissue surrounding the

text: brain

text: , which is known as nonparalytic aseptic meningitis. Penetration of the CNS provides no known benefit to the virus, and is quite possibly an incidental deviation of a normal gastrointestinal infection. The mechanisms by which poliovirus spreads to the CNS are poorly understood, but it appears to be primarily a chance event—largely independent of the age, gender, or

text: socioeconomic

text: position of the individual.

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text: Mitochondrial DNA can play a role in the onset of disease in a variety of ways. Point mutations in or alternative gene arrangements of mtDNA have been linked to several diseases that affect the heart, central nervous system, endocrine system, gastrointestinal tract, eye, and kidney.     Loss of the amount of mtDNA present in the mitochondria can lead to a whole subset of diseases known as mitochondrial depletion syndromes (MDDs) which affect the liver, central and peripheral nervous systems, smooth muscle and hearing in humans.   There have been mixed, and sometimes conflicting, results in studies that attempt to link mtDNA copy number to the risk of developing certain cancers.  Studies have been conducted that show an association between both increased and decreased mtDNA levels and the increased risk of developing breast cancer.  A positive association between increased mtDNA levels and an increased risk for developing kidney tumors has been observed but there does not appear to be a link between mtDNA levels and the development of stomach cancer.

text: Mitochondrial DNA can play a role in the onset of disease in a variety of ways.

text: Point mutations

text: in or alternative gene arrangements of mtDNA have been linked to several diseases that affect the heart,

text: central nervous system

text: , endocrine system, gastrointestinal tract, eye, and kidney.     Loss of the amount of mtDNA present in the mitochondria can lead to a whole subset of diseases known as mitochondrial depletion syndromes (MDDs) which affect the liver, central and

text: peripheral nervous

text: systems, smooth muscle and hearing in humans.   There have been mixed, and sometimes conflicting, results in studies that attempt to link mtDNA copy number to the risk of developing certain cancers.  Studies have been conducted that show an association between both increased and decreased mtDNA levels and the increased risk of developing

text: breast cancer

text: .  A positive association between increased mtDNA levels and an increased risk for developing kidney tumors has been observed but there does not appear to be a link between mtDNA levels and the development of

text: stomach cancer

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text: Despite their naming, glia function more as partners to neurons than as "glue".They are also crucial in the development of the nervous system and in processes such as synaptic plasticity and synaptogenesis. Glia have a role in the regulation of repair of neurons after injury. In the central nervous system (CNS), glia suppress repair. Glial cells known as astrocytes enlarge and proliferate to form a scar and produce inhibitory molecules that inhibit regrowth of a damaged or severed axon. In the peripheral nervous system (PNS), glial cells known as Schwann cells promote repair. After axonal injury, Schwann cells regress to an earlier developmental state to encourage regrowth of the axon. This difference between the CNS and the PNS, raises hopes for the regeneration of nervous tissue in the CNS. For example, a spinal cord may be able to be repaired following injury or severance. Schwann cells are also known as neuri-lemmocytes.  These cells envelop nerve fibers of the PNS by winding repeatedly around a nerve fiber with the nucleus inside of it.  This process creates a myelin sheath, which not only aids in conductivity but also assists in the regeneration of damaged fibers.

text: Despite their naming, glia function more as partners to neurons than as "glue".They are also crucial in the development of the nervous system and in processes such as

text: synaptic plasticity

text: and

text: synaptogenesis

text: . Glia have a role in the regulation of repair of neurons after injury. In the

text: central nervous system

text: (CNS), glia suppress repair. Glial cells known as

text: astrocytes

text: enlarge and proliferate to form a scar and produce inhibitory molecules that inhibit regrowth of a damaged or severed axon. In the

text: peripheral nervous system

text: (PNS), glial cells known as

text: Schwann cell

text: s promote repair. After axonal injury, Schwann cells regress to an earlier developmental state to encourage regrowth of the axon. This difference between the CNS and the PNS, raises hopes for the regeneration of nervous tissue in the CNS. For example, a spinal cord may be able to be repaired following injury or severance. Schwann cells are also known as neuri-lemmocytes.  These cells envelop nerve fibers of the PNS by winding repeatedly around a nerve fiber with the nucleus inside of it.  This process creates a myelin sheath, which not only aids in conductivity but also assists in the regeneration of damaged fibers.

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text: Until recently, the medical literature did not indicate a connection among many genetic disorders, both genetic syndromes and genetic diseases, that are now being found to be related.  As a result of new genetic research, some of these are, in fact, highly related in their root cause (genotype) despite the widely varying set of medical characteristics (phenotype) that are clinically visible in the disorders.  Dandy–Walker syndrome is one such  disease, part of an emerging class of diseases called ciliopathies.  The underlying cause may be a dysfunctional molecular mechanism in the primary cilia structures of the cell, organelles which are present in many cellular types throughout the human body.  The cilia defects adversely affect "numerous critical developmental signaling pathways" essential to cellular development and thus offer a plausible hypothesis for the often multi-symptom nature of a large set of syndromes and diseases.  Known ciliopathies include primary ciliary dyskinesia, Bardet-Biedl syndrome, polycystic kidney and liver disease, nephronophthisis, Alstrom syndrome, Meckel-Gruber syndrome and some forms of retinal degeneration.

text: Until recently, the medical literature did not indicate a connection among many

text: genetic disorder

text: s, both

text: genetic syndromes

text: and

text: genetic diseases

text: , that are now being found to be related.  As a result of new genetic research, some of these are, in fact, highly related in their root cause (

text: genotype

text: ) despite the widely varying set of medical characteristics (

text: phenotype

text: ) that are clinically visible in the

text: disorders

text: .  Dandy–Walker syndrome is one such  disease, part of an

text: emerging

text: class of diseases called

text: ciliopathies

text: .  The underlying cause may be a dysfunctional molecular mechanism in the primary

text: cilia

text: structures of the

text: cell

text: organelle

text: s which are present in many cellular types throughout the

text: human

text: body.  The cilia defects adversely affect "numerous critical developmental signaling pathways" essential to cellular development and thus offer a plausible hypothesis for the often

text: multi-symptom

text: nature of a large set of syndromes and diseases.  Known ciliopathies include

text: primary ciliary dyskinesia

text: Bardet-Biedl syndrome

text: polycystic kidney

text: and

text: liver disease

text: nephronophthisis

text: Alstrom syndrome

text: Meckel-Gruber syndrome

text: and some forms of

text: retinal degeneration

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answer: No

answer: No

answer: No

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answer: 1

text: The vertebrate nervous system relies on the myelin sheath for insulation and as a method of decreasing membrane capacitance in the axon. The action potential jumps from node to node, in a process called saltatory conduction, which can increase conduction velocity up to ten times, without an increase in axonal diameter. In this sense, Schwann cells are the peripheral nervous system's analogues of the central nervous system's oligodendrocytes. However, unlike oligodendrocytes, each myelinating Schwann cell provides insulation to only one axon (see image). This arrangement permits saltatory conduction of action potentials with repropagation at the nodes of Ranvier. In this way, myelination greatly increases speed of conduction and saves energy.

text: The

text: vertebrate

text: nervous system

text: relies on the myelin sheath for

text: insulation

text: and as a method of decreasing membrane

text: capacitance

text: in the axon. The

text: action potential

text: jumps from node to node, in a process called

text: saltatory conduction

text: , which can increase

text: conduction

text: velocity up to ten times, without an increase in axonal diameter. In this sense, Schwann cells are the peripheral nervous system's analogues of the

text: central nervous system

text: 's

text: oligodendrocyte

text: s. However, unlike oligodendrocytes, each myelinating Schwann cell provides insulation to only one axon (see image). This arrangement permits saltatory conduction of action potentials with

text: repropagation

text: at the

text: nodes of Ranvier

text: . In this way, myelination greatly increases speed of conduction and saves energy.

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answers: Peripheral

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answer: No

answer: No

answer: No

answer: No

answer: 1

text: In general, a neurological examination is focused on finding out whether there are lesions in the central and peripheral nervous systems or there is another diffuse process that is troubling the patient. Once the patient has been thoroughly tested, it is then the role of the physician to determine whether these findings combine to form a recognizable medical syndrome or neurological disorder such as Parkinson's disease or motor neurone disease. Finally, it is the role of the physician to find the cause for why such a problem has occurred, for example finding whether the problem is due to inflammation or is congenital.

text: In general, a neurological examination is focused on finding out whether there are

text: lesion

text: s in the

text: central

text: and

text: peripheral nervous system

text: s or there is another diffuse process that is troubling the patient. Once the patient has been thoroughly tested, it is then the role of the

text: physician

text: to determine whether these findings combine to form a recognizable medical syndrome or

text: neurological disorder

text: such as

text: Parkinson's disease

text: or

text: motor neurone disease

text: . Finally, it is the role of the physician to find the cause for why such a problem has occurred, for example finding whether the problem is due to inflammation or is congenital.

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text: Meningitis due to anaerobic bacteria is infrequent and may follow respiratory tract infection or complicate a cerebrospinal ﬂuid shunt. Nerological shunt infections are often caused by skin bacteria such as Propionibacterium acnes, or in instances of ventriculoperitoneal shunts that perforate the gut, by anaerobes of enteric origin (i.e. Bacteroides fragilis).Clostridium perfringens can cause of brain abscesses and meningitis following intracranial surgery or head trauma.

text: Meningitis

text: due to anaerobic bacteria is infrequent and may follow respiratory tract infection or complicate a cerebrospinal ﬂuid shunt. Nerological shunt infections are often caused by skin bacteria such as Propionibacterium acnes, or in instances of ventriculoperitoneal shunts that perforate the gut, by anaerobes of enteric origin (i.e. Bacteroides fragilis).Clostridium perfringens can cause of brain abscesses and meningitis following intracranial surgery or head trauma.

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answers: Meningitis

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answers: cerebrospinal fluid

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answers: Meningitis

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answers: cerebrospinal fluid shunt

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answer: 1

text: At the most basic level, the function of the nervous system is to send signals from one cell to others, or from one part of the body to others. There are multiple ways that a cell can send signals to other cells. One is by releasing chemicals called hormones into the internal circulation, so that they can diffuse to distant sites. In contrast to this "broadcast" mode of signaling, the nervous system provides "point-to-point" signals—neurons project their axons to specific target areas and make synaptic connections with specific target cells. Thus, neural signaling is capable of a much higher level of specificity than hormonal signaling. It is also much faster: the fastest nerve signals travel at speeds that exceed 100 meters per second.

text: At the most basic level, the function of the nervous system is to send signals from one cell to others, or from one part of the body to others. There are multiple ways that a cell can send signals to other cells. One is by releasing chemicals called

text: hormone

text: s into the internal circulation, so that they can diffuse to distant sites. In contrast to this "broadcast" mode of signaling, the nervous system provides "point-to-point" signals—neurons project their axons to specific target areas and make synaptic connections with specific target cells. Thus, neural signaling is capable of a much higher level of specificity than hormonal signaling. It is also much faster: the fastest nerve signals travel at speeds that exceed 100 meters per second.

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answers: It is also much faster

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answers: It is also much faster

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answer: No

answer: No

answer: No

answer: No

answer: No

answer: 1

text: Epilepsies with onset in childhood are a complex group of diseases with a variety of causes and characteristics. Some people have no obvious underlying neurological problems or metabolic disturbances. They may be associated with variable degrees of intellectual disability, elements of autism, other mental disorders, and motor difficulties. Others have underlying inherited metabolic diseases, chromosomal abnormalities, specific eye, skin and nervous system features, or malformations of cortical development. Some of these epilepsies can be categorized into the traditional epilepsy syndromes. Furthermore, a variety of clinical syndromes exist of which the main feature is not epilepsy but which are associated with a higher risk of epilepsy. For instance between 1 and 10% of those with Down syndrome and 90% of those with Angelman syndrome have epilepsy.

text: Epilepsies with onset in childhood are a complex group of diseases with a variety of causes and characteristics. Some people have no obvious underlying neurological problems or

text: metabolic

text: disturbances. They may be associated with variable degrees of intellectual disability, elements of

text: autism

text: , other

text: mental disorder

text: s, and motor difficulties. Others have underlying inherited metabolic diseases,

text: chromosomal

text: abnormalities,

text: specific eye, skin and nervous system features

text: , or malformations of cortical development. Some of these epilepsies can be categorized into the traditional epilepsy syndromes. Furthermore, a variety of clinical syndromes exist of which the main feature is not epilepsy but which are associated with a higher risk of epilepsy. For instance between 1 and 10% of those with

text: Down syndrome

text: and 90% of those with

text: Angelman syndrome

text: have epilepsy.

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answers: Down syndrome

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: Neurocytoma (or neuroepithelioma) is a type of nervous system tumor which is primarily derived from nervous tissue. This is in contrast to the gliomas (such as oligodendroglioma), which are derived from glial cells, which are not derived from nervous tissue.

text: Neurocytoma (or neuroepithelioma) is a type of nervous system tumor which is primarily derived from

text: nervous tissue

text: . This is in contrast to the

text: glioma

text: s (such as

text: oligodendroglioma

text: ), which are derived from

text: glial cell

text: s, which are not derived from nervous tissue.

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text: The Mexican Reyes should not be confused with the Cuban bassist Jorge Reyes, the Cuban author Jorge Ivan Reyes, the Brooklyn television writer Jorge A. Reyes,  or the Oregon baseball player Jorge Reyes.

text: The Mexican Reyes should not be confused with the Cuban bassist Jorge Reyes, the Cuban author

text: Jorge Ivan Reyes

text: , the Brooklyn television writer Jorge A. Reyes,  or the Oregon baseball player Jorge Reyes.

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text: Central nervous system imaging: Central nervous system abnormalities, such as cortical or subependymal tubers and astrocytomas, are common in patients with TSC, including those with TSC-LAM, but are not found in women with S-LAM. Moss and associates reported that women with S-LAM and TSC-LAM may have an increased incidence of meningioma, but the significance of that finding has been challenged.

text: Central nervous system imaging: Central nervous system abnormalities, such as

text: cortical

text: or subependymal tubers and

text: astrocytomas

text: , are common in patients with TSC, including those with TSC-LAM, but are not found in women with S-LAM. Moss and associates reported that women with S-LAM and TSC-LAM may have an increased incidence of meningioma, but the significance of that finding has been challenged.

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answers: Central nervous system abnormalities

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answer: Yes

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answer: Yes

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text: Reye syndrome progresses through five stages:

text: Reye syndrome progresses through five stages:

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answer: 1

text: Though it does not produce severe immunodeficiency, visna shares many characteristics with human immunodeficiency virus, including the establishment of persistent infection with chronic active lymphoproliferation; however, visna virus does not infect T-lymphocytes. The relationship of visna and HIV as lentiviruses was first published in 1985 by visna researcher Janice E. Clements and colleagues in the HIV field. It has been postulated that the effects of maedi-visna infection in sheep are the "equivalent" of central nervous system disease and wasting syndrome found in human AIDS patients. Despite limited sequence homology with HIV, the genomic organization of visna is very similar, allowing visna infection to be used as an in vivo and in vitro model system for HIV infection.

text: Though it does not produce severe

text: immunodeficiency

text: , visna shares many characteristics with

text: human immunodeficiency virus

text: , including the establishment of persistent infection with chronic active lymphoproliferation; however, visna virus does not infect

text: T-lymphocyte

text: s. The relationship of visna and HIV as

text: lentivirus

text: es was first published in 1985 by visna researcher

text: Janice E. Clements

text: and colleagues in the HIV field. It has been postulated that the effects of maedi-visna infection in sheep are the "equivalent" of central nervous system disease and wasting syndrome found in human AIDS patients. Despite limited

text: sequence homology

text: with HIV, the genomic organization of visna is very similar, allowing visna infection to be used as an

text: in vivo

text: and

text: in vitro

text: model system

text: for HIV infection.

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answers: central nervous system disease and wasting syndrome found in human AIDS patients

answers: NDQ_010490

answers: maedi-visna infection

answers: NDQ_010494

answers: AIDS

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answers: immunodeficiency

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answers: wasting syndrome

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answers: wasting syndrome

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answers: wasting syndrome

answer: Yes

answer: Yes

answer: No

answer: Yes

answer: No

answer: 1

text: The hypereosinophilic syndrome (HES) is a disease characterized by a persistently elevated eosinophil count (≥ 1500 eosinophils/mm³) in the blood for at least six months without any recognizable cause, with involvement of either the heart, nervous system, or bone marrow.

text: The hypereosinophilic syndrome (HES) is a disease characterized by a persistently

text: elevated eosinophil count

text: (≥ 1500 eosinophils/mm³) in the blood for at least six months without any recognizable cause, with involvement of either the

text: heart

text: nervous system

text: , or

text: bone marrow

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text: He was perhaps the first (1882) to recognize angioedema which is often referred to as "Quincke's edema". "Quincke's pulse", with redness and pallor seen under the fingernails, is one of the signs of aortic insufficiency. "Quincke's puncture" is a somewhat outdated eponym for lumbar puncture, used for the examination of the cerebrospinal fluid in numerous diseases such as meningitis and multiple sclerosis. In 1893 he described what is now known as idiopathic intracranial hypertension, which he labeled "serous meningitis".

text: He was perhaps the first (1882) to recognize

text: angioedema

text: which is often referred to as "Quincke's edema". "Quincke's pulse", with redness and pallor seen under the fingernails, is one of the signs of

text: aortic insufficiency

text: . "Quincke's puncture" is a somewhat outdated eponym for

text: lumbar puncture

text: , used for the examination of the

text: cerebrospinal fluid

text: in numerous diseases such as

text: meningitis

text: and

text: multiple sclerosis

text: . In 1893 he described what is now known as

text: idiopathic intracranial hypertension

text: , which he labeled "serous meningitis".

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answer: Yes

answer: No

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answer: 1

text: Itraconazole has a broader spectrum of activity than fluconazole (but not as broad as voriconazole or posaconazole). In particular, it is active against Aspergillus, which fluconazole is not. It is also licensed for use in blastomycosis, sporotrichosis, histoplasmosis, and onychomycosis. Itraconazole is over 99% protein-bound and has virtually no penetration into cerebrospinal fluid. Therefore, it should never be used to treat meningitis or other central nervous system infections. According to the Johns Hopkins Abx Guide, it has "negligible CSF penetration, however treatment has been successful for cryptococcal and coccidioidal meningitis".

text: Itraconazole has a broader spectrum of activity than

text: fluconazole

text: (but not as broad as

text: voriconazole

text: or

text: posaconazole

text: ). In particular, it is active against

text: Aspergillus

text: , which fluconazole is not. It is also licensed for use in

text: blastomycosis

text: sporotrichosis

text: histoplasmosis

text: , and

text: onychomycosis

text: . Itraconazole is over 99% protein-bound and has virtually no penetration into

text: cerebrospinal fluid

text: . Therefore, it should never be used to treat

text: meningitis

text: or other

text: central nervous system

text: infections. According to the Johns Hopkins Abx Guide, it has "negligible CSF penetration, however treatment has been successful for cryptococcal and coccidioidal meningitis".

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answers: Meningitis or other central nervous system infections

answers: NDQ_010490

answers: meningitis

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answers: meningitis

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answers: meningitis

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answers: meningitis

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answers: meningitis

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answer: Yes

answer: Yes

answer: No

answer: Yes

answer: No

answer: 1

text: Widal was the author of a remarkable series of essays on infectious diseases, erysipelas, diseases of the heart, liver, nervous system, etc., besides being a prolific contributor to various medical journals and encyclopedias. His name is associated with the Widal test, a diagnostic test for typhoid fever, and with hematologist Georges Hayem (1841-1933), he described acquired haemolytic anaemia, a disease that was historically referred to as "Hayem-Widal syndrome".

text: Widal was the author of a remarkable series of essays on infectious

text: disease

text: s,

text: erysipelas

text: , diseases of the

text: heart

text: liver

text: nervous system

text: , etc., besides being a prolific contributor to various

text: medical journal

text: s and

text: encyclopedia

text: s. His name is associated with the

text: Widal test

text: , a diagnostic test for

text: typhoid fever

text: , and with

text: hematologist

text: Georges Hayem

text: (1841-1933), he described acquired

text: haemolytic anaemia

text: , a disease that was historically referred to as "Hayem-Widal syndrome".

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answers: Widal

answers: NDQ_010490

answers: nervous system

answers: NDQ_010494

answers: typhoid

answers: NDQ_010495

answers: acquired haemolytic anaemia

answers: NDQ_010496

answers: typhoid fever

answers: NDQ_010497

answers: haemolytic anaemia

answers: NDQ_010498

answers: erysipelas

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: The middle element of the meninges is the arachnoid mater, so named because of its spider web-like appearance. It cushions the central nervous system. This thin, transparent membrane is composed of fibrous tissue and, like the pia mater, is covered by flat cells also thought to be impermeable to fluid.

text: The middle element of the meninges is the

text: arachnoid mater

text: , so named because of its

text: spider web

text: -like appearance. It cushions the

text: central nervous system

text: . This thin, transparent membrane is composed of fibrous tissue and, like the pia mater, is covered by flat cells also thought to be impermeable to fluid.

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answers: It cushions the central nervous system

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text: The witch's mark also factors into the theory proposed by M.M. Drymon that Lyme disease is a diagnosis for both witches and witch affliction, finding that many of the afflicted and accused in Salem and elsewhere lived in areas that were tick-risky, had a variety of red marks and rashes that looked like bite marks on their skin, and suffered from neurological and arthritic symptoms. The appearance of the witches' mark in Europe is only noted after Colombian contact with the New World in 1492 and may be the result of the transfer of a virulent form of borrelia infection from America into Europe, especially in areas under the control of the Spanish Empire, including parts of the Rhine River Valley that are now in Germany. This topic is the subject of a recent work in the study of witchcraft. This theory is an expansion of the idea first proposed by Laurie Winn Carlson that the bewitched in Salem suffered from encephalitis. Neurological Lyme disease is probably the only form of mild or acute encephalitis that is accompanied by a round red mark or bull's eye rash on the skin, which can appear after tick attachment.

text: The witch's mark also factors into the theory proposed by M.M. Drymon that

text: Lyme disease

text: is a diagnosis for both witches and witch affliction, finding that many of the afflicted and accused in Salem and elsewhere lived in areas that were tick-risky, had a variety of red marks and rashes that looked like bite marks on their skin, and suffered from neurological and arthritic symptoms. The appearance of the witches' mark in Europe is only noted after Colombian contact with the New World in 1492 and may be the result of the transfer of a virulent form of

text: borrelia infection

text: from America into Europe, especially in areas under the control of the

text: Spanish Empire

text: , including parts of the

text: Rhine

text: River Valley that are now in Germany. This topic is the subject of a recent work in the study of witchcraft. This theory is an expansion of the idea first proposed by Laurie Winn Carlson that the bewitched in Salem suffered from

text: encephalitis

text: . Neurological Lyme disease is probably the only form of mild or acute encephalitis that is accompanied by a round red mark or bull's eye rash on the skin, which can appear after tick attachment.

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answer: Yes

answer: Yes

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text: 1970s: Legionnaires' disease, Ebola, and Reye syndrome.

text: Legionnaires' disease

text: Ebola

text: , and

text: Reye syndrome

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answers: Reye syndrome

answers: NDQ_010490

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answers: Reye syndrome

answer: No

answer: No

answer: No

answer: No

answer: No

answer: 1

text: WEE was discovered in 1930 when a number of horses in the San Joaquin Valley of California, USA died of a mysterious encephalitis. Karl Friedrich Meyer investigated but was not able to isolate the pathogen from necropsies of horses that had been dead for some time and needed samples from an animal in the earlier stages of disease. When the team heard of a horse that appeared to have encephalitis, its owner threatened to shoot the scientists. However Meyer was able to convince the farmer's wife that the horse was dying anyway, and to secretly signal him when the farmer was asleep in exchange for $20 (as this was during the Great Depression, this was a substantial amount of money). Meyer and his colleagues hid in the bushes until the signal, euthanized the horse and stole its head. They successfully isolated WEEV from the brain tissue.

text: WEE was discovered in 1930 when a number of

text: horse

text: s in the

text: San Joaquin Valley

text: of California, USA died of a mysterious encephalitis.

text: Karl Friedrich Meyer

text: investigated but was not able to isolate the pathogen from

text: necropsies

text: of horses that had been dead for some time and needed samples from an animal in the earlier stages of disease. When the team heard of a horse that appeared to have encephalitis, its owner threatened to shoot the scientists. However Meyer was able to convince the farmer's wife that the horse was dying anyway, and to secretly signal him when the farmer was asleep in exchange for $20 (as this was during the

text: Great Depression

text: , this was a substantial amount of money). Meyer and his colleagues hid in the bushes until the signal,

text: euthanized

text: the horse and stole its head. They successfully isolated WEEV from the brain tissue.

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answers: encephalitis

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answers: NDQ_010498

answers: encephalitis

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: This differentiates the central nervous system from the peripheral nervous system, which consists of neurons, axons and Schwann cells. Oligodendrocytes and Schwann cells have similar functions in the central and peripheral nervous system respectively. Both act to add myelin sheaths to the axons, which acts as a form of insulation allowing for better and faster proliferation of electrical signals along the nerves. Axons in the central nervous system are often very short (barely a few millimeters) and do not need the same degree of isolation as peripheral nerves do. Some peripheral nerves can be over 1m in length, such as the nerves to the big toe. To ensure signals move at sufficient speed, myelination is needed.

text: This differentiates the central nervous system from the peripheral nervous system, which consists of neurons, axons and

text: Schwann cells

text: . Oligodendrocytes and Schwann cells have similar functions in the central and peripheral nervous system respectively. Both act to add

text: myelin

text: sheaths to the axons, which acts as a form of insulation allowing for better and faster proliferation of electrical signals along the nerves. Axons in the central nervous system are often very short (barely a few millimeters) and do not need the same degree of isolation as peripheral nerves do. Some peripheral nerves can be over 1m in length, such as the nerves to the big toe. To ensure signals move at sufficient speed, myelination is needed.

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text: The symptoms the fetus develops from CVV infection are largely age dependent. At less than 28 days of gestation, the embryo usually dies and is reabsorbed by the mother. Between 28 and 45 days of gestation, infection leads to malformations in the developing fetus and occasionally leads to abortions. Early in this window, between 28 and 36 days, the virus leads to both central nervous system and musculoskeletal defects, while after 36 days of gestation infection only leads to musculoskeletal deformities. Death of the fetus usually occurs between 27 and 35 days gestation, when the central nervous system tissues are most susceptible. After 45–50 days of gestation CVV infection is not expected to cause harmful effects. After 76 days the fetus has a functioning immune system and antibodies to the virus are produced.

text: The symptoms the fetus develops from CVV infection are largely age dependent. At less than 28 days of gestation, the embryo usually dies and is reabsorbed by the mother. Between 28 and 45 days of gestation, infection leads to malformations in the developing fetus and occasionally leads to abortions. Early in this window, between 28 and 36 days, the virus leads to both central nervous system and musculoskeletal defects, while after 36 days of gestation infection only leads to musculoskeletal deformities. Death of the fetus usually occurs between 27 and 35 days gestation, when the central nervous system tissues are most susceptible. After 45–50 days of gestation CVV infection is not expected to cause harmful effects. After 76 days the fetus has a functioning immune system and antibodies to the virus are produced.

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text: Parkinson's disease (PD) is a long-term degenerative disorder of the central nervous system that mainly affects the motor system. The symptoms generally come on slowly over time. Early in the disease, the most obvious are shaking, rigidity, slowness of movement, and difficulty with walking. Thinking and behavioral problems may also occur. Dementia becomes common in the advanced stages of the disease. Depression and anxiety are also common occurring in more than a third of people with PD. Other symptoms include sensory, sleep, and emotional problems. The main motor symptoms are collectively called "parkinsonism", or a "parkinsonian syndrome".

text: Parkinson's disease (PD) is a long-term degenerative disorder of the

text: central nervous system

text: that mainly affects the

text: motor system

text: . The symptoms generally come on slowly over time. Early in the disease, the most obvious are

text: shaking

text: rigidity

text: slowness of movement

text: , and

text: difficulty with walking

text: Thinking

text: and behavioral problems may also occur.

text: Dementia

text: becomes common in the advanced stages of the disease.

text: Depression

text: and anxiety are also common occurring in more than a third of people with PD. Other symptoms include sensory,

text: sleep

text: , and

text: emotion

text: al problems. The main motor symptoms are collectively called "

text: parkinsonism

text: ", or a "parkinsonian syndrome".

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text: Until the mid 1990s, infectious complications of the Central Nervous System (CNS) caused by VZV reactivation were regarded as rare.  The presence of rash, as well as speciﬁc neurological symptoms, were required to diagnose a CNS infection caused by VZV.  Since 2000, PCR testing has become more widely used, and the number of diagnosed cases of CNS infection has increased.

text: Until the mid 1990s, infectious complications of the

text: Central Nervous System

text: (CNS) caused by VZV reactivation were regarded as rare.  The presence of rash, as well as speciﬁc neurological symptoms, were required to diagnose a CNS infection caused by VZV.  Since 2000, PCR testing has become more widely used, and the number of diagnosed cases of CNS infection has increased.

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text: While many approaches to defining butoh—as with any performative tradition—will focus on formalism or semantic layers, another approach is to focus on physical technique. While butoh does not have a codified classical technique rigidly adhered to within an authoritative controlled lineage, Hijikata Tatsumi did have a substantive methodical body of movement techniques called Butoh Fu. Butoh Fu can be described as a series of cues largely based on incorporating visualizations that directly affect the nervous system, producing qualities of movement that are then used to construct the form and expression of the dance. This mode of engaging the nervous system directly has much in common with other mimetic techniques to be found in the history of dance, such as Lecoq's range of nervous system qualities, Decroux's rhythm and density within movement, and Zeami Motokiyo's qualitative descriptions for character types.

text: While many approaches to defining butoh—as with any performative tradition—will focus on formalism or semantic layers, another approach is to focus on physical technique. While butoh does not have a codified classical technique rigidly adhered to within an authoritative controlled lineage, Hijikata Tatsumi did have a substantive methodical body of movement techniques called Butoh Fu. Butoh Fu can be described as a series of cues largely based on incorporating visualizations that directly affect the nervous system, producing qualities of movement that are then used to construct the form and expression of the dance. This mode of engaging the nervous system directly has much in common with other mimetic techniques to be found in the history of dance, such as

entity_name: L'École Internationale de Théâtre Jacques Lecoq

text: Lecoq

text: 's range of nervous system qualities,

text: Decroux

text: 's rhythm and density within movement, and Zeami Motokiyo's qualitative descriptions for character types.

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text: Since physical abnormalities can affect all body parts along the midline of the body, many different body systems are at risk of being damaged. Those systems commonly affected are the nervous, digestive, cardiovascular, respiratory and reproductive. Typically, those diagnosed with either type of Opitz G/BBB syndrome will also have ocular hypertelorism, or eyes that are excessively spaced apart. Patients will commonly experience physical defects in the larynx, trachea and esophagus and will often cause breathing and swallowing difficulties. Further facial anomalies may occur, such as a prominent forehead, widow’s peak, broad nasal bridge. Those with the disease may also experience hypospadias (urinary opening in males is not at the head of the penis) and developmental delay, as well.

text: Since physical abnormalities can affect all body parts along the midline of the body, many different body systems are at risk of being damaged. Those systems commonly affected are the

text: nervous

text: digestive

text: cardiovascular

text: respiratory

text: and

text: reproductive

text: . Typically, those diagnosed with either type of Opitz G/BBB syndrome will also have

text: ocular hypertelorism

text: , or eyes that are excessively spaced apart. Patients will commonly experience physical defects in the

text: larynx

text: trachea

text: and

text: esophagus

text: and will often cause breathing and swallowing difficulties. Further facial

text: anomalies

text: may occur, such as a prominent forehead,

text: widow’s peak

text: , broad nasal bridge. Those with the disease may also experience

text: hypospadias

text: (urinary opening in males is not at the head of the penis) and

text: developmental delay

text: , as well.

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text: Neuropathology is a specialty within the study of pathology focused on the disease of the brain, spinal cord, and neural tissue. This includes the central nervous system and the peripheral nervous system. Tissue analysis comes from either surgical biopsies or post mortem autopsies. Common tissue samples include muscle fibers and nervous tissue. Common applications of neuropathology include studying samples of tissue in patients who have Parkinson's disease, Alzheimer's disease, dementia, Huntington's disease, amyotrophic lateral sclerosis, mitochondria disease, and any disorder that has neural deterioration in the brain or spinal cord.

text: Neuropathology

text: is a specialty within the study of

text: pathology

text: focused on the disease of the brain, spinal cord, and neural tissue. This includes the central nervous system and the peripheral nervous system. Tissue analysis comes from either surgical

text: biopsies

text: or post mortem

text: autopsies

text: . Common tissue samples include muscle fibers and nervous tissue. Common applications of neuropathology include studying samples of tissue in patients who have

text: Parkinson's disease

text: Alzheimer's disease

text: dementia

text: Huntington's disease

text: amyotrophic lateral sclerosis

text: mitochondria disease

text: , and any disorder that has neural deterioration in the brain or spinal cord.

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answers: Neuropathology

answers: NDQ_010490

answers: neuropathology

answers: NDQ_010494

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answers: Huntington's disease

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answers: Parkinson's disease

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answers: mitochondria disease

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answers: amyotrophic lateral sclerosis

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: The southern house mosquito can transmit zoonotic diseases that affect humans and wild and domestic animals, such as lymphatic filariasis, avian malaria, St. Louis encephalitis, Western equine encephalitis, and West Nile fever, and may be a vector of the Zika virus. It causes infection through biting during blood meal. In the southern U.S., it is the primary vector of St. Louis encephalitis virus. In India and Southeast Asia, it is the primary vector of Wuchereria bancrofti, a nematode that causes lymphatic filariasis. It acts as an intermediate host for the helminth parasite by harbouring the larval stages. It also transmits Plasmodium relictum, a malarial parasite of birds, and is the principal vector in Hawaii. It is the definitive host for malarial parasite as it harbours the sexual cycle.

text: The southern house mosquito can transmit

text: zoonotic diseases

text: that affect humans and wild and domestic animals, such as

text: lymphatic filariasis

text: avian malaria

text: St. Louis encephalitis

text: Western equine encephalitis

text: , and

text: West Nile fever

text: , and may be a

text: vector

text: of the

text: Zika virus

text: . It causes infection through biting during blood meal. In the southern U.S., it is the primary vector of St. Louis encephalitis virus. In India and Southeast Asia, it is the primary vector of Wuchereria bancrofti, a nematode that causes lymphatic filariasis. It acts as an intermediate host for the helminth parasite by harbouring the larval stages. It also transmits

text: Plasmodium relictum

text: , a malarial parasite of birds, and is the principal vector in

text: Hawaii

text: . It is the definitive host for malarial parasite as it harbours the sexual cycle.

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answers: St. Louis encephalitis

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answers: encephalitis

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answers: encephalitis

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: The intestinal mucosal barrier, also referred to as intestinal barrier, refers to the property of the intestinal mucosa that ensures adequate containment of undesirable luminal contents within the intestine while preserving the ability to absorb nutrients. The separation it provides between the body and the gut prevents the uncontrolled translocation of luminal contents into the body proper. Its role in protecting the mucosal tissues and circulatory system from exposure to pro-inflammatory molecules, such as microorganisms, toxins, and antigens is vital for the maintenance of health and well-being. Intestinal mucosal barrier dysfunction has been implicated in numerous health conditions such as: food allergies, microbial infections, irritable bowel syndrome, inflammatory bowel disease, celiac disease, metabolic syndrome, non-alcoholic fatty liver disease, diabetes, and septic shock.

text: The intestinal mucosal barrier, also referred to as intestinal barrier, refers to the property of the

text: intestinal mucosa

text: that ensures adequate containment of undesirable

text: luminal

text: contents within the intestine while preserving the ability to absorb

text: nutrient

text: s. The separation it provides between the body and the gut prevents the uncontrolled translocation of luminal contents into the body proper. Its role in protecting the mucosal tissues and

text: circulatory system

text: from exposure to

text: pro-inflammatory molecules

text: , such as

text: microorganism

text: s,

text: toxin

text: s, and

text: antigen

text: s is vital for the maintenance of health and well-being. Intestinal mucosal barrier dysfunction has been implicated in numerous health conditions such as:

text: food allergies

text: microbial infections

text: irritable bowel syndrome

text: inflammatory bowel disease

text: celiac disease

text: metabolic syndrome

text: non-alcoholic fatty liver disease

text: diabetes,

text: and

text: septic shock

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text: Neurosyphilis refers to an infection involving the central nervous system. It may occur early, being either asymptomatic or in the form of syphilitic meningitis, or late as meningovascular syphilis, general paresis, or tabes dorsalis, which is associated with poor balance and lightning pains in the lower extremities. Late neurosyphilis typically occurs 4 to 25 years after the initial infection. Meningovascular syphilis typically presents with apathy and seizure, and general paresis with dementia and tabes dorsalis. Also, there may be Argyll Robertson pupils, which are bilateral small pupils that constrict when the person focuses on near objects but do not constrict when exposed to bright light.

text: Neurosyphilis

text: refers to an infection involving the

text: central nervous system

text: . It may occur early, being either asymptomatic or in the form of syphilitic

text: meningitis

text: , or late as meningovascular syphilis,

text: general paresis

text: , or

text: tabes dorsalis

text: , which is associated with poor balance and lightning pains in the lower extremities. Late neurosyphilis typically occurs 4 to 25 years after the initial infection. Meningovascular syphilis typically presents with apathy and

text: seizure

text: , and general paresis with

text: dementia

text: and

text: tabes dorsalis

text: . Also, there may be

text: Argyll Robertson pupil

text: s, which are bilateral small pupils that constrict when the person focuses on near objects but do not constrict when exposed to bright light.

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answers: The central nervous system

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answer: Yes

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text: Reyes was also a recording artist with two albums under her belt. Reyes became one of the hosts of DOCTORS ON TV, aired on UNTV.

text: Reyes was also a recording artist with two albums under her belt. Reyes became one of the hosts of DOCTORS ON TV, aired on

text: UNTV

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text: Most healthy people clear the infection without treatment, but in 5 to 14 percent of individuals, the organisms disseminate and infect the liver, spleen, eye, or central nervous system. Although some experts recommend not treating typical CSD in immunocompetent patients with mild to moderate illness, treatment of all patients with antimicrobial agents (Grade 2B) is suggested due to the probability of disseminated disease. The preferred antibiotic for treatment is azithromycin since this agent is the only one studied in a randomized controlled study.

text: Most healthy people clear the infection without treatment, but in 5 to 14 percent of individuals, the organisms disseminate and infect the liver, spleen, eye, or central nervous system. Although some experts recommend not treating typical CSD in immunocompetent patients with mild to moderate illness, treatment of all patients with antimicrobial agents (Grade 2B) is suggested due to the probability of disseminated disease. The preferred antibiotic for treatment is

text: azithromycin

text: since this agent is the only one studied in a randomized controlled study.

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text: Viral encephalitis is a type of encephalitis caused by a virus.

text: Viral encephalitis is a type of

text: encephalitis

text: caused by a

text: virus

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answers: encephalitis

answers: NDQ_010490

answers: encephalitis

answers: NDQ_010494

answers: encephalitis

answers: NDQ_010495

answers: encephalitis

answers: NDQ_010496

answers: viral encephalitis

answers: NDQ_010497

answers: encephalitis

answers: NDQ_010498

answers: encephalitis

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: Many different risk factors play a role in causing a neonatal stroke. Some maternal disorders that may contribute to neonatal strokes include: autoimmune disorders, coagulation disorders, prenatal cocaine exposure, infection, congenital heart disease, diabetes, and trauma. Placental disorders that increase the risk of stroke include placental thrombosis, placental abruption, placental infection, and chorioamnionitis. Other disorders that may increase the risk of a neonatal stroke are blood, homocysteine and lipid disorders, such as polycythemia, disseminated intravascular coagulopathy, prothrombin mutation, lipoprotein (a) deficiency, factor VIII deficiency (hemophilia A), and factor V Leiden mutation.  Infectious disorders such as central nervous system (CNS) infection or systemic infection may also contribute.

text: Many different risk factors play a role in causing a neonatal stroke. Some maternal disorders that may contribute to neonatal strokes include:

text: autoimmune disorders

text: coagulation

text: disorders,

text: prenatal cocaine exposure

text: infection

text: congenital heart disease

text: diabetes

text: , and trauma.

text: Placenta

text: l disorders that increase the risk of stroke include placental

text: thrombosis

text: placental abruption

text: , placental infection, and

text: chorioamnionitis

text: . Other disorders that may increase the risk of a neonatal stroke are blood,

text: homocysteine

text: and

text: lipid

text: disorders, such as

text: polycythemia

text: disseminated intravascular coagulopathy

text: prothrombin mutation

text: lipoprotein (a)

text: deficiency,

text: factor VIII

text: deficiency (hemophilia A), and

text: factor V Leiden

text: mutation.  Infectious disorders such as

text: central nervous system (CNS)

text: infection or

text: systemic infection

text: may also contribute.

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answers: NDQ_010489

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answers: 4

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answers: NDQ_010497

answers: 0

answers: NDQ_010498

answers: 0

answers: NDQ_010489

answers: central nervous system

answers: NDQ_010490

answers: Infectious

answers: NDQ_010494

answers: stroke

answers: NDQ_010495

answers: autoimmune disorders

answers: NDQ_010496

answers: stroke

answers: NDQ_010497

answers: prothrombin mutation

answers: NDQ_010498

answers: central nervous system

answer: Yes

answer: Yes

answer: No

answer: Yes

answer: No

answer: 1

text: Most reviews have been negative: "A SICKENING book", "truly grotesque", "from the title alone, you can tell that Melanie's Marvelous Measles is going to be a wild ride" and "atrocious". Some call the book's supporters "crazies", and the author "some hippie-dippie Age of Aquarius type who thinks Natural Equals Good". Other reviewers talk about the effects measles and other vaccine preventable illnesses have on children; Australian Medical Association President Steve Hambleton said: "Last time I saw a kid with measles with the rash they were carried into the surgery and the child looked like a rag doll. The mother was terrified. It's still fatal. About one in 10,000 children will die because of encephalitis". "Even in 'industrialized countries', measles can cause encephalitis, seizures, blindness, heart and nervous system disorders, and (rarely) death". David Gorski writes: "I'm sure children with whooping cough who are coughing so hard that they can't catch their breath for hours on end, with haemophilus influenzae type B who develop pneumonia or meningitis, with polio who develop paralysis, or with measles who develop pneumonia or subacute sclerosing panencephalitis will feel happy to 'embrace childhood disease', at least those who don't end up dead, who can't embrace anything other than the grave." William Sears says: "Complications of the disease include pneumonia and encephalitis, swelling of the brain, that can result in deafness or mental retardation. I've seen a case of measles encephalitis and it's not pretty. There's nothing awesome about it at all".

text: Most reviews have been negative: "A SICKENING book", "truly grotesque", "from the title alone, you can tell that Melanie's Marvelous Measles is going to be a wild ride" and "atrocious". Some call the book's supporters "crazies", and the author "some hippie-dippie Age of Aquarius type who thinks Natural Equals Good". Other reviewers talk about the effects measles and other vaccine preventable illnesses have on children;

text: Australian Medical Association

text: President Steve Hambleton said: "Last time I saw a kid with measles with the rash they were carried into the surgery and the child looked like a rag doll. The mother was terrified. It's still fatal. About one in 10,000 children will die because of encephalitis". "Even in 'industrialized countries', measles can cause encephalitis, seizures, blindness, heart and nervous system disorders, and (rarely) death".

text: David Gorski

text: writes: "I'm sure children with

text: whooping cough

text: who are coughing so hard that they can't catch their breath for hours on end, with

text: haemophilus influenzae

text: type B who develop pneumonia or

text: meningitis

text: , with

text: polio

text: who develop paralysis, or with measles who develop

text: pneumonia

text: or

text: subacute sclerosing panencephalitis

text: will feel happy to 'embrace childhood disease', at least those who don't end up dead, who can't embrace anything other than the grave."

text: William Sears

text: says: "Complications of the disease include pneumonia and encephalitis, swelling of the brain, that can result in deafness or mental retardation. I've seen a case of measles encephalitis and it's not pretty. There's nothing awesome about it at all".

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answers: NDQ_010489

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answers: nervous system disorders

answers: NDQ_010490

answers: encephalitis

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answers: mental retardation

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answers: encephalitis

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answers: polio

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answer: Yes

answer: Yes

answer: No

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answer: No

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text: The pathogenesis of BSE is not well understood or documented like other diseases of this nature. Even though BSE is disease that results in neurological defects, its pathogenesis occurs in areas that reside outside of the nervous system. There was a strong deposition of PrP initially located in the Ileal Peyer’s patches of the small intestine. The lymphatic system has been identified in the pathogenesis of scrapies, it has not, however, been determined to be an essential part of the pathogenesis of BSE. The Ileal Peyer’s patches have been the only organ from this system that has been found to play a major role in the pathogenesis. Infectivity of the Ileal Peyer's patches has been observed as early as 4 months after inoculation. PrP accumulation was found to occur mostly in tangible body macrophages of the Ileal Peyer’s patches. Tangible body macrophages involved in PrP clearance are thought to play a role in in PrP accumulation in the Peyer’s patches. Accumulation of PrP was also found in follicular dendritic cells; however, it was of a lesser degree. Six months after inoculation, there was no infectivity in any tissues, only that of the ileum. This led researchers to believe that the disease agent replicates here. In naturally confirmed cases, there have been no reports of infectivity in the Ileal Peyer’s patches. Generally, in clinical experiments, high doses of the disease are administered. In natural cases, it was hypothesized that low doses of the agent were present, and therefore, infectivity could not be observed.

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answers: BSE

answer: No

answer: No

answer: Yes

answer: No

answer: Yes

answer: 1

text: P. brasiliensis causes a disease known as paracoccidioidomycosis characterized by slow, progressive granulomatous changes in the head mucosa, notably the nose and sinuses or the skin. Uncommonly, the disease affects the lymphatic system, the central nervous system, the gastrointestinal tract, or the skeletal system. Due to the high proportion of cases affecting the oral mucosa,  these tissues were originally thought to be the primary route of entry of fungus. However, strong evidence now indicates  the respiratory tract is the chief point of entry and P. brasiliensis lung lesions occur in nearly a third of progressive cases. The disease is not contagious.Paracoccidioidomycosis is more frequently seen in adult males than females. The hormone estrogen is thought to inhibit the transformation of the mycelial to the yeast form, as supported by in vitro experimental data, and this factor may account for the relative resistance of women to infection.

text: P. brasiliensis causes a disease known as

text: paracoccidioidomycosis

text: characterized by slow, progressive granulomatous changes in the head mucosa, notably the nose and sinuses or the skin. Uncommonly, the disease affects the lymphatic system, the central nervous system, the gastrointestinal tract, or the skeletal system. Due to the high proportion of cases affecting the oral mucosa,  these tissues were originally thought to be the primary route of entry of fungus. However, strong evidence now indicates  the respiratory tract is the chief point of entry and P. brasiliensis lung lesions occur in nearly a third of progressive cases. The disease is not contagious.Paracoccidioidomycosis is more frequently seen in adult males than females. The hormone

text: estrogen

text: is thought to inhibit the transformation of the mycelial to the yeast form, as supported by in vitro experimental data, and this factor may account for the relative resistance of women to infection.

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answer: No

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answer: 1

text: Glial cells, sometimes called neuroglia or simply glia (Greek γλία and γλοία "glue"; pronounced in English as either  or ), are non-neuronal cells that maintain homeostasis, form myelin, and provide support and protection for neurons in the central and peripheral nervous systems. In the central nervous system, glial cells include oligodendrocytes, astrocytes, ependymal cells and microglia, and in the peripheral nervous system glial cells include Schwann cells and satellite cells.

text: Glial cells, sometimes called neuroglia or simply glia (

text: Greek

text: γλία and γλοία "glue"; pronounced in English as either  or ), are non-

text: neuron

text: al

text: cells

text: that maintain

text: homeostasis

text: , form

text: myelin

text: , and provide support and protection for

text: neuron

text: s in the

text: central

text: and

text: peripheral nervous system

text: s. In the central nervous system, glial cells include

text: oligodendrocyte

text: s,

text: astrocyte

text: s,

text: ependymal cell

text: s and

text: microglia

text: , and in the peripheral nervous system glial cells include

text: Schwann cell

text: s and

text: satellite cells

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answer: No

answer: No

answer: No

answer: No

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answer: 1

text: Orofaciodigital syndrome 1 (OFD1), also called Papillon-League and Psaume syndrome, is an X-linked congenital disorder characterized by malformations of the face, oral cavity, and digits with polycystic kidney disease and variable involvement of the central nervous system.

text: Orofaciodigital syndrome 1 (OFD1), also called Papillon-League and Psaume syndrome, is an

text: X-linked

text: congenital disorder

text: characterized by malformations of the

text: face

text: oral cavity

text: , and

text: digits

text: with

text: polycystic kidney disease

text: and variable involvement of the

text: central nervous system

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answers: central nervous system

answers: NDQ_010497

answers: central nervous system

answers: NDQ_010498

answers: central nervous system

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: No

answer: 1

text: The inflammation may be caused by infection with viruses, bacteria, or other microorganisms, and less commonly by certain drugs. Meningitis can be life-threatening because of the inflammation's proximity to the brain and spinal cord; therefore, the condition is classified as a medical emergency. A lumbar puncture diagnoses or excludes meningitis. A needle is inserted into the spinal canal to collect a sample of cerebrospinal fluid (CSF), that envelops the brain and spinal cord. The CSF is examined in a medical laboratory.

text: The inflammation may be caused by infection with

text: virus

text: es,

text: bacteria

text: , or other

text: microorganism

text: s, and less commonly by certain

text: drugs

text: . Meningitis can be life-threatening because of the inflammation's proximity to the brain and spinal cord; therefore, the condition is classified as a

text: medical emergency

text: . A

text: lumbar puncture

text: diagnoses or excludes meningitis. A needle is inserted into the

text: spinal canal

text: to collect a sample of

text: cerebrospinal fluid

text: (CSF), that envelops the brain and spinal cord. The CSF is examined in a medical laboratory.

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answers: NDQ_010497

answers: 0

answers: NDQ_010498

answers: 0

answers: NDQ_010489

answers: Meningitis

answers: NDQ_010490

answers: Meningitis

answers: NDQ_010494

answers: Meningitis

answers: NDQ_010495

answers: Meningitis

answers: NDQ_010496

answers: Meningitis

answers: NDQ_010497

answers: Meningitis

answers: NDQ_010498

answers: Meningitis

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: No

answer: 1

text: The hospital was founded in 1859 by Johanna Chandler and originally called The National Hospital for Diseases of the Nervous System including Paralysis and Epilepsy and later the National Hospital for Nervous Diseases.  The current name, National Hospital for Neurology and Neurosurgery, came into use in 1988. It was merged in 1948 with the Maida Vale Hospital for Nervous Diseases (originally called The London Infirmary for Epilepsy and Paralysis), which in turn owed its foundation in 1866 to a German, Julius Althaus. Great neurologists of the time worked at The National, including John Hughlings Jackson, David Ferrier, MacDonald Critchley, Charles-Édouard Brown-Séquard, William Allen Sturge (discoverer of the Sturge-Weber syndrome), Sir Roger Bannister and many others.

text: The hospital was founded in 1859 by

text: Johanna Chandler

text: and originally called The National Hospital for Diseases of the Nervous System including Paralysis and Epilepsy and later the National Hospital for Nervous Diseases.  The current name, National Hospital for Neurology and Neurosurgery, came into use in 1988. It was merged in 1948 with the Maida Vale Hospital for Nervous Diseases (originally called The London Infirmary for Epilepsy and Paralysis), which in turn owed its foundation in 1866 to a German,

text: Julius Althaus

text: . Great

text: neurologists

text: of the time worked at The National, including

text: John Hughlings Jackson

text: David Ferrier

text: MacDonald Critchley

text: Charles-Édouard Brown-Séquard

text: William Allen Sturge

text: (discoverer of the

text: Sturge-Weber syndrome

text: ), Sir

text: Roger Bannister

text: and many others.

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answers: 2: The answer has limited relevance and completeness, with significant gaps or inaccuracies.

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answers: NDQ_010489

answers: Diseases of the Nervous System

answers: NDQ_010490

answers: It was merged in 1948 with the Maida Vale Hospital for Nervous Diseases

answers: NDQ_010494

answers: Epilepsy

answers: NDQ_010495

answers: Sturge-Weber syndrome

answers: NDQ_010496

answers: Epilepsy

answers: NDQ_010497

answers: Sturge-Weber syndrome

answers: NDQ_010498

answers: Epilepsy

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: There are no currently known causes of this disease. There are studies currently proposing several theories of the causes which include inflammation of the adipose tissue, nervous system malfunction and endocrine malfunction. None of the theories that are currently proposed have been found viable. Since little is known about Dercum's Disease, there are currently no known modes of prevention. Some theories state that maintaining a healthy weight and diet can help prevent Dercum's although it has not been proven.

text: There are no currently known causes of this disease. There are studies currently proposing several theories of the causes which include inflammation of the adipose tissue, nervous system malfunction and endocrine malfunction. None of the theories that are currently proposed have been found viable. Since little is known about Dercum's Disease, there are currently no known modes of prevention. Some theories state that maintaining a healthy weight and diet can help prevent Dercum's although it has not been proven.

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answers: NDQ_010489

answers: 4

answers: NDQ_010490

answers: 0

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answers: 0

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answers: 0

answers: NDQ_010496

answers: 0

answers: NDQ_010497

answers: 0

answers: NDQ_010498

answers: 0

answers: NDQ_010489

answers: nervous system malfunction

answers: NDQ_010490

answers: no currently known causes

answers: NDQ_010494

answers: Dercum's Disease

answers: NDQ_010495

answers: Dercum's Disease

answers: NDQ_010496

answers: Dercum's Disease

answers: NDQ_010497

answers: Dercum's Disease

answers: NDQ_010498

answers: Dercum's Disease

answer: No

answer: No

answer: Yes

answer: No

answer: Yes

answer: 1

text: C cell– c-erbB-2– c-kit– CA 19-9 assay– CA-125– CA-125 test– cachexia– calcitonin– calcitriol– CAM– Campath-1H– camptothecin– camptothecin analog– cancer– cancer induction– Cancer Information Service– cancer of unknown primary origin– Cancer stem cell– cancer vaccine– Cancer.gov– Candidiasis– Candidosis– CAP-1– capecitabine– capsaicin– captopril– carbendazim– carbogen– carbon-11 acetate– carboplatin– carboxyamidotriazole– carboxypeptidase-G2– carcinoembryonic antigen– carcinoembryonic antigen peptide-1– carcinogen– carcinogenesis– carcinoid– carcinoid syndrome– carcinoma– carcinoma in situ– carcinomatosis– carcinosarcoma– carcinosis– carcinostatic– cardin (oncology)– carmustine– carnitine– carotenoid– carzelesin– case report– case series– case-control study– caspofungin acetate– Castleman's disease– CAT scan– catechol– cauterization– cauterize– cBR96-doxorubicin immunoconjugate– CC-1088– CC-49 monoclonal antibody– CC-5013– CC-8490– CCI-779– CD34 antigen– CD40-ligand– CEA– CEA assay– cecum– cefalexin– cefepime– cefixime– ceftriaxone– celecoxib– celiac disease– cell– cell differentiation– cell motility– cell proliferation– cell respiration– cell adhesion– cellular adoptive immunotherapy– cellular metabolism– cellulitis– central nervous system primitive neuroectodermal tumor– central venous access catheter– CEP-2563 dihydrochloride– CEP-701– cephalosporin– ceramide– cerebellar hemangioblastoma– cerebellopontine– cerebral hemisphere– cerebrospinal fluid– cerebrospinal fluid diversion– cervical– cervical intraepithelial neoplasia– cervix– cetuximab– cevimeline– CGP 48664– Chamberlain procedure– chemoembolization– chemoimmunotherapy– chemoprevention– chemoprevention studies– chemoprotective– chemoradiation– chemoradiotherapy– chemosensitivity– chemosensitivity assay– chemosensitizer– chemotherapeutic agent– chemotherapy– chemotherapy-induced peripheral neuropathy– chest x-ray– chiasma– child-life worker– chitin– chlorambucil– chlorine– chloroma– chloroquinoxaline sulfonamide– cholangiocarcinoma– cholangiosarcoma– cholelith– cholestasis– chondrocyte– chondroitin sulfate– chondrosarcoma– chordoma– chorioallantoic membrane– choriocarcinoma– choroid plexus tumor– CHPP– chronic granulocytic leukemia– chronic idiopathic myelofibrosis– chronic leukemia– chronic lymphoblastic leukemia– chronic lymphocytic leukemia– chronic myelogenous leukemia– chronic myeloid leukemia– chronic myelomonocytic leukemia– chronic phase– chronic phase chronic myelogenous leukemia– CHS 828– CI-1033– CI-958– CI-980– CI-994– ciclosporin– cidofovir– cilengitide– cimetidine– Cipro– ciprofloxacin– circulatory system– cirrhosis– CIS– cisplatin– citric acid/potassium-sodium citrate– cladribine– clarithromycin– clear cell adenocarcinoma– clear-cell sarcoma– clear cell sarcoma of the kidney– clinical breast exam– clinical resistance– clinical series– clinical study– clinical trial– CLL– clodronate– clofarabine– CML– CMML– CMV– cnicin– CNS– CNS metastasis– CNS prophylaxis– CNS sanctuary– CNS tumor– co-culture– co-trimoxazole– coactivated T cell– cobalt 60– Cockayne syndrome– coenzyme Q10– cohort study– COL-3– cold nodule- Coley's toxins– collagen disease– collagenase– collecting duct– coloanal anastomosis– coloanal pull-through– Colorectal cancer (colon cancer)– colon polyp– colonoscope– colonoscopy– colony-stimulating factor– colorectal– colposcope– colposcopy– combination chemotherapy– combretastatin A4 phosphate– comedo carcinoma– common bile duct– comorbidity– compassionate use trial– complementary and alternative medicine– complete blood count (CBC)– complete hysterectomy– complete metastasectomy– complete remission– complete response– compound nevus– compression bandage– computed tomographic colonography– computed tomography– computed tomography colography– computerized axial tomography– computerized tomography– concurrent therapy– condylomata acuminata– cone biopsy– congestive heart failure– conization– consecutive case series– consolidation therapy– contiguous lymphoma– continent reservoir– contingency management– continuous hyperthermic peritoneal perfusion– continuous infusion– contralateral– control animal– control group– controlled clinical trial– controlled study– conventional therapy– conventional treatment– cooperative group– CoQ10– cordectomy– cordycepin– core biopsy– corticosteroid– Corynebacterium granulosum– coumestan– coumestrol– CP-358,774– CP-609,754– CP-724,714– CP4071– CpG 7909– CPT 11– CQS– craniopharyngioma– craniotomy– creatine– creatinine– cribriform– crisnatol mesylate– Crohn's disease– cryopreservation– cryosurgery– cryotherapy– cryptorchidism– CSF– CT scan– CT-2103– CT-2106– CT-2584– CTC– cultured cell– cultured cell line– cumulative dose– curcumin– cutaneous breast cancer– cutaneous T-cell lymphoma– cyanogenic glucoside– cyclooxygenase-2 selective inhibitor– cyclophosphamide– cyclosporine– cyproheptadine– cyproterone acetate– cyst– cystectomy– cystosarcoma phyllodes– cystoscope– cystoscopy– cytarabine– cytochlor– cytogenetics– cytokine– cytology– cytomegalovirus– cytopenia– cytoplasm– cytotoxic– cytotoxic chemotherapy– cytotoxic T cell

text: C cell

text: c-kit

text: CA 19-9 assay

text: CA-125 test

text: cachexia

text: calcitonin

text: calcitriol

text: CAM

text: camptothecin

text: camptothecin analog

text: cancer

text: cancer induction

text: Cancer Information Service

text: cancer of unknown primary origin

text: Cancer stem cell

text: cancer vaccine

text: Cancer.gov

text: Candidiasis

text: Candidosis

text: capecitabine

text: capsaicin

text: captopril

text: carbendazim

text: carbogen

text: carbon-11 acetate

text: carboplatin

text: carboxyamidotriazole

text: carcinoembryonic antigen

text: carcinoembryonic antigen peptide-1

text: carcinogen

text: carcinogenesis

text: carcinoid

text: carcinoid syndrome

text: carcinoma

text: carcinoma in situ

text: carcinomatosis

text: carcinosarcoma

text: carcinosis

text: carcinostatic

text: cardin (oncology)

text: carmustine

text: carnitine

text: carotenoid

text: carzelesin

text: case report

text: case series

text: case-control study

text: caspofungin acetate

text: Castleman's disease

text: CAT scan

text: catechol

text: cauterization

text: cauterize

text: cBR96-doxorubicin immunoconjugate

text: CC-49 monoclonal antibody

text: CD34 antigen

text: CEA

text: CEA assay

text: cecum

text: cefalexin

text: cefepime

text: cefixime

text: ceftriaxone

text: celecoxib

text: celiac disease

text: cell

text: cell differentiation

text: cell motility

text: cell proliferation

text: cell respiration

text: cell adhesion

text: cellular adoptive immunotherapy

text: cellular metabolism

text: cellulitis

text: central nervous system primitive neuroectodermal tumor

text: central venous access catheter

text: CEP-2563 dihydrochloride

text: cephalosporin

text: ceramide

text: cerebellar hemangioblastoma

text: cerebellopontine

text: cerebral hemisphere

text: cerebrospinal fluid

text: cerebrospinal fluid diversion

text: cervical

text: cervical intraepithelial neoplasia

text: cervix

text: cetuximab

text: cevimeline

text: CGP 48664

text: Chamberlain procedure

text: chemoembolization

text: chemoimmunotherapy

text: chemoprevention

text: chemoprevention studies

text: chemoprotective

text: chemoradiation

text: chemoradiotherapy

text: chemosensitivity

text: chemosensitivity assay

text: chemosensitizer

text: chemotherapeutic agent

text: chemotherapy

text: chemotherapy-induced peripheral neuropathy

text: chest x-ray

text: chiasma

text: child-life worker

text: chitin

text: chlorambucil

text: chlorine

text: chloroma

text: chloroquinoxaline sulfonamide

text: cholangiocarcinoma

text: cholangiosarcoma

text: cholelith

text: cholestasis

text: chondrocyte

text: chondroitin sulfate

text: chondrosarcoma

text: chordoma

text: chorioallantoic membrane

text: choriocarcinoma

text: choroid plexus tumor

entity_name: Continuous hyperthermic peritoneal perfusion

text: CHPP

text: chronic granulocytic leukemia

text: chronic idiopathic myelofibrosis

text: chronic leukemia

text: chronic lymphoblastic leukemia

text: chronic lymphocytic leukemia

text: chronic myelogenous leukemia

text: chronic myeloid leukemia

text: chronic myelomonocytic leukemia

text: chronic phase

entity_name: Chronic phase chronic myelogenous leukemia

text: chronic phase chronic myelogenous leukemia

text: CHS 828

text: ciclosporin

text: cidofovir

text: cilengitide

text: cimetidine

text: Cipro

text: ciprofloxacin

text: circulatory system

text: cirrhosis

text: CIS

text: cisplatin

text: citric acid/potassium-sodium citrate

text: cladribine

text: clarithromycin

text: clear cell adenocarcinoma

text: clear-cell sarcoma

text: clear cell sarcoma of the kidney

text: clinical breast exam

text: clinical resistance

text: clinical series

text: clinical study

text: clinical trial

text: CLL

text: clodronate

text: clofarabine

text: CML

text: CMML

text: CMV

text: cnicin

text: CNS

text: CNS metastasis

text: CNS prophylaxis

text: CNS sanctuary

text: CNS tumor

text: co-culture

text: co-trimoxazole

text: coactivated T cell

text: cobalt 60

text: Cockayne syndrome

text: coenzyme Q10

text: cohort study

text: cold nodule

text: Coley's toxins

text: collagen disease

text: collagenase

text: collecting duct

text: coloanal anastomosis

text: coloanal pull-through

text: Colorectal cancer

text: (colon cancer)–

text: colon polyp

text: colonoscope

text: colonoscopy

text: colony-stimulating factor

text: colorectal

text: colposcope

text: colposcopy

text: combination chemotherapy

text: combretastatin A4 phosphate

text: comedo carcinoma

text: common bile duct

text: comorbidity

text: compassionate use trial

text: complementary and alternative medicine

text: complete blood count

text: (CBC)–

text: complete hysterectomy

text: complete metastasectomy

text: complete remission

text: complete response

text: compound nevus

text: compression bandage

text: computed tomographic colonography

text: computed tomography

text: computed tomography colography

text: computerized axial tomography

text: computerized tomography

text: concurrent therapy

text: condylomata acuminata

text: cone biopsy

text: congestive heart failure

text: conization

text: consecutive case series

text: consolidation therapy

text: contiguous lymphoma

text: continent reservoir

text: contingency management

text: continuous hyperthermic peritoneal perfusion

text: continuous infusion

text: contralateral

text: control animal

text: control group

text: controlled clinical trial

text: controlled study

text: conventional therapy

text: conventional treatment

text: cooperative group

text: cordectomy

text: cordycepin

text: core biopsy

text: corticosteroid

text: Corynebacterium granulosum

text: coumestan

text: coumestrol

text: CpG 7909

text: CPT 11

text: CQS

text: craniopharyngioma

text: craniotomy

text: creatine

text: creatinine

text: cribriform

text: crisnatol mesylate

text: Crohn's disease

text: cryopreservation

text: cryosurgery

text: cryotherapy

text: cryptorchidism

text: CSF

text: CT scan

text: CTC

text: cultured cell

text: cultured cell line

text: cumulative dose

text: curcumin

text: cutaneous breast cancer

text: cutaneous T-cell lymphoma

text: cyanogenic glucoside

text: cyclooxygenase-2 selective inhibitor

text: cyclophosphamide

text: cyclosporine

text: cyproheptadine

text: cyproterone acetate

text: cyst

text: cystectomy

text: cystosarcoma phyllodes

text: cystoscope

text: cystoscopy

text: cytarabine

text: cytochlor

text: cytogenetics

text: cytokine

text: cytology

text: cytomegalovirus

text: cytopenia

text: cytoplasm

text: cytotoxic

text: cytotoxic chemotherapy

text: cytotoxic T cell

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answers: NDQ_010489

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answers: 0

answers: NDQ_010489

answers: CEP-2563 dihydrochloride– CEP-701– cephalosporin

answers: NDQ_010490

answers: cns diseases can be caused by bacteria and viruses getting into the brain or spinal cord.

answers: NDQ_010494

answers: Castleman's disease

answers: NDQ_010495

answers: cellulitis

answers: NDQ_010496

answers: Castleman's disease

answers: NDQ_010497

answers: central nervous system primitive neuroectodermal tumor

answers: NDQ_010498

answers: Castleman's disease

answer: No

answer: Yes

answer: No

answer: No

answer: No

answer: 1

text: The syndrome is named after Dr. Douglas Reye, who, along with fellow physicians Drs. Graeme Morgan and Jim Baral, published the first study of the syndrome in 1963 in The Lancet. In retrospect, the occurrence of the syndrome may have first been reported in 1929. Also in 1964, Dr. George Johnson and colleagues published an investigation of an outbreak of influenza B that described 16 children who developed neurological problems, four of whom had a profile remarkably similar to Reye syndrome.  Some investigators refer to this disorder as Reye-Johnson syndrome, although it is more commonly called Reye syndrome. In 1979, Dr. Karen Starko and colleagues conducted a case-control study in Phoenix, Arizona and found the first statistically significant link between aspirin use and Reye syndrome. Studies in Ohio and Michigan soon confirmed her findings  pointing to the use of aspirin during an upper respiratory tract or chickenpox infection as a possible trigger of the syndrome.  Beginning in 1980, the CDC cautioned physicians and parents about the association between Reye syndrome and the use of salicylates in children and teenagers with chickenpox or virus-like illnesses.  In 1982 the U.S. Surgeon General issued an advisory, and in 1986 the Food and Drug Administration required a Reye syndrome-related warning label for all aspirin-containing medications.

text: The syndrome is named after Dr.

text: Douglas Reye

text: , who, along with fellow physicians Drs. Graeme Morgan and Jim Baral, published the first study of the syndrome in 1963 in

text: The Lancet

text: Food and Drug Administration

text: required a Reye syndrome-related warning label for all aspirin-containing medications.

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answers: neurological problems

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answers: neurological problems

answers: NDQ_010494

answers: Reye syndrome

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answers: Reye syndrome

answers: NDQ_010496

answers: Reye syndrome

answers: NDQ_010497

answers: Reye syndrome

answers: NDQ_010498

answers: neurological problems

answer: Yes

answer: Yes

answer: No

answer: Yes

answer: No

answer: 1

text: Neuroscientists focus primarily on the study and research of the nervous system. The nervous system is composed of the brain, spinal cord and nerve cells. Studies of the nervous system may focus on the cellular level, as in studies of the ion channels, or instead may focus on broader aspects of nervous system function as in behavioral studies. A significant portion of nervous system studies is devoted to understanding the diseases that affect the nervous system, like multiple sclerosis, Alzheimer's, Parkinson's, and Lou Gehrig's. Research commonly occurs in private, government and public research institutions and universities.

text: Neuroscientists focus primarily on the study and research of the

text: nervous system

text: . The nervous system is composed of the brain, spinal cord and

text: nerve

text: cells. Studies of the nervous system may focus on the cellular level, as in studies of the

text: ion channel

text: s, or instead may focus on broader aspects of nervous system function as in behavioral studies. A significant portion of nervous system studies is devoted to understanding the diseases that affect the nervous system, like

text: multiple sclerosis

text: Alzheimer's

text: Parkinson's

text: , and

text: Lou Gehrig's

text: . Research commonly occurs in private, government and public research institutions and universities.

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answers: Multiple sclerosis, Alzheimer's, Parkinson's

answers: NDQ_010490

answers: The nervous system is composed of the brain, spinal cord and nerve cells

answers: NDQ_010494

answers: Alzheimer's

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answers: Lou Gehrig's

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answers: Parkinson's

answers: NDQ_010497

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answers: Lou Gehrig's

answer: Yes

answer: Yes

answer: No

answer: Yes

answer: No

answer: 1

text: California encephalitis virus belongs to the Bunyaviridae family of viruses. The La Crosse Virus from the same genus is also a common cause of encephalitis in the United States. Other viruses with similar disease symptoms but genetically unrelated include: St. Louis Encephalitis and West Nile Virus.

text: California encephalitis virus belongs to the

text: Bunyaviridae

text: family of viruses. The

text: La Crosse Virus

text: from the same genus is also a common cause of encephalitis in the United States. Other viruses with similar disease symptoms but genetically unrelated include:

text: St. Louis Encephalitis

text: and

text: West Nile Virus

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answers: encephalitis

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answers: encephalitis

answers: NDQ_010496

answers: California encephalitis

answers: NDQ_010497

answers: encephalitis

answers: NDQ_010498

answers: California encephalitis virus

answer: Yes

answer: Yes

answer: No

answer: Yes

answer: Yes

answer: 1

text: All Down syndrome (DS) patients progress neuropathological changes identical to the pathogenesis of Alzheimer disease (AD) after their middle age, such as neurotic plaques and neuronal loss in their brain, Therefor, DS patients are perfect models to study AD pathogenesis. Chronic DSCR1 overexpression is related with DS and, while its shortage is reported in Huntington’s disease. DSCR1 expressed excessively in the Central nervous system of embryos during their development, this protein is later overexpressed in differentiating neurons regions such as Brains of DS patients. However, neurotrophic peptide PACAP (or Pituitary adenylate cyclase-activating peptide) which is responsible for the development, differentiation, and survival, and various parts of memory and learning, Target RCNA1 in a Down syndrome related gene, induces the expression of regulator of calcineurin 1, through activation of the PKA-CREB pathway, and this is important to understand the mechanisms of neural differentiation and aim for proper expression of RCAN1.

text: All Down syndrome (DS) patients progress neuropathological changes identical to the pathogenesis of

text: Alzheimer disease

text: (AD) after their middle age, such as neurotic plaques and neuronal loss in their brain, Therefor, DS patients are perfect models to study AD pathogenesis. Chronic DSCR1 overexpression is related with DS and, while its shortage is reported in

text: Huntington’s disease

text: . DSCR1 expressed excessively in the Central nervous system of embryos during their development, this protein is later overexpressed in differentiating neurons regions such as Brains of DS patients. However, neurotrophic peptide

entity_name: Pituitary adenylate cyclase-activating peptide

text: PACAP

text: (or Pituitary adenylate cyclase-activating peptide) which is responsible for the development, differentiation, and survival, and various parts of memory and learning, Target RCNA1 in a Down syndrome related gene, induces the expression of regulator of calcineurin 1, through activation of the PKA-CREB pathway, and this is important to understand the mechanisms of neural differentiation and aim for proper expression of RCAN1.

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answer: Yes

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text: This criteria is typically validated through biopsy of the cutaneous lesions and imaging of the central nervous system. It is important to establish that the cutaneous lesions are benign. If not, then the melanocytic deposits in the central nervous system may be the result of metastasis of cutaneous melanoma and not neurocutaneous melanosis.

text: This criteria is typically validated through

text: biopsy

text: of the cutaneous lesions and imaging of the central nervous system. It is important to establish that the cutaneous lesions are benign. If not, then the melanocytic deposits in the central nervous system may be the result of metastasis of cutaneous melanoma and not neurocutaneous melanosis.

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answer: No

answer: No

answer: No

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answer: 1

text: The serious symptoms of Reye syndrome appear to result from damage to cellular mitochondria, at least in the liver, and there are a number of ways that aspirin could cause or exacerbate mitochondrial damage. A potential increased risk of developing Reye syndrome is one of the main reasons that aspirin has not been recommended for use in children and teenagers, the age group for which the risk of lasting serious effects is highest.

text: The serious symptoms of Reye syndrome appear to result from damage to cellular

text: mitochondria

text: , at least in the liver, and there are a number of ways that aspirin could cause or exacerbate mitochondrial damage. A potential increased risk of developing Reye syndrome is one of the main reasons that aspirin has not been recommended for use in children and teenagers, the age group for which the risk of lasting serious effects is highest.

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text: Other causes include medication side effects and various disease processes. The use of stimulants such as cocaine and amphetamines which can result in urinary retention, and the use of CNS depressants and other psychoactive drugs can also lead to urinary retention and constipation thus leading to autonomic dysreflexia when in use over an extended period of time. Guillain–Barré syndrome is a demyelinating disease that can result in peripheral paralysis and can progress to encompass autonomic functions, leading to a loss of normal respiratory, bladder and bowel function resulting in autonomic dysreflexia. Severe head trauma and other brain injuries can instigate autonomic dysreflexia at the central nervous system by interfering with the reception of the signal that brings the urge to void the bladder and bowels and with the voluntary ability to micturate and defecate.Other causal theories for autonomic dysreflexia include  noxious stimuli, or painful stimuli arising from the peripheral sensory neurons. These stimuli are interrupted in their journey to the brain due to a transection of the spine result in a paradoxical stimulation of autonomic pathways of the autonomic nervous system.

text: Other causes include medication side effects and various disease processes. The use of stimulants such as cocaine and amphetamines which can result in urinary retention, and the use of CNS depressants and other psychoactive drugs can also lead to urinary retention and constipation thus leading to autonomic dysreflexia when in use over an extended period of time.

text: Guillain–Barré syndrome

text: is a demyelinating disease that can result in peripheral paralysis and can progress to encompass autonomic functions, leading to a loss of normal respiratory, bladder and bowel function resulting in autonomic dysreflexia. Severe head trauma and other brain injuries can instigate autonomic dysreflexia at the

text: central nervous system

text: by interfering with the reception of the signal that brings the urge to void the bladder and bowels and with the voluntary ability to micturate and defecate.Other causal theories for autonomic dysreflexia include

text: noxious stimuli

text: , or painful stimuli arising from the peripheral sensory neurons. These stimuli are interrupted in their journey to the brain due to a

text: transection

text: of the spine result in a paradoxical stimulation of autonomic pathways of the

text: autonomic nervous system

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text: Arbovirus encephalitis refers to encephalitis that is caused by arbovirus infection.

text: Arbovirus encephalitis refers to

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text: Chaetomium atrobrunneum is a rare pathogen of humans that tends to infect the tissues of the central nervous system. Its pathogenicity is thought to be supported by its ability to grow at high temperatures. This species has been reported to be an agent of fatal brain abscesses in immunologically impaired people. It can also cause systemic disseminated phaeohyphomycosis affecting other organs including the lungs. Infections due to this species have typically occurred following invasive procedures such as intravenous drug administration and renal transplantation.

text: Chaetomium atrobrunneum is a rare pathogen of humans that tends to infect the tissues of the

text: central nervous system

text: . Its pathogenicity is thought to be supported by its ability to grow at high temperatures. This species has been reported to be an agent of fatal brain abscesses in immunologically impaired people. It can also cause systemic disseminated

text: phaeohyphomycosis

text: affecting other organs including the lungs. Infections due to this species have typically occurred following invasive procedures such as intravenous drug administration and renal transplantation.

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answers: Chaetomium atrobrunneum

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answer: Yes

answer: Yes

answer: Yes

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answer: No

answer: 1

text: The enteric nervous system has been described as a "second brain" for several reasons. The enteric nervous system can operate autonomously. It normally communicates with the central nervous system (CNS) through the parasympathetic (e.g., via the vagus nerve) and sympathetic (e.g., via the prevertebral ganglia) nervous systems.  However, vertebrate studies show that when the vagus nerve is severed, the enteric nervous system continues to function.

text: The enteric nervous system has been described as a "second brain" for several reasons. The enteric nervous system can operate autonomously. It normally communicates with the

text: central nervous system

text: (CNS) through the

text: parasympathetic

text: (e.g., via the

text: vagus nerve

text: ) and

text: sympathetic

text: (e.g., via the

text: prevertebral ganglia

text: ) nervous systems.  However,

text: vertebrate

text: studies show that when the

text: vagus nerve

text: is severed, the enteric nervous system continues to function.

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answers: The central nervous system

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answer: No

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text: Opsoclonus Myoclonus Syndrome (OMS), also known as Opsoclonus-Myoclonus-Ataxia (OMA), is a rare neurological disorder of unknown causes which appears to be the result of an autoimmune process involving the nervous system. It is an extremely rare condition, affecting as few as 1 in 10,000,000 people per year. It affects 2 to 3% of children with neuroblastoma and has been reported to occur with celiac disease and diseases of neurologic and autonomic dysfunction.

text: Opsoclonus Myoclonus Syndrome (OMS), also known as Opsoclonus-Myoclonus-Ataxia (OMA), is a rare

text: neurological

text: disorder

text: of unknown causes which appears to be the result of an

text: autoimmune

text: process involving the

text: nervous system

text: . It is an extremely rare condition, affecting as few as 1 in 10,000,000 people per year. It affects 2 to 3% of children with

text: neuroblastoma

text: and has been reported to occur with

text: celiac disease

text: and diseases of neurologic and autonomic dysfunction.

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answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: The major causes of CVI are as follows: asphyxia, hypoxia (a lack of sufficient oxygen in the body’s blood cells), or ischemia (not enough blood supply to the brain), all of which may occur during the birth process; developmental brain defects; head injury; hydrocephalus (when the cerebrospinal fluid does not circulate properly around the brain, and collects in the head, putting pressure on the brain); a stroke involving the occipital lobe; and infections of the central nervous system, such as meningitis and encephalitis.

text: The major causes of CVI are as follows: asphyxia, hypoxia (a lack of sufficient oxygen in the body’s blood cells), or ischemia (not enough blood supply to the brain), all of which may occur during the birth process; developmental brain defects; head injury; hydrocephalus (when the cerebrospinal fluid does not circulate properly around the brain, and collects in the head, putting pressure on the brain); a

text: stroke

text: involving the

text: occipital lobe

text: ; and infections of the central nervous system, such as meningitis and encephalitis.

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text: The neuraxis or sometimes neuroaxis is the axis of the central nervous system. It denotes the direction in which the central nervous system lies. During embryological development, the neuraxis is bent by various flexures, contributing to the mature structure of the brain and spinal cord.

text: The neuraxis or sometimes neuroaxis is the axis of the

text: central nervous system

text: . It denotes the direction in which the central nervous system lies. During embryological development, the neuraxis is bent by various flexures, contributing to the mature structure of the

text: brain

text: and

text: spinal cord

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text: suPARnostic is a simplified double monoclonal antibody sandwich enzyme-linked immunosorbent assay (ELISA) that measures the amount of soluble urokinase plasminogen activator receptor (suPAR) in blood. Elevated plasma suPAR levels have been observed in various infectious, inflammatory and autoimmune diseases.  suPAR concentration positively correlates to the activation level of the immune system. suPARnostic can be used as a prognostic tool to determine the severity of a disease within a patient, but is not used as a reliable diagnostic tool, as it can detect the severity of the immune response in a patient, but does not reveal the specific disease from which the patient may be suffering. Recently, increase suPAR levels were shown to be associated with increased risk of systemic inflammatory response syndrome (SIRS)/sepsis, cardiovascular disease, type 2 diabetes, infectious diseases, HIV, cancer tuberculosis, malaria, bacterial and viral CNS infections, rheumatoid arthritis, multiple sclerosis and mortality in the general population.

text: suPARnostic is a simplified double monoclonal antibody sandwich enzyme-linked immunosorbent assay (

text: ELISA

text: ) that measures the amount of soluble urokinase plasminogen activator receptor (

text: suPAR

text: ) in blood. Elevated plasma suPAR levels have been observed in various

text: infectious

text: inflammatory

text: and

text: autoimmune diseases

text: .  suPAR concentration positively correlates to the activation level of the immune system. suPARnostic can be used as a prognostic tool to determine the severity of a disease within a patient, but is not used as a reliable diagnostic tool, as it can detect the severity of the immune response in a patient, but does not reveal the specific disease from which the patient may be suffering. Recently, increase suPAR levels were shown to be associated with increased risk of systemic inflammatory response syndrome (

text: SIRS

text: sepsis

text: cardiovascular disease

text: type 2 diabetes

text: infectious diseases

text: HIV

text: cancer

text: tuberculosis

text: malaria

text: , bacterial and viral

entity_name: List of infections of the central nervous system

text: CNS infection

text: s,

text: rheumatoid arthritis

text: multiple sclerosis

text: and

text: mortality

text: in the general population.

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answers: multiple sclerosis

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answer: Yes

answer: Yes

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text: Controversies were raised about the cooperation of Levi-Montalcini with the Italian pharmaceutical industry Fidia . While working for Fidia, she improved the understanding of gangliosides. Beginning in 1975, the scientist supported the drug Cronassial (a particular ganglioside) produced by Fidia from bovine brain tissue. Independent studies showed that the drug actually could be successful in treatment of intended diseases (periphrastic nervous system neuropathies). Years later, some patients under treatment with Cronassial reported a severe neurological syndrome (Guillain–Barré syndrome). As per the normal cautionary routine, Germany banned Cronassial in 1983, followed by other countries. Italy prohibited the drug only in 1993; at the same time, an investigation revealed that Fidia paid the Italian Ministry of Health for a quick approval of Cronassial and later paid for pushing use of the drug in treatment of diseases where it had not been tested.  Levi-Montalcini's relationship with the company was revealed during the investigation, and she was criticized publicly.

text: Controversies were raised about the cooperation of Levi-Montalcini with the Italian pharmaceutical industry

text: Fidia

text: . While working for Fidia, she improved the understanding of

text: gangliosides

text: . Beginning in 1975, the scientist supported the drug

text: Cronassial

text: (a particular ganglioside) produced by Fidia from bovine

text: brain

text: tissue. Independent studies showed that the drug actually could be successful in treatment of intended diseases (periphrastic nervous system neuropathies). Years later, some patients under treatment with Cronassial reported a severe neurological syndrome (

text: Guillain–Barré syndrome

text: ). As per the normal cautionary routine, Germany banned Cronassial in 1983, followed by other countries. Italy prohibited the drug only in 1993; at the same time, an investigation revealed that Fidia paid the Italian Ministry of Health for a quick approval of Cronassial and later paid for pushing use of the drug in treatment of diseases where it had not been tested.  Levi-Montalcini's relationship with the company was revealed during the investigation, and she was criticized publicly.

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answer: Yes

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text: Westphal reported the first documented case of post-viral cerebellar ataxia in 1872, where associations of reversible cerebellar syndrome were observed. Another early case was documented in 1905. Batten described in detail cases of post-infectious cerebellar ataxia in five children. The cause of the disease was unknown until 1978 when Weiss and Guberman proposed that ACA could be due to direct invasion of the central nervous system by infectious agents. Since then many case studies have followed to understand the underlying conditions, symptoms and causes of the disease. The largest study of retrospective childhood ACA was done in 1994 by Connolly. This disease is still commonly used as a reference in clinical practice for other inflammatory and autoimmune disorders of the nervous system.

text: Westphal reported the first documented case of post-viral cerebellar ataxia in 1872, where associations of reversible cerebellar syndrome were observed. Another early case was documented in 1905. Batten described in detail cases of post-infectious cerebellar ataxia in five children. The cause of the disease was unknown until 1978 when Weiss and Guberman proposed that ACA could be due to direct invasion of the central nervous system by infectious agents. Since then many case studies have followed to understand the underlying conditions, symptoms and causes of the disease. The largest study of retrospective childhood ACA was done in 1994 by Connolly. This disease is still commonly used as a reference in clinical practice for other inflammatory and autoimmune disorders of the nervous system.

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text: CFS is not included as a coded term in the 1992 ICD-10, WHO created a new category G93, Other disorders of brain, in Chapter VI, Diseases of the Nervous System, and created a new code G93.3, post-viral fatigue syndrome (PVFS), a condition which was previously in the symptom chapter of ICD-9. WHO also moved benign myalgic encephalomyelitis to G93.3, subordinate to PVFS. The alphabetic index contains other terms, such as chronic fatigue syndrome, to which WHO assigned the same code.

text: CFS is not included as a coded term in the 1992

entity_name: ICD-10 Chapter VI: Diseases of the nervous system

text: , WHO created a new category G93, Other disorders of brain, in Chapter VI, Diseases of the Nervous System, and created a new code G93.3,

text: post-viral fatigue syndrome

text: (PVFS), a condition which was previously in the symptom chapter of ICD-9. WHO also moved benign myalgic encephalomyelitis to G93.3, subordinate to PVFS. The alphabetic index contains other terms, such as chronic fatigue syndrome, to which WHO assigned the same code.

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answer: 1

text: The sympathetic nervous system (SNS) is one of the two main divisions of the autonomic nervous system, the other being the parasympathetic nervous system. The autonomic nervous system functions to regulate the body's unconscious actions. The sympathetic nervous system's primary process is to stimulate the body's fight-or-flight response. It is, however, constantly active at a basic level to maintain homeostasis. The sympathetic nervous system is described as being complementary to the parasympathetic nervous system which stimulates the body to "rest-and-digest" or "Feed and breed".

text: The sympathetic nervous system (SNS) is one of the two main divisions of the

text: autonomic nervous system

text: , the other being the

text: parasympathetic nervous system

text: . The autonomic nervous system functions to regulate the body's unconscious actions. The sympathetic nervous system's primary process is to stimulate the body's

text: fight-or-flight response

text: . It is, however, constantly active at a basic level to maintain

text: homeostasis

text: . The sympathetic nervous system is described as being complementary to the parasympathetic nervous system which stimulates the body to "rest-and-digest" or "Feed and breed".

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answers: The autonomic nervous system functions to regulate the body's unconscious actions

answers: NDQ_010490

answers: The sympathetic nervous system

answers: NDQ_010494

answers: autonomic

answers: NDQ_010495

answers: immune system

answers: NDQ_010496

answers: autonomic nervous system

answers: NDQ_010497

answers: autonomic

answers: NDQ_010498

answers: autonomic nervous system

answer: No

answer: No

answer: No

answer: No

answer: No

answer: 1

text: Tafamidis functions by kinetic stabilization of the correctly folded tetrameric form of the transthyretin (TTR) protein. In patients with FAP, this protein dissociates in a process that is rate limiting for aggregation including amyloid fibril formation, causing failure of the autonomic nervous system and/or the peripheral nervous system (neurodegeneration) initially and later failure of the heart. Kinetic Stabilization of tetrameric transthyretin in familial amyloid polyneuropathy patients provides the first pharmacologic evidence that the process of amyloid fibril formation causes this disease, as treatment with tafamidis dramatically slows the process of amyloid fibril formation and the degeneration of post-mitotic tissue. Sixty % of the patients enrolled in the initial clinical trial have the same or an improved neurologic impairment score after six years of taking tafamidis, whereas 30% of the patients progress at a rate ≤ 1/5 of that predicted by the natural history. Importantly, all of the V30M FAP patients remain stage 1 patients after 6 years on tafamidis out of four stages of disease progression. [Data presented orally by Professor Coelho in Brazil in 2013]

text: Tafamidis functions by kinetic stabilization of the correctly folded

text: tetrameric

text: form of the

text: transthyretin

text: (TTR) protein. In patients with FAP, this protein dissociates in a process that is rate limiting for aggregation including

text: amyloid

text: fibril formation, causing failure of the autonomic nervous system and/or the peripheral nervous system (

text: neurodegeneration

text: ) initially and later failure of the heart. Kinetic Stabilization of tetrameric transthyretin in familial amyloid polyneuropathy patients provides the first pharmacologic evidence that the process of amyloid fibril formation causes this disease, as treatment with tafamidis dramatically slows the process of amyloid fibril formation and the degeneration of post-mitotic tissue. Sixty % of the patients enrolled in the initial clinical trial have the same or an improved neurologic impairment score after six years of taking tafamidis, whereas 30% of the patients progress at a rate ≤ 1/5 of that predicted by the natural history. Importantly, all of the V30M FAP patients remain stage 1 patients after 6 years on tafamidis out of four stages of disease progression. [Data presented orally by Professor Coelho in Brazil in 2013]

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answers: NDQ_010489

answers: neurologic impairment

answers: NDQ_010490

answers: FAP

answers: NDQ_010494

answers: amyloid polyneuropathy

answers: NDQ_010495

answers: familial amyloid polyneuropathy

answers: NDQ_010496

answers: autonomic nervous system

answers: NDQ_010497

answers: familial amyloid polyneuropathy

answers: NDQ_010498

answers: amyloid fibril formation

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: No

answer: 1

text: Bees that have been infected with CBPV may house millions of viral particles, with half of them concentrated in the head region, allowing it to cause symptoms similar to diseases relating to nervous system damage. In fact, viral particles have been found to concentrate in two centers; mushroom bodies involved in sensory processing, memory, learning, and motor control as well as the center dealing with locomotive control, behavior and orientation, and arousal.,

text: Bees that have been infected with CBPV may house millions of viral particles, with half of them concentrated in the head region, allowing it to cause symptoms similar to diseases relating to nervous system damage. In fact, viral particles have been found to concentrate in two centers; mushroom bodies involved in sensory processing, memory, learning, and motor control as well as the center dealing with locomotive control, behavior and orientation, and arousal.,

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answers: CBPV

answers: NDQ_010490

answers: CBPV

answers: NDQ_010494

answers: CBPV

answers: NDQ_010495

answers: CBPV

answers: NDQ_010496

answers: CBPV

answers: NDQ_010497

answers: CBPV

answers: NDQ_010498

answers: CBPV

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: Until recently, the medical literature did not indicate a connection among many genetic disorders, both genetic syndromes and genetic diseases, that are now being found to be related. As a result of new genetic research, some of these are, in fact, highly related in their root cause despite the widely varying symptoms apparent on clinical examination. Agenesis of the corpus callosum is one such disease, part of an emerging class of diseases called ciliopathies. The underlying cause may be a dysfunctional molecular mechanism in the primary cilia structures of the cell organelles that are present in many cellular types throughout the human body. The cilia defects adversely affect "numerous critical developmental signaling pathways" essential to cellular development and thus offer a plausible hypothesis for the often multi-symptom nature of a large set of syndromes and diseases. Known ciliopathies include primary ciliary dyskinesia, Bardet-Biedl syndrome, polycystic kidney and liver disease, nephronophthisis, Alstrom syndrome, Meckel-Gruber syndrome, and some forms of retinal degeneration.

text: Until recently, the medical literature did not indicate a connection among many

text: genetic disorder

text: s, both

text: genetic syndromes

text: and

text: genetic diseases

text: , that are now being found to be related. As a result of new genetic research, some of these are, in fact, highly related in their

text: root cause

text: despite the widely varying

text: symptoms

text: apparent on clinical examination. Agenesis of the corpus callosum is one such disease, part of an emerging class of diseases called

text: ciliopathies

text: . The underlying cause may be a dysfunctional molecular mechanism in the primary

text: cilia

text: structures of the

text: cell

text: organelle

text: s that are present in many cellular types throughout the

text: human

text: body. The cilia defects adversely affect "numerous critical developmental signaling pathways" essential to cellular development and thus offer a plausible hypothesis for the often

text: multi-symptom

text: nature of a large set of syndromes and diseases. Known ciliopathies include

text: primary ciliary dyskinesia

text: Bardet-Biedl syndrome

text: polycystic kidney

text: and

text: liver disease

text: nephronophthisis

text: Alstrom syndrome

text: Meckel-Gruber syndrome

text: , and some forms of

text: retinal degeneration

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answers: Agenesis of the corpus callosum

answers: NDQ_010490

answers: no

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answers: Meckel-Gruber syndrome

answers: NDQ_010495

answers: retinal degeneration

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answers: Agenesis of the corpus callosum

answers: NDQ_010497

answers: retinal degeneration

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answers: primary ciliary dyskinesia

answer: Yes

answer: Yes

answer: No

answer: No

answer: No

answer: 1

text: The most severe symptoms of Listeriosis result from involvement of the central nervous system (CNS). These severe and often fatal symptoms include meningitis, rhombencephalitis, and encephalitis.  These forms of disease are a direct result of Listeria pathogenicity mechanisms at the cellular level. Listerial infection involving the CNS can occur via three known routes: through the blood, through intracellular delivery, or through neuronal intracellular spread. Paracytophagous cell to cell spread offers Listeria access to the CNS by the latter two mechanisms.

text: The most severe symptoms of

text: Listeriosis

text: result from involvement of the central nervous system (CNS). These severe and often fatal symptoms include

text: meningitis

text: rhombencephalitis

text: , and

text: encephalitis

text: .  These forms of disease are a direct result of Listeria pathogenicity mechanisms at the cellular level. Listerial infection involving the CNS can occur via three known routes: through the blood, through intracellular delivery, or through neuronal intracellular spread. Paracytophagous cell to cell spread offers Listeria access to the CNS by the latter two mechanisms.

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answers: unanswerable

answers: NDQ_010490

answers: Listeriosis

answers: NDQ_010494

answers: encephalitis

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answers: encephalitis

answers: NDQ_010496

answers: encephalitis

answers: NDQ_010497

answers: encephalitis

answers: NDQ_010498

answers: encephalitis

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: Together with the other component of the autonomic nervous system, the parasympathetic nervous system, the sympathetic nervous system aids in the control of most of the body's internal organs. Reaction to stress—as in the flight-or-fight response—is thought to counteract the parasympathetic system, which generally works to promote maintenance of the body at rest. The comprehensive functions of both the parasympathetic and sympathetic nervous systems are not so straightforward, but this is a useful rule of thumb.

text: Together with the other component of the

text: autonomic nervous system

text: , the parasympathetic nervous system, the sympathetic nervous system aids in the control of most of the body's internal organs. Reaction to

text: stress

text: —as in the flight-or-fight response—is thought to counteract the

text: parasympathetic system

text: , which generally works to promote maintenance of the body at rest. The comprehensive functions of both the parasympathetic and sympathetic nervous systems are not so straightforward, but this is a useful rule of thumb.

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answers: Reaction to stress

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answers: autonomic nervous system

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answers: autonomic

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answers: autonomic nervous system

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answers: autonomic

answer: No

answer: No

answer: No

answer: No

answer: No

answer: 1

text: There are many central nervous system diseases and conditions, including infections of the central nervous system such as encephalitis and poliomyelitis, early-onset neurological disorders including ADHD and autism, late-onset neurodegenerative diseases such as Alzheimer's disease, Parkinson's disease, and essential tremor, autoimmune and inflammatory diseases such as multiple sclerosis and acute disseminated encephalomyelitis, genetic disorders such as Krabbe's disease and Huntington's disease, as well as amyotrophic lateral sclerosis and adrenoleukodystrophy. Lastly, cancers of the central nervous system can cause severe illness and, when malignant, can have very high mortality rates.

text: There are many central nervous system diseases and conditions, including

text: infections

text: of the central nervous system such as

text: encephalitis

text: and

text: poliomyelitis

text: , early-onset

text: neurological disorders

text: including

entity_name: Attention deficit hyperactivity disorder

text: ADHD

text: and

text: autism

text: , late-onset

text: neurodegenerative diseases

text: such as

text: Alzheimer's disease

text: Parkinson's disease

text: , and

text: essential tremor

text: autoimmune

text: and inflammatory diseases such as

text: multiple sclerosis

text: and

text: acute disseminated encephalomyelitis

text: , genetic disorders such as

text: Krabbe's disease

text: and

text: Huntington's disease

text: , as well as

text: amyotrophic lateral sclerosis

text: and

text: adrenoleukodystrophy

text: . Lastly, cancers of the central nervous system can cause severe illness and, when

text: malignant

text: , can have very high mortality rates.

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answers: poliomyelitis

answer: Yes

answer: Yes

answer: Yes

answer: Yes

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answer: 2

text: Meningism, also called meningismus or pseudomeningitis, is a set  of symptoms similar to those of meningitis but not caused by meningitis. Whereas meningitis is inflammation of the meninges (membranes that cover the central nervous system), meningism is caused by nonmeningitic irritation of the meninges, usually associated with acute febrile illness, especially in children and adolescents. Meningism involves the triad (3-symptom syndrome) of nuchal rigidity (neck stiffness), photophobia (intolerance of bright light), and headache. It therefore requires differentiating from other CNS problems with similar symptoms, including meningitis and some types of intracranial hemorrhage. Related clinical signs include Kernig's sign and three signs all named Brudzinski's sign.

text: Meningism, also called meningismus or pseudomeningitis, is a set  of

text: symptom

text: s similar to those of

text: meningitis

text: but not caused by meningitis. Whereas meningitis is

text: inflammation

text: of the

text: meninges

text: (membranes that cover the central nervous system), meningism is caused by nonmeningitic irritation of the meninges, usually associated with acute

text: febrile

text: illness, especially in children and adolescents. Meningism involves the triad (3-symptom

text: syndrome

text: ) of nuchal rigidity (

text: neck stiffness

text: photophobia

text: (intolerance of bright light), and

text: headache

text: . It therefore requires

text: differentiating

text: from other CNS problems with similar symptoms, including meningitis and some types of

text: intracranial hemorrhage

text: . Related clinical signs include

text: Kernig's sign

text: and three signs all named

text: Brudzinski's sign

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answers: NDQ_010489

answers: Meningism

answers: NDQ_010490

answers: intracranial hemorrhage

answers: NDQ_010494

answers: Meningitis

answers: NDQ_010495

answers: meningitis

answers: NDQ_010496

answers: Meningitis

answers: NDQ_010497

answers: Meningitis

answers: NDQ_010498

answers: Meningitis

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: Alcelaphine herpesvirus 2 is primarily transmitted by nasal emissions, by contact with water, flood, feces, or close contact. It can be destroyed by prolonged exposure to sunlight, but has a very long incubation period and thus may have a long period of time to infect other animals. There are three forms usually seen. The first a nasal form with fever, nasal inflammation and digestive problems that is very mild and only lasts a few days. The second is a more severe form that involves fever, diarrhea, heavy nasal discharge, and lymph system failure, with a morbidity of 28 percent. The so-called "head and eye" form is the most lethal and serious form, which results in all known effects and central nervous system damage, predated by nervous shakes and eventually muscular failure. Morbidity in some cases is as high as 90 percent.

text: Alcelaphine herpesvirus 2 is primarily transmitted by nasal emissions, by contact with water, flood, feces, or close contact. It can be destroyed by prolonged exposure to sunlight, but has a very long

text: incubation period

text: and thus may have a long period of time to infect other animals. There are three forms usually seen. The first a nasal form with fever, nasal inflammation and digestive problems that is very mild and only lasts a few days. The second is a more severe form that involves fever, diarrhea, heavy nasal discharge, and lymph system failure, with a

text: morbidity

text: of 28 percent. The so-called "head and eye" form is the most lethal and serious form, which results in all known effects and

text: central nervous system

text: damage, predated by nervous shakes and eventually muscular failure. Morbidity in some cases is as high as 90 percent.

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answers: NDQ_010489

answers: "head and eye" form

answers: NDQ_010490

answers: central nervous system

answers: NDQ_010494

answers: central nervous system damage

answers: NDQ_010495

answers: lymph system failure

answers: NDQ_010496

answers: muscular failure

answers: NDQ_010497

answers: central nervous system damage

answers: NDQ_010498

answers: nervous shakes

answer: No

answer: Yes

answer: No

answer: Yes

answer: No

answer: 1

text: Adult patients with encephalitis present with acute onset of fever, headache, confusion, and sometimes seizures. Younger children or infants may present irritability, poor appetite and fever.Neurological examinations usually reveal a drowsy or confused patient. Stiff neck, due to the irritation of the meninges covering the brain, indicates that the patient has either meningitis or meningoencephalitis.

text: Adult patients with encephalitis present with acute onset of fever, headache, confusion, and sometimes seizures. Younger children or infants may present irritability, poor appetite and fever.Neurological examinations usually reveal a drowsy or confused patient. Stiff neck, due to the irritation of the meninges covering the brain, indicates that the patient has either meningitis or

text: meningoencephalitis

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text: The nervous system and immune system require the appropriate degrees of cellular differentiation, organizational integrity, and neural network connectivity.  These operational features of the brain and nervous system may make signaling difficult to duplicate in severely diseased scenarios.  There are currently three classes of therapies that have been utilized in both animal models of disease and in human clinical trials.  These three classes include DNA methylation inhibitors, HDAC inhibitors, and RNA-based approaches.  DNA methylation inhibitors are used to activate previously silenced genes.  HDACs are a class of enzymes that have a broad set of biochemical modifications and can affect DNA demethylation and synergy with other therapeutic agents.  The final therapy includes using RNA-based approaches to enhance stability, specificity, and efficacy, especially in diseases that are caused by RNA alterations.  Emerging concepts concerning the complexity and versatility of the epigenome may suggest ways to target genomewide cellular processes.  Other studies suggest that eventual seminal regulator targets may be identified allowing with alterations to the massive epigenetic reprogramming during gametogenesis.  Many future treatments may extend beyond being purely therapeutic and may be preventable perhaps in the form of a vaccine. Newer high throughput technologies when combined with advances in imaging modalities such as in vivo optical nanotechnologies may give rise to even greater knowledge of genomic architecture, nuclear organization, and the interplay between the immune and nervous systems.

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answers: These operational features of the brain and nervous system may make signaling difficult to duplicate in severely diseased scenarios

answers: NDQ_010490

answers: RNA alterations

answers: NDQ_010494

answers: Alzheimer's disease

answers: NDQ_010495

answers: severely diseased

answers: NDQ_010496

answers: Parkinson's disease

answers: NDQ_010497

answers: HDAC

answers: NDQ_010498

answers: severely diseased scenarios

answer: Yes

answer: Yes

answer: Yes

answer: No

answer: Yes

answer: 1

text: Nervous system lipomas are rare congenital benign tumors of the central nervous system, mostly located in the medial line and especially in the corpus callosum. Generally, patients with these lipomas present with strokes. However, patients with the Pai syndrome don’t. That is why it is suggested that isolated nervous system lipomas have a different embryological origin than the lipomas present in the Pai syndrome. The treatment of CNS lipomas mainly consists of observation and follow up.

text: Nervous system lipomas are rare congenital benign tumors of the central nervous system, mostly located in the medial line and especially in the corpus callosum. Generally, patients with these lipomas present with strokes. However, patients with the Pai syndrome don’t. That is why it is suggested that isolated nervous system lipomas have a different embryological origin than the lipomas present in the Pai syndrome. The treatment of CNS lipomas mainly consists of observation and follow up.

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answers: 0

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answers: Generally, patients with these lipomas present with strokes

answers: NDQ_010490

answers: unanswerable

answers: NDQ_010494

answers: Pai syndrome

answers: NDQ_010495

answers: Pai syndrome

answers: NDQ_010496

answers: Pai syndrome

answers: NDQ_010497

answers: Pai syndrome

answers: NDQ_010498

answers: Pai syndrome

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: Apolipoprotein E (APOE) is a class of apolipoprotein found in the chylomicron and Intermediate-density lipoprotein (IDLs) that is essential for the normal catabolism of triglyceride-rich lipoprotein constituents. In peripheral tissues, APOE is primarily produced by the liver and macrophages, and mediates cholesterol metabolism in an isoform-dependent manner. In the central nervous system, APOE is mainly produced by astrocytes, and transports cholesterol to neurons via APOE receptors, which are members of the low density lipoprotein receptor gene family. APOE is the principal cholesterol carrier in the brain. This protein is involved in Alzheimer’s disease and cardiovascular disease.

text: Apolipoprotein E (APOE) is a class of

text: apolipoprotein

text: found in the

text: chylomicron

text: and

text: Intermediate-density lipoprotein (IDLs)

text: that is essential for the normal catabolism of triglyceride-rich lipoprotein constituents. In peripheral tissues, APOE is primarily produced by the

text: liver

text: and

text: macrophages

text: , and mediates

text: cholesterol

text: metabolism in an

text: isoform

text: -dependent manner. In the

text: central nervous system

text: , APOE is mainly produced by

text: astrocytes

text: , and transports

text: cholesterol

text: to

text: neurons

text: via APOE receptors, which are members of the

entity_name: Low-density lipoprotein receptor gene family

text: low density lipoprotein receptor gene family

text: . APOE is the principal

text: cholesterol

text: carrier in the brain. This protein is involved in

text: Alzheimer’s disease

text: and

text: cardiovascular disease

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answers: NDQ_010489

answers: Alzheimer’s disease

answers: NDQ_010490

answers: unanswerable

answers: NDQ_010494

answers: Alzheimer’s disease

answers: NDQ_010495

answers: Alzheimer’s disease

answers: NDQ_010496

answers: Alzheimer’s disease

answers: NDQ_010497

answers: Alzheimer’s disease

answers: NDQ_010498

answers: Alzheimer’s disease

answer: Yes

answer: Yes

answer: No

answer: Yes

answer: No

answer: 1

text: Cockayne syndrome (CS), also called Neill-Dingwall syndrome, is a rare fatal autosomal recessive neurodegenerative disorder characterized by growth failure, impaired development of the nervous system, abnormal sensitivity to sunlight (photosensitivity), eye disorders and premature aging. Failure to thrive and neurological disorders are criteria for diagnosis, while photosensitivity, hearing loss, eye abnormalities, and cavities are other very common features.  Problems with any or all of the internal organs are possible.  It is associated with a group of disorders called leukodystrophies, which are conditions characterized by degradation of neurological white matter. The underlying disorder is a defect in a DNA repair mechanism. Unlike other defects of DNA repair, patients with CS are not predisposed to cancer or infection. Cockayne syndrome is a rare but destructive disease usually resulting in death within the first or second decade of life. The mutation of specific genes in Cockayne syndrome is known, but the widespread effects and its relationship with DNA repair is yet to be well understood.

text: Cockayne syndrome (CS), also called Neill-Dingwall syndrome, is a rare fatal

text: autosomal

text: recessive

text: neurodegenerative

text: disorder characterized by growth failure, impaired development of the

text: nervous system

text: , abnormal sensitivity to

text: sunlight

text: photosensitivity

text: ), eye disorders and

text: premature aging

text: Failure to thrive

text: and neurological disorders are criteria for diagnosis, while photosensitivity, hearing loss, eye abnormalities, and cavities are other very common features.  Problems with any or all of the internal organs are possible.  It is associated with a group of disorders called

text: leukodystrophies

text: , which are conditions characterized by degradation of neurological

text: white matter

text: . The underlying disorder is a defect in a

text: DNA repair

text: mechanism. Unlike other defects of DNA repair, patients with CS are not predisposed to cancer or infection. Cockayne syndrome is a rare but destructive disease usually resulting in death within the first or second decade of life. The mutation of specific genes in Cockayne syndrome is known, but the widespread effects and its relationship with DNA repair is yet to be well understood.

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text: Other notable people include Mordechai Gebirtig, who was one of the most influential and popular writers of Yiddish songs and poems. He died there in 1942. Miriam Akavia, an Israeli writer, survived the Kraków ghetto and concentration camps.  Renowned dermatologist and co-discoverer of Reyes Syndrome, Dr Jim (Jacob) Baral was also a Kraków Ghetto survivor. Bernard Offen, born in 1929 in Kraków survived the Ghetto and several Nazi concentration camps.

text: Other notable people include

text: Mordechai Gebirtig

text: , who was one of the most influential and popular writers of Yiddish songs and poems. He died there in 1942.

text: Miriam Akavia

text: , an Israeli writer, survived the Kraków ghetto and concentration camps.  Renowned dermatologist and co-discoverer of

text: Reyes Syndrome

text: , Dr Jim (Jacob) Baral was also a Kraków Ghetto survivor.

text: Bernard Offen

text: , born in 1929 in Kraków survived the Ghetto and several Nazi concentration camps.

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text: It has been known since the 1970s that when fat cells become too big, they may become bloated and dysfunctional, or “sick.”  It has also been known since the 1940s that if fat gain occurs in the belly or abdominal (visceral) region, that this is another example of sick fat that promotes metabolic diseases.  Finally, if fat growth exceeds its blood vessel supply, then the lack of oxygen delivery by the blood may also result in pathologic responses from fat tissue. In summary, it has been known for decades that adverse changes in fat cell and fat tissue anatomy result in sick fat which causes metabolic disease.More recently, an additional event that has prompted the concept and term of “adiposopathy” is the evolving recognition of the profound hormone and immune importance of fat tissue.  In the past, fat cells and fat tissue were considered by many as being inert, or hormonally and immunologically inactive.  However, this has been proven incorrect, and it is now generally accepted that fat tissue is an active hormone, and immune organ.Yet another historical event that moved medical science towards recognizing fat tissue as an underlying cause of metabolic disease has been the problematic issues that have arisen with the “metabolic syndrome.”  The metabolic syndrome is a commonly used medical term to describe atherosclerotic coronary heart disease risk factors that tend to cluster together.  Over the years, there have been at least 15 other similar terms used to generally describe the same clustering. According to one common definition, a patient is said to have “metabolic syndrome” if he or she has 3 or more the 5 following criteria: (1) abdominal obesity, (2) elevated triglycerides, (3) reduced high density lipoprotein cholesterol levels, (4) high blood pressure, and (5) high blood sugar.  However, different scientific and medical organizations have different definitions for the “metabolic syndrome.” Also, the term “metabolic syndrome” does not describe, nor is it intended to describe a unifying cause of any disease.  Since “metabolic syndrome” is not a disease, regulatory agencies (such as the Food and Drug Administration) do not approve drugs to treat metabolic syndrome, as a specific indicated use.  Finally, the “metabolic syndrome” may be no better at predicting atherosclerotic coronary heart disease than an assessment of its individual components. As such, major scientific and medical organizations have questioned the usefulness of the metabolic syndrome, sometimes in open conflict with other major scientific and medical organizations.

text: It has been known since the 1970s that when fat cells become too big, they may become bloated and dysfunctional, or “sick.”  It has also been known since the 1940s that if fat gain occurs in the belly or abdominal (visceral) region, that this is another example of sick fat that promotes metabolic diseases.  Finally, if fat growth exceeds its blood vessel supply, then the lack of oxygen delivery by the blood may also result in pathologic responses from fat tissue. In summary, it has been known for decades that adverse changes in fat cell and fat tissue anatomy result in sick fat which causes metabolic disease.More recently, an additional event that has prompted the concept and term of “adiposopathy” is the evolving recognition of the profound

text: hormone

text: and

text: immune

text: importance of fat tissue.  In the past, fat cells and fat tissue were considered by many as being inert, or hormonally and immunologically inactive.  However, this has been proven incorrect, and it is now generally accepted that fat tissue is an active hormone, and immune organ.Yet another historical event that moved medical science towards recognizing fat tissue as an underlying cause of metabolic disease has been the problematic issues that have arisen with the “

text: metabolic syndrome

text: .”  The

text: metabolic syndrome

text: is a commonly used medical term to describe atherosclerotic coronary heart disease risk factors that tend to cluster together.  Over the years, there have been at least 15 other similar terms used to generally describe the same clustering. According to one common definition, a patient is said to have “metabolic syndrome” if he or she has 3 or more the 5 following criteria: (1) abdominal obesity, (2) elevated triglycerides, (3) reduced high density lipoprotein cholesterol levels, (4) high blood pressure, and (5) high blood sugar.  However, different scientific and medical organizations have different definitions for the “metabolic syndrome.” Also, the term “

text: metabolic syndrome

text: ” does not describe, nor is it intended to describe a unifying cause of any disease.  Since “

text: metabolic syndrome

text: ” is not a disease, regulatory agencies (such as the

text: Food and Drug Administration

text: ) do not approve drugs to treat metabolic syndrome, as a specific indicated use.  Finally, the “

text: metabolic syndrome

text: ” may be no better at predicting atherosclerotic coronary heart disease than an assessment of its individual components. As such, major scientific and medical organizations have questioned the usefulness of the

text: metabolic syndrome

text: , sometimes in open conflict with other major scientific and medical organizations.

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answers: It has been known for decades that adverse changes in fat cell and fat tissue anatomy result in sick fat which causes metabolic disease.

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answers: It has been known for decades that adverse changes in fat cell and fat tissue anatomy result in sick fat which causes metabolic disease.

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answers: metabolic syndrome” is not a disease

answer: No

answer: No

answer: No

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text: While advanced research at the University is carried out in virtually every field of modern medicine, its contribution is most widely recognized in such disciplines as immunology, endocrinology, oncology, nuclear medicine, anaesthesiology, surgery, nephrology, gynaecology and obstetrics (especially perinatology), ecology, parasitic and tropical diseases, AIDS, viral hepatitis, hematology, child oncology, cytophysiology and cytopathology, development of the central nervous system, immunology of skin diseases, clinical pharmacology, genetics, audiology, phoniatrics and optometry.

text: While advanced research at the University is carried out in virtually every field of modern medicine, its contribution is most widely recognized in such disciplines as immunology, endocrinology, oncology, nuclear medicine, anaesthesiology, surgery, nephrology, gynaecology and obstetrics (especially perinatology), ecology, parasitic and tropical diseases, AIDS, viral hepatitis, hematology, child oncology, cytophysiology and cytopathology, development of the central nervous system, immunology of skin diseases, clinical pharmacology, genetics, audiology, phoniatrics and optometry.

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answers: central nervous system

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answers: AIDS

answer: Yes

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answer: 1

text: Neuro: of or having to do with the nervous system. Nervous System: An organ system that coordinates the activities of muscles, monitors organs, constructs and processes data received from the senses and initiates actions. The human nervous system coordinates the functions of itself and all organ systems including but not limited to the cardiovascular system, respiratory system, skin, digestive system, immune system, hormonal, metabolic, musculoskeletal, endocrine system, blood and reproductive system. Optimal function of the organism as a whole depends upon the proper function of the nervous system.

text: Neuro: of or having to do with the nervous system.

text: Nervous System

text: : An

text: organ

text: system that

text: coordinate

text: s the activities of muscles, monitors organs, constructs and processes data received from the senses and initiates actions. The human nervous system coordinates the functions of itself and all organ systems including but not limited to the

text: cardiovascular system

text: respiratory system

text: skin

text: digestive system

text: immune system

text: , hormonal, metabolic, musculoskeletal,

text: endocrine system

text: , blood and

text: reproductive system

text: . Optimal function of the organism as a whole depends upon the proper function of the nervous system.

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answers: The nervous system coordinates the functions of itself and all organ systems

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answer: No

answer: No

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text: At the same developmental time, it is expressed in the ventricular zone in the telecephalon and hindbrain.  PCP-2 was also detected in the developing inner nuclear layer of the retina, in the olfactory epithelium of the nasal cavities, and in the meningeal coverings of the brain.   In the developing chick nervous system, PCP-2 mRNA is expressed in the ventral midline of the neural tube and in the border between the midbrain and hindbrain, known as the mid-hindbrain boundary.  PCP-2 mRNA is also observed in the ventricular zone of the developing chick neural retina.

text: At the same developmental time, it is expressed in the ventricular zone in the telecephalon and hindbrain.  PCP-2 was also detected in the developing inner nuclear layer of the retina, in the olfactory epithelium of the nasal cavities, and in the meningeal coverings of the brain.   In the developing chick nervous system, PCP-2 mRNA is expressed in the ventral midline of the neural tube and in the border between the midbrain and hindbrain, known as the mid-hindbrain boundary.  PCP-2 mRNA is also observed in the ventricular zone of the developing chick neural retina.

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answers: In the developing chick nervous system

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answers: PCP-2

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answer: No

answer: No

answer: No

answer: No

answer: No

answer: 1

text: The phlebotomic action opens a channel for contamination of the host species with bacteria, viruses and blood-borne parasites contained in the hematophagous organism. Thus, many animal and human infectious diseases are transmitted by hematophagous species, such as the bubonic plague, Chagas disease, dengue fever, eastern equine encephalitis, filariasis, leishmaniasis, Lyme disease, malaria, rabies, sleeping sickness, St. Louis encephalitis, tularemia, typhus, Rocky Mountain spotted fever, West Nile fever, Zika fever, and many others.

text: The phlebotomic action opens a channel for contamination of the host species with

text: bacteria

text: viruses

text: and blood-borne

text: parasite

text: s contained in the hematophagous organism. Thus, many animal and human

text: infectious disease

text: s are transmitted by hematophagous species, such as the

text: bubonic plague

text: Chagas disease

text: dengue fever

text: eastern equine encephalitis

text: filariasis

text: leishmaniasis

text: Lyme disease

text: malaria

text: rabies

text: sleeping sickness

text: St. Louis encephalitis

text: tularemia

text: typhus

text: Rocky Mountain spotted fever

text: West Nile fever

text: Zika fever

text: , and many others.

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answer: Yes

answer: No

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text: The demyelinating diseases of the peripheral nervous system include:

text: The demyelinating diseases of the

text: peripheral nervous system

text: include:

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answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: AIF1 is a protein that exists in the cytoplasm, and it is highly evolutionarily conserved. It is also possibly identical to three other proteins, Iba-1, MRF-1 (microglia response factor) and daintain. However complete functional profiles of all three proteins and how they overlap is unknown. IBA1 is a 17-kDa EF hand protein that is specifically expressed in macrophages / microglia and is upregulated during the activation of these cells. Iba1 expression is up-regulated in microglia following nerve injury, central nervous system ischemia, and several other brain diseases.

text: AIF1 is a protein that exists in the

text: cytoplasm

text: , and it is highly evolutionarily conserved. It is also possibly identical to three other proteins, Iba-1, MRF-1 (microglia response factor) and daintain. However complete functional profiles of all three proteins and how they overlap is unknown. IBA1 is a 17-

text: kDa

text: EF hand

text: protein that is specifically expressed in

text: macrophage

text: s /

text: microglia

text: and is upregulated during the activation of these cells. Iba1 expression is up-regulated in microglia following nerve injury, central nervous system ischemia, and several other brain diseases.

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text: In adults, testing electrolytes, blood glucose and calcium levels is important to rule these out as causes, as is an electrocardiogram. A lumbar puncture may be useful to diagnose a central nervous system infection but is not routinely needed. Routine antiseizure medical levels in the blood are not required in adults or children. In children additional tests may be required.

text: In adults, testing electrolytes,

text: blood glucose

text: and calcium levels is important to rule these out as causes, as is an

text: electrocardiogram

text: . A lumbar puncture may be useful to diagnose a

text: central nervous system

text: infection but is not routinely needed. Routine antiseizure medical levels in the blood are not required in adults or children. In children additional tests may be required.

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text: The nervous system also controls limb stiffness to modulate the degree of accuracy that is required for a given task. For example, the accuracy required to grab a cup off of a table if very different from that of a surgeon performing a precise task with a scalpel. To accomplish these tasks with varying degrees of required accuracy, the nervous system adjusts limb stiffness. To accomplish very accurate tasks higher stiffness is required, however, when performing tasks where accuracy is not as imperative, lower limb stiffness is needed. In the case of accurate movements, the central nervous system is able to precisely control limb stiffness to limit movement variability. The cerebellum also plays a large role in controlling for the accuracy of movements.

text: The

text: nervous system

text: also controls limb stiffness to modulate the degree of accuracy that is required for a given task. For example, the accuracy required to grab a cup off of a table if very different from that of a surgeon performing a precise task with a

text: scalpel

text: . To accomplish these tasks with varying degrees of required accuracy, the nervous system adjusts limb stiffness. To accomplish very accurate tasks higher stiffness is required, however, when performing tasks where accuracy is not as imperative, lower limb stiffness is needed. In the case of accurate movements, the central nervous system is able to precisely control limb stiffness to limit movement variability. The

text: cerebellum

text: also plays a large role in controlling for the accuracy of movements.

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answers: The nervous system

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answer: No

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answer: 1

text: Anti-CASPR2 nervous system manifestations: Patients with anti-CASPIR2 antibodies develop symptoms from the CNS and/or the peripheral nervous system. The classic presentation is with Morvan’s syndrome, a disease with the features of neuromyotonia (i.e. peripheral hyperexcetability) and limbic encephalitis. Other patients present with isolated neuromyotonia or limbic encephalitis.

text: Anti-CASPR2 nervous system manifestations: Patients with anti-CASPIR2 antibodies develop symptoms from the CNS and/or the peripheral nervous system. The classic presentation is with Morvan’s syndrome, a disease with the features of neuromyotonia (i.e. peripheral hyperexcetability) and limbic encephalitis. Other patients present with isolated neuromyotonia or limbic encephalitis.

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answers: Morvan’s syndrome

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answers: Morvan’s syndrome

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answers: Morvan’s syndrome

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answers: Morvan’s syndrome

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answers: Morvan’s syndrome

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answers: Morvan’s syndrome

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answers: Morvan’s syndrome

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: The causative agent for Japanese encephalitis virus is an enveloped virus of the genus flavivirus and is closely related to the West Nile virus and the St. Louis encephalitis virus. The positive sense single-stranded RNA genome is packaged in the capsid which is formed by the capsid protein. The outer envelope is formed by envelope protein and is the protective antigen. It aids in entry of the virus into the inside of the cell. The genome also encodes several nonstructural proteins (NS1, NS2a, NS2b, NS3, N4a, NS4b, NS5). NS1 is produced as secretory form also. NS3 is a putative helicase, and NS5 is the viral polymerase. It has been noted that Japanese encephalitis infects the lumen of the endoplasmic reticulum (ER) and rapidly accumulates substantial amounts of viral proteins for the Japanese Encephalitis.

text: The causative agent for Japanese encephalitis virus is an enveloped virus of the genus

text: flavivirus

text: and is closely related to the

text: West Nile virus

text: and the

text: St. Louis encephalitis

text: virus. The positive sense single-stranded

text: RNA

text: genome is packaged in the

text: capsid

text: which is formed by the capsid protein. The outer envelope is formed by envelope protein and is the protective antigen. It aids in entry of the virus into the inside of the cell. The genome also encodes several nonstructural proteins (NS1, NS2a, NS2b, NS3, N4a, NS4b, NS5). NS1 is produced as secretory form also. NS3 is a putative

text: helicase

text: , and NS5 is the viral

text: polymerase

text: . It has been noted that Japanese encephalitis infects the

text: lumen

text: of the

text: endoplasmic reticulum

text: (ER) and rapidly accumulates substantial amounts of viral proteins for the Japanese Encephalitis.

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answers: Japanese encephalitis

answer: Yes

answer: Yes

answer: No

answer: Yes

answer: Yes

answer: 1

text: Tbx5 is a gene that is located on the long arm of chromosome 12. Tbx5 produces a protein called T-box 5 that acts as a transcription factor. The Tbx5 gene is involved with forelimb and heart development. This gene impacts the early development of the forelimb by triggering FGF-10 (Fibroblast Growth Factor 10). Tbx5 is involved with the development of the four chambers in the heart, the electrical conducting system, and the septum separating the right and left sides of the heart. A mutation in this gene can cause Holt-Oram syndrome or Amelia syndrome. Holt-Oram syndrome can cause several different defects. One effect of Holt-Oram syndrome is a hole in the septum. Another symptom of this syndrome is bone abnormalities in the fingers, wrists, or arms. An additional defect that Holt-Oram syndrome can cause is a conduction disease leading to abnormal heart rates and arrhythmias. Amelia syndrome is a condition where forelimb malformation occurs because FGF-10 is not triggered due to Tbx5 mutations. This condition can lead to the absence of one or both forelimbs.

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answers: Holt-Oram syndrome

answers: NDQ_010490

answers: unanswerable

answers: NDQ_010494

answers: Amelia syndrome

answers: NDQ_010495

answers: conduction disease

answers: NDQ_010496

answers: Amelia syndrome

answers: NDQ_010497

answers: Amelia syndrome

answers: NDQ_010498

answers: Holt-Oram syndrome

answer: No

answer: No

answer: No

answer: No

answer: No

answer: 1

text: Although the bodies of dead sea otters often sink at sea, necropsies of beached carcasses provide some insights into the causes of mortality. A study of 105 sea otters that had washed ashore between 1998 and 2001 determined the major causes of death to be protozoal encephalitis, acanthocephalan parasite infection, shark attack, and cardiac disease. Infectious disease alone caused 63.8% of deaths, and in most of these cases the disease was caused by parasites. Infection, particularly Toxoplasma gondii encephalitis, was often present in otters that had died of cardiac disease, suggesting that infection may have contributed to the onset of cardiac disease. T. gondii encephalitis was also strongly associated with shark bites, perhaps because the disease causes abnormal behavior that increases the likelihood of shark attack.

text: Although the bodies of dead sea otters often sink at sea,

text: necropsies

text: of beached carcasses provide some insights into the causes of mortality. A study of 105 sea otters that had washed ashore between 1998 and 2001 determined the major causes of death to be

text: protozoal

text: encephalitis

text: acanthocephalan

text: parasite infection,

text: shark

text: attack, and

text: cardiac disease

text: . Infectious disease alone caused 63.8% of deaths, and in most of these cases the disease was caused by parasites. Infection, particularly

text: Toxoplasma gondii

text: encephalitis, was often present in otters that had died of cardiac disease, suggesting that infection may have contributed to the onset of cardiac disease. T. gondii encephalitis was also strongly associated with shark bites, perhaps because the disease causes abnormal behavior that increases the likelihood of

text: shark attack

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text: Research into autoimmune encephalitis have advanced a lot recently. Both tumour associated (paraneoplastic) and non-tumour associated conditions are recognized. Different antibodies are more or less selective for different parts of the brain. Antibodies are directed towards intracellular antigens in the classic paraneoplastic syndromes or synaptic proteins in paraneoplastic or non-tumour associated conditions. The VGKC-complex associated autoimmune encephalitis is an example of the latter form.

text: Research into

text: autoimmune encephalitis

text: have advanced a lot recently. Both tumour associated (paraneoplastic) and non-tumour associated conditions are recognized. Different antibodies are more or less selective for different parts of the brain. Antibodies are directed towards intracellular antigens in the classic paraneoplastic syndromes or synaptic proteins in paraneoplastic or non-tumour associated conditions. The VGKC-complex associated autoimmune encephalitis is an example of the latter form.

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answers: encephalitis

answer: Yes

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answer: 1

text: Bapineuzumab (nicknamed "bapi") is a humanized monoclonal antibody that acts on the nervous system and may have potential therapeutic value for the treatment of Alzheimer's disease and possibly glaucoma. However, in 2012 it failed to produce significant cognitive improvements in patients in two major trials, despite lowering key biomarkers of AD, amyloid brain plaque and hyperphosphorylated tau protein in CSF.

text: Bapineuzumab (nicknamed "bapi") is a

text: humanized monoclonal antibody

text: that acts on the

text: nervous system

text: and may have potential therapeutic value for the treatment of

text: Alzheimer's disease

text: and possibly

text: glaucoma

text: . However, in 2012 it failed to produce significant cognitive improvements in patients in two major trials, despite lowering key biomarkers of AD, amyloid brain plaque and

text: hyperphosphorylated

text: tau protein

text: in

text: CSF

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answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: Chronic oral exposure in animals affects the liver, the kidney, the spleen, the adrenal and thyroid glands, the central nervous system, and the immune system.  Toxaphene stimulates the central nervous system by antagonizing neurons leading to hyperpolarization of neurons and increased neuronal activity.

text: Chronic oral exposure in animals affects the liver, the kidney, the spleen, the adrenal and thyroid glands, the central nervous system, and the immune system.  Toxaphene stimulates the central nervous system by antagonizing neurons leading to hyperpolarization of neurons and increased neuronal activity.

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answers: Toxaphene

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answers: Toxaphene

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: Vogt–Koyanagi–Harada disease (VKH), also known as Vogt–Koyanagi–Harada syndrome and uveomeningitis syndrome, is a multisystem disease of presumed autoimmune  cause, that affects pigmented tissues, which have melanin. The most significant manifestation is bilateral, diffuse uveitis, which affects the eye. VKH may variably also involve the inner ear with effects on hearing, the skin, and the meninges of the central nervous system.

text: Vogt–Koyanagi–Harada disease (VKH), also known as Vogt–Koyanagi–Harada syndrome and uveomeningitis syndrome, is a multisystem disease of presumed

text: autoimmune

text: cause, that affects pigmented tissues, which have

text: melanin

text: . The most significant manifestation is bilateral, diffuse

text: uveitis

text: , which affects the eye. VKH may variably also involve the

text: inner ear

text: with effects on hearing, the skin, and the

text: meninges

text: of the

text: central nervous system

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answers: VKH

answers: NDQ_010490

answers: autoimmune

answers: NDQ_010494

answers: central nervous system

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answers: autoimmune

answers: NDQ_010496

answers: uveomeningitis

answers: NDQ_010497

answers: central nervous system

answers: NDQ_010498

answers: central nervous system

answer: Yes

answer: Yes

answer: No

answer: No

answer: Yes

answer: 1

text: Encephalitis is an inflammation of the brain. It is usually caused by a foreign substance or a viral infection. Symptoms of this disease include headache, neck pain, drowsiness, nausea, and fever. If caused by the West Nile virus, it may be lethal to humans, as well as birds and horses.

text: Encephalitis is an inflammation of the brain. It is usually caused by a foreign substance or a

text: viral infection

text: . Symptoms of this disease include headache, neck pain, drowsiness, nausea, and fever. If caused by the

text: West Nile virus

text: , it may be lethal to humans, as well as birds and horses.

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answers: Encephalitis

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answers: Encephalitis

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answers: Encephalitis

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answers: Encephalitis

answers: NDQ_010497

answers: Encephalitis

answers: NDQ_010498

answers: Encephalitis

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: Yes

answer: 1

text: The large-scale inflammation that occurs in the subarachnoid space during meningitis is not a direct result of bacterial infection but can rather largely be attributed to the response of the immune system to the entry of bacteria into the central nervous system. When components of the bacterial cell membrane are identified by the immune cells of the brain (astrocytes and microglia), they respond by releasing large amounts of cytokines, hormone-like mediators that recruit other immune cells and stimulate other tissues to participate in an immune response. The blood–brain barrier becomes more permeable, leading to "vasogenic" cerebral edema (swelling of the brain due to fluid leakage from blood vessels). Large numbers of white blood cells enter the CSF, causing inflammation of the meninges and leading to "interstitial" edema (swelling due to fluid between the cells). In addition, the walls of the blood vessels themselves become inflamed (cerebral vasculitis), which leads to decreased blood flow and a third type of edema, "cytotoxic" edema. The three forms of cerebral edema all lead to increased intracranial pressure; together with the lowered blood pressure often encountered in acute infection, this means that it is harder for blood to enter the brain, consequently brain cells are deprived of oxygen and undergo apoptosis (programmed cell death).

text: The large-scale

text: inflammation

text: that occurs in the subarachnoid space during meningitis is not a direct result of bacterial infection but can rather largely be attributed to the response of the

text: immune system

text: to the entry of bacteria into the

text: central nervous system

text: . When components of the bacterial

text: cell membrane

text: are identified by the immune cells of the brain (

text: astrocyte

text: s and

text: microglia

text: ), they respond by releasing large amounts of

text: cytokine

text: s, hormone-like mediators that recruit other immune cells and stimulate other tissues to participate in an immune response. The blood–brain barrier becomes more permeable, leading to

text: "vasogenic" cerebral edema

text: (swelling of the brain due to fluid leakage from blood vessels). Large numbers of

text: white blood cell

text: s enter the CSF, causing inflammation of the meninges and leading to

text: "interstitial" edema

text: (swelling due to fluid between the cells). In addition, the walls of the blood vessels themselves become inflamed (cerebral vasculitis), which leads to decreased blood flow and a third type of edema,

text: "cytotoxic" edema

text: . The three forms of cerebral edema all lead to increased

text: intracranial pressure

text: ; together with the lowered blood pressure often encountered in

text: acute infection

text: , this means that it is harder for blood to enter the brain, consequently

text: brain cell

text: s are deprived of oxygen and undergo

text: apoptosis

text: programmed cell death

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answers: Meningitis

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answers: NDQ_010498

answers: meningitis

answer: Yes

answer: Yes

answer: Yes

answer: No

answer: No

answer: 1

text: Flaviviruses constitute a family of linear, single-stranded RNA(+) viruses. Flaviviruses include the West Nile virus, dengue virus, Tick-borne Encephalitis Virus, Yellow Fever Virus, and several other viruses. Many flavivirus species can replicate in both mammalian and insect cells. Most flaviviruses are arthropod borne and multiply in both vertebrates and arthropods. The viruses in this family that are of veterinary importance include Japanese encephalitis virus, St. Louis encephalitis virus, West Nile virus, Israel turkey meningoencephalomyelitis virus, Sitiawan virus, Wesselsbron virus, yellow fever virus and the tick-borne flaviviruses e.g. louping ill virus.

text: Flavivirus

text: es constitute a family of linear, single-stranded RNA(+) viruses. Flaviviruses include the

text: West Nile virus

text: dengue virus

text: Tick-borne Encephalitis

text: Virus,

text: Yellow Fever

text: Virus, and several other viruses. Many flavivirus species can replicate in both mammalian and insect cells. Most flaviviruses are

text: arthropod

text: borne and multiply in both vertebrates and arthropods. The viruses in this family that are of veterinary importance include

text: Japanese encephalitis virus

text: St. Louis encephalitis virus

text: West Nile virus

text: Israel turkey meningoencephalomyelitis virus

text: Sitiawan virus

text: Wesselsbron virus

text: , yellow fever virus and the tick-borne flaviviruses e.g.

text: louping ill virus

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answers: Flaviviruses

answers: NDQ_010490

answers: Tick-borne Encephalitis Virus

answers: NDQ_010494

answers: encephalitis

answers: NDQ_010495

answers: Meningoencephalomyelitis

answers: NDQ_010496

answers: encephalitis

answers: NDQ_010497

answers: Tick-borne Encephalitis Virus

answers: NDQ_010498

answers: St. Louis encephalitis virus

answer: No

answer: No

answer: No

answer: No

answer: No

answer: 1

text: Drinking more than one drink a day for women or two drinks for men increases the risk of heart disease, high blood pressure, atrial fibrillation, and stroke. Risk is greater in younger people due to binge drinking which may result in violence or accidents. About 3.3 million deaths (5.9% of all deaths) are believed to be due to alcohol each year. Alcoholism reduces a person's life expectancy by around ten years and alcohol use is the third leading cause of early death in the United States. No professional medical association recommends that people who are nondrinkers should start drinking wine. Long-term alcohol abuse can cause a number of physical symptoms, including cirrhosis of the liver, pancreatitis, epilepsy, polyneuropathy, alcoholic dementia, heart disease, nutritional deficiencies, peptic ulcers and sexual dysfunction, and can eventually be fatal. Other physical effects include an increased risk of developing cardiovascular disease, malabsorption, alcoholic liver disease, and cancer. Damage to the central nervous system and peripheral nervous system can occur from sustained alcohol consumption. A wide range of immunologic defects can result and there may be a generalized skeletal fragility, in addition to a recognized tendency to accidental injury, resulting a propensity to bone fractures.

text: Drinking more than one drink a day for women or two drinks for men increases the risk of heart disease,

text: high blood pressure

text: atrial fibrillation

text: , and

text: stroke

text: . Risk is greater in younger people due to

text: binge drinking

text: which may result in violence or accidents. About 3.3 million deaths (5.9% of all deaths) are believed to be due to alcohol each year. Alcoholism reduces a person's life expectancy by around ten years and alcohol use is the third leading cause of early death in the United States. No professional medical association recommends that people who are nondrinkers should start drinking wine. Long-term alcohol abuse can cause a number of physical symptoms, including

text: cirrhosis

text: of the liver,

text: pancreatitis

text: epilepsy

text: polyneuropathy

text: alcoholic dementia

text: , heart disease, nutritional deficiencies,

text: peptic ulcers

text: and

text: sexual dysfunction

text: , and can eventually be fatal. Other physical effects include an increased risk of developing

text: cardiovascular disease

text: malabsorption

text: alcoholic liver disease

text: , and

text: cancer

text: . Damage to the

text: central nervous system

text: and

text: peripheral nervous system

text: can occur from sustained alcohol consumption. A wide range of immunologic defects can result and there may be a generalized skeletal fragility, in addition to a recognized tendency to accidental injury, resulting a propensity to bone fractures.

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answers: NDQ_010489

answers: Damage to the central nervous system

answers: NDQ_010490

answers: unanswerable

answers: NDQ_010494

answers: alcoholic dementia

answers: NDQ_010495

answers: polyneuropathy

answers: NDQ_010496

answers: epilepsy

answers: NDQ_010497

answers: polyneuropathy

answers: NDQ_010498

answers: epilepsy

answer: Yes

answer: Yes

answer: No

answer: Yes

answer: No

answer: 1

text: Nicotinic acetylcholine receptors, or nAChRs, are neuron receptor proteins that respond to the neurotransmitter acetylcholine. Nicotinic receptors also respond to drugs, including the nicotinic receptor agonist nicotine. They are found in the central nervous system of humans, and also play two important roles in the peripheral nervous system: (1) they transmit outgoing signals from the presynaptic to the postsynaptic cells within the sympathetic and parasympathetic nervous system, and (2) they are the receptors found on skeletal muscle that receive acetylcholine released to signal for muscular contraction. Nicotinic receptors are also found in other creatures. In insects, the cholinergic system is limited to the central nervous system.

text: Nicotinic acetylcholine receptors, or nAChRs, are neuron receptor proteins that respond to the neurotransmitter

text: acetylcholine

text: . Nicotinic receptors also respond to drugs, including the nicotinic receptor agonist

text: nicotine

text: . They are found in the central nervous system of humans, and also play two important roles in the peripheral nervous system: (1) they transmit outgoing signals from the presynaptic to the postsynaptic cells within the sympathetic and parasympathetic nervous system, and (2) they are the receptors found on skeletal muscle that receive acetylcholine released to signal for muscular contraction. Nicotinic receptors are also found in other creatures. In

text: insect

text: s, the

text: cholinergic

text: system is limited to the

text: central nervous system

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